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Anticancer Drugs Brief of Cancer Cancer starts when cells in a part of the body start to grow out of control. Cancer cell growth is different from normal cell growth. Instead of dying, cancer cells continue to grow and form new, abnormal cells. Cancer cells can also invade (grow into) other tissues, something that normal cells cant do. Growing out of control and invading other tissues are what makes a cell a cancer cell. Cells become cancer cells because of DNA damage. DNA is in every cell and it directs all its actions. In a normal cell, when DNA is damaged the cell either repairs the damage or dies. In cancer cells, the damaged DNA is not repaired, but the cell doesnt die like it should. Instead, the cell goes on making new cells that the body doesnt need. These new cells all have the same damaged DNA as the first abnormal cell does. People can inherit abnormal DNA (its passed on from their parents), but most often DNA damage is caused by mistakes that happen while the normal cell is reproducing or by something in the environment. Sometimes the cause of the DNA damage may be something obvious likecigarette smokingorsun exposure. In most cases, the cancer cells form a tumor. Over time, the tumors can replace normal tissue, crowd it, or push it aside. Some cancers, likeleukemia, rarely form tumors. "Drivers" of Cancer The genetic changes that contribute to cancer tend to affect three main types of genes. proto-oncogenes,tumor suppressor genes and DNA repair genes. These changes are sometimes called drivers of cancer. Proto-oncogenes are involved in normal cell growth and division. However, when these genes are altered in certain ways or are more active than normal, they may become cancer-causing genes (or oncogenes), allowing cells to grow and survive when they should not. Tumor suppressor genes are also involved in controlling cell growth and division. Cells with certain alterations in tumor suppressor genes may divide in an uncontrolled manner. DNA repair genes are involved in fixing damaged DNA. Cells with mutations in these genes tend to develop additional mutations in other genes. Together, these mutations may cause the cells to become cancerous. How cancer spreads? Cancer cells often travel to other parts of the body where they can grow and form new tumors. This happens when the cancer cells get into the bodys bloodstream or lymph vessels. The process of cancer spreading is calledmetastasis.]]>

Anticancer Drugs Brief of Cancer Cancer starts when cells in a part of the body start to grow out of control. Cancer cell growth is different from normal cell growth. Instead of dying, cancer cells continue to grow and form new, abnormal cells. Cancer cells can also invade (grow into) other tissues, something that normal cells cant do. Growing out of control and invading other tissues are what makes a cell a cancer cell. Cells become cancer cells because of DNA damage. DNA is in every cell and it directs all its actions. In a normal cell, when DNA is damaged the cell either repairs the damage or dies. In cancer cells, the damaged DNA is not repaired, but the cell doesnt die like it should. Instead, the cell goes on making new cells that the body doesnt need. These new cells all have the same damaged DNA as the first abnormal cell does. People can inherit abnormal DNA (its passed on from their parents), but most often DNA damage is caused by mistakes that happen while the normal cell is reproducing or by something in the environment. Sometimes the cause of the DNA damage may be something obvious likecigarette smokingorsun exposure. In most cases, the cancer cells form a tumor. Over time, the tumors can replace normal tissue, crowd it, or push it aside. Some cancers, likeleukemia, rarely form tumors. "Drivers" of Cancer The genetic changes that contribute to cancer tend to affect three main types of genes. proto-oncogenes,tumor suppressor genes and DNA repair genes. These changes are sometimes called drivers of cancer. Proto-oncogenes are involved in normal cell growth and division. However, when these genes are altered in certain ways or are more active than normal, they may become cancer-causing genes (or oncogenes), allowing cells to grow and survive when they should not. Tumor suppressor genes are also involved in controlling cell growth and division. Cells with certain alterations in tumor suppressor genes may divide in an uncontrolled manner. DNA repair genes are involved in fixing damaged DNA. Cells with mutations in these genes tend to develop additional mutations in other genes. Together, these mutations may cause the cells to become cancerous. How cancer spreads? Cancer cells often travel to other parts of the body where they can grow and form new tumors. This happens when the cancer cells get into the bodys bloodstream or lymph vessels. The process of cancer spreading is calledmetastasis.]]>
Sat, 25 Apr 2015 07:35:41 GMT /slideshow/anticancer-drugs-47402286/47402286 DocNody@slideshare.net(DocNody) Anticancer drugs DocNody Anticancer Drugs Brief of Cancer Cancer starts when cells in a part of the body start to grow out of control. Cancer cell growth is different from normal cell growth. Instead of dying, cancer cells continue to grow and form new, abnormal cells. Cancer cells can also invade (grow into) other tissues, something that normal cells cant do. Growing out of control and invading other tissues are what makes a cell a cancer cell. Cells become cancer cells because of DNA damage. DNA is in every cell and it directs all its actions. In a normal cell, when DNA is damaged the cell either repairs the damage or dies. In cancer cells, the damaged DNA is not repaired, but the cell doesnt die like it should. Instead, the cell goes on making new cells that the body doesnt need. These new cells all have the same damaged DNA as the first abnormal cell does. People can inherit abnormal DNA (its passed on from their parents), but most often DNA damage is caused by mistakes that happen while the normal cell is reproducing or by something in the environment. Sometimes the cause of the DNA damage may be something obvious likecigarette smokingorsun exposure. In most cases, the cancer cells form a tumor. Over time, the tumors can replace normal tissue, crowd it, or push it aside. Some cancers, likeleukemia, rarely form tumors. "Drivers" of Cancer鐃 The genetic changes that contribute to cancer tend to affect three main types of genes. proto-oncogenes,tumor suppressor genes and DNA repair genes. These changes are sometimes called drivers of cancer. Proto-oncogenes are involved in normal cell growth and division. However, when these genes are altered in certain ways or are more active than normal, they may become cancer-causing genes (or oncogenes), allowing cells to grow and survive when they should not. Tumor suppressor genes are also involved in controlling cell growth and division. Cells with certain alterations in tumor suppressor genes may divide in an uncontrolled manner. DNA repair genes are involved in fixing damaged DNA. Cells with mutations in these genes tend to develop additional mutations in other genes. Together, these mutations may cause the cells to become cancerous. How cancer spreads? Cancer cells often travel to other parts of the body where they can grow and form new tumors. This happens when the cancer cells get into the bodys bloodstream or lymph vessels. The process of cancer spreading is calledmetastasis. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/anticancerdrugs-150425073541-conversion-gate01-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Anticancer Drugs Brief of Cancer Cancer starts when cells in a part of the body start to grow out of control. Cancer cell growth is different from normal cell growth. Instead of dying, cancer cells continue to grow and form new, abnormal cells. Cancer cells can also invade (grow into) other tissues, something that normal cells cant do. Growing out of control and invading other tissues are what makes a cell a cancer cell. Cells become cancer cells because of DNA damage. DNA is in every cell and it directs all its actions. In a normal cell, when DNA is damaged the cell either repairs the damage or dies. In cancer cells, the damaged DNA is not repaired, but the cell doesnt die like it should. Instead, the cell goes on making new cells that the body doesnt need. These new cells all have the same damaged DNA as the first abnormal cell does. People can inherit abnormal DNA (its passed on from their parents), but most often DNA damage is caused by mistakes that happen while the normal cell is reproducing or by something in the environment. Sometimes the cause of the DNA damage may be something obvious likecigarette smokingorsun exposure. In most cases, the cancer cells form a tumor. Over time, the tumors can replace normal tissue, crowd it, or push it aside. Some cancers, likeleukemia, rarely form tumors. &quot;Drivers&quot; of Cancer鐃 The genetic changes that contribute to cancer tend to affect three main types of genes. proto-oncogenes,tumor suppressor genes and DNA repair genes. These changes are sometimes called drivers of cancer. Proto-oncogenes are involved in normal cell growth and division. However, when these genes are altered in certain ways or are more active than normal, they may become cancer-causing genes (or oncogenes), allowing cells to grow and survive when they should not. Tumor suppressor genes are also involved in controlling cell growth and division. Cells with certain alterations in tumor suppressor genes may divide in an uncontrolled manner. DNA repair genes are involved in fixing damaged DNA. Cells with mutations in these genes tend to develop additional mutations in other genes. Together, these mutations may cause the cells to become cancerous. How cancer spreads? Cancer cells often travel to other parts of the body where they can grow and form new tumors. This happens when the cancer cells get into the bodys bloodstream or lymph vessels. The process of cancer spreading is calledmetastasis.
Anticancer drugs from Doc Nody
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