ºÝºÝߣshows by User: NAJEEBULLAHSOFI / http://www.slideshare.net/images/logo.gif ºÝºÝߣshows by User: NAJEEBULLAHSOFI / Thu, 26 Nov 2020 15:02:01 GMT ºÝºÝߣShare feed for ºÝºÝߣshows by User: NAJEEBULLAHSOFI Transcatheter therapies for congenital heart disease /slideshow/transcatheter-therapies-for-congenital-heart-disease-239503506/239503506 transcathetertherapiesforcongenitalheartdisease-201126150201
Transcatheter therapies for congenital heart disease]]>
Transcatheter therapies for congenital heart disease]]> Thu, 26 Nov 2020 15:02:01 GMT /slideshow/transcatheter-therapies-for-congenital-heart-disease-239503506/239503506 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Transcatheter therapies for congenital heart disease NAJEEBULLAHSOFI Transcatheter therapies for congenital heart disease <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/transcathetertherapiesforcongenitalheartdisease-201126150201-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Transcatheter therapies for congenital heart disease
Transcatheter therapies for congenital heart disease from NAJEEB ULLAH SOFI
]]> 157 0 https://cdn.slidesharecdn.com/ss_thumbnails/transcathetertherapiesforcongenitalheartdisease-201126150201-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Cardiac troponin elevation in patients without a specific diagnosis /NAJEEBULLAHSOFI/cardiac-troponin-elevation-in-patients-without-a-specific-diagnosis-238876629 cardiactroponinelevationinpatientswithoutaspecificdiagnosis-201014171101
Cardiac troponin (cTn) elevation is a common finding in acutely admitted patients, even in the absence of acute coronary syndrome. In some of these patients, no etiology of cTn elevation can be identified. The term troponinemia is sometimes used to describe this scenario. The proportion of patients discharged from the emergency department without a specified diagnosis but with cTn levels above the 99th percentile has been reported as 31%. Acute but subtle increases in cTn levels may also be difficult to distinguish from chronic cTn elevation which is a common finding in the elderly, patients with renal failure, or patients with chronic cardiac conditions This often causes frustration among clinicians, and the term troponinemia has been coined to label this scenario. ]]>
Cardiac troponin (cTn) elevation is a common finding in acutely admitted patients, even in the absence of acute coronary syndrome. In some of these patients, no etiology of cTn elevation can be identified. The term troponinemia is sometimes used to describe this scenario. The proportion of patients discharged from the emergency department without a specified diagnosis but with cTn levels above the 99th percentile has been reported as 31%. Acute but subtle increases in cTn levels may also be difficult to distinguish from chronic cTn elevation which is a common finding in the elderly, patients with renal failure, or patients with chronic cardiac conditions This often causes frustration among clinicians, and the term troponinemia has been coined to label this scenario. ]]> Wed, 14 Oct 2020 17:11:00 GMT /NAJEEBULLAHSOFI/cardiac-troponin-elevation-in-patients-without-a-specific-diagnosis-238876629 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Cardiac troponin elevation in patients without a specific diagnosis NAJEEBULLAHSOFI Cardiac troponin (cTn) elevation is a common finding in acutely admitted patients, even in the absence of acute coronary syndrome. In some of these patients, no etiology of cTn elevation can be identified. The term troponinemia is sometimes used to describe this scenario. The proportion of patients discharged from the emergency department without a specified diagnosis but with cTn levels above the 99th percentile has been reported as 31%. Acute but subtle increases in cTn levels may also be difficult to distinguish from chronic cTn elevation which is a common finding in the elderly, patients with renal failure, or patients with chronic cardiac conditions This often causes frustration among clinicians, and the term troponinemia has been coined to label this scenario. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cardiactroponinelevationinpatientswithoutaspecificdiagnosis-201014171101-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Cardiac troponin (cTn) elevation is a common finding in acutely admitted patients, even in the absence of acute coronary syndrome. In some of these patients, no etiology of cTn elevation can be identified. The term troponinemia is sometimes used to describe this scenario. The proportion of patients discharged from the emergency department without a specified diagnosis but with cTn levels above the 99th percentile has been reported as 31%. Acute but subtle increases in cTn levels may also be difficult to distinguish from chronic cTn elevation which is a common finding in the elderly, patients with renal failure, or patients with chronic cardiac conditions This often causes frustration among clinicians, and the term troponinemia has been coined to label this scenario.
Cardiac troponin elevation in patients without a specific diagnosis from NAJEEB ULLAH SOFI
]]> 46 0 https://cdn.slidesharecdn.com/ss_thumbnails/cardiactroponinelevationinpatientswithoutaspecificdiagnosis-201014171101-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Dual antiplatelet therapy duration based on ischemic and bleeding risks after coronary stenting /NAJEEBULLAHSOFI/dual-antiplatelet-therapy-duration-based-on-ischemic-and-bleeding-risks-after-coronary-stenting dualantiplatelettherapydurationbasedonischemicandbleedingrisksaftercoronarystenting-201014170648
The PRECISE-DAPT score is a 5-item bleeding risk prediction model developed to estimate the bleeding risk in patients who receive dual antiplatelet therapy (DAPT) after stent implantation The categorization of patients based on the PRECISE-DAPT score was shown to be useful to inform decision-making for duration of DAPT in stented patients ]]>
The PRECISE-DAPT score is a 5-item bleeding risk prediction model developed to estimate the bleeding risk in patients who receive dual antiplatelet therapy (DAPT) after stent implantation The categorization of patients based on the PRECISE-DAPT score was shown to be useful to inform decision-making for duration of DAPT in stented patients ]]> Wed, 14 Oct 2020 17:06:48 GMT /NAJEEBULLAHSOFI/dual-antiplatelet-therapy-duration-based-on-ischemic-and-bleeding-risks-after-coronary-stenting NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Dual antiplatelet therapy duration based on ischemic and bleeding risks after coronary stenting NAJEEBULLAHSOFI The PRECISE-DAPT score is a 5-item bleeding risk prediction model developed to estimate the bleeding risk in patients who receive dual antiplatelet therapy (DAPT) after stent implantation The categorization of patients based on the PRECISE-DAPT score was shown to be useful to inform decision-making for duration of DAPT in stented patients <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/dualantiplatelettherapydurationbasedonischemicandbleedingrisksaftercoronarystenting-201014170648-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> The PRECISE-DAPT score is a 5-item bleeding risk prediction model developed to estimate the bleeding risk in patients who receive dual antiplatelet therapy (DAPT) after stent implantation The categorization of patients based on the PRECISE-DAPT score was shown to be useful to inform decision-making for duration of DAPT in stented patients
Dual antiplatelet therapy duration based on ischemic and bleeding risks after coronary stenting from NAJEEB ULLAH SOFI
]]> 91 0 https://cdn.slidesharecdn.com/ss_thumbnails/dualantiplatelettherapydurationbasedonischemicandbleedingrisksaftercoronarystenting-201014170648-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 CMR in nstemi /slideshow/cmr-in-nstemi/238876387 cmrinnstemi-201014165951
Acute myocardial infarction (MI) typically occurs from a plaque rupture or erosion within a coronary artery,known as the infarct-related artery (IRA) In patients with NSTEMI compared with those with ST-segment–elevation MI, identification of the IRA can be challenging because patients are more likely to present with either multivessel coronary artery disease (CAD) or insignificant CAD ]]>
Acute myocardial infarction (MI) typically occurs from a plaque rupture or erosion within a coronary artery,known as the infarct-related artery (IRA) In patients with NSTEMI compared with those with ST-segment–elevation MI, identification of the IRA can be challenging because patients are more likely to present with either multivessel coronary artery disease (CAD) or insignificant CAD ]]> Wed, 14 Oct 2020 16:59:51 GMT /slideshow/cmr-in-nstemi/238876387 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) CMR in nstemi NAJEEBULLAHSOFI Acute myocardial infarction (MI) typically occurs from a plaque rupture or erosion within a coronary artery,known as the infarct-related artery (IRA) In patients with NSTEMI compared with those with ST-segment–elevation MI, identification of the IRA can be challenging because patients are more likely to present with either multivessel coronary artery disease (CAD) or insignificant CAD <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cmrinnstemi-201014165951-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Acute myocardial infarction (MI) typically occurs from a plaque rupture or erosion within a coronary artery,known as the infarct-related artery (IRA) In patients with NSTEMI compared with those with ST-segment–elevation MI, identification of the IRA can be challenging because patients are more likely to present with either multivessel coronary artery disease (CAD) or insignificant CAD
CMR in nstemi from NAJEEB ULLAH SOFI
]]> 31 0 https://cdn.slidesharecdn.com/ss_thumbnails/cmrinnstemi-201014165951-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Coronary Calcium Modification /NAJEEBULLAHSOFI/coronary-calcium-modification coronaryarterycalcification-1-201014165605
Coronary artery calcification (CAC) results in reduced vascular compliance, abnormal vasomotor responses, and impaired myocardial perfusion. The presence of CAC is associated with worse outcomes in the general population and in patients undergoing revascularization Two recognized types of CAC are Atherosclerotic (Intimal) Medial artery calcification]]>
Coronary artery calcification (CAC) results in reduced vascular compliance, abnormal vasomotor responses, and impaired myocardial perfusion. The presence of CAC is associated with worse outcomes in the general population and in patients undergoing revascularization Two recognized types of CAC are Atherosclerotic (Intimal) Medial artery calcification]]> Wed, 14 Oct 2020 16:56:05 GMT /NAJEEBULLAHSOFI/coronary-calcium-modification NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Coronary Calcium Modification NAJEEBULLAHSOFI Coronary artery calcification (CAC) results in reduced vascular compliance, abnormal vasomotor responses, and impaired myocardial perfusion. The presence of CAC is associated with worse outcomes in the general population and in patients undergoing revascularization Two recognized types of CAC are Atherosclerotic (Intimal) Medial artery calcification <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/coronaryarterycalcification-1-201014165605-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Coronary artery calcification (CAC) results in reduced vascular compliance, abnormal vasomotor responses, and impaired myocardial perfusion. The presence of CAC is associated with worse outcomes in the general population and in patients undergoing revascularization Two recognized types of CAC are Atherosclerotic (Intimal) Medial artery calcification
Coronary Calcium Modification from NAJEEB ULLAH SOFI
]]> 690 0 https://cdn.slidesharecdn.com/ss_thumbnails/coronaryarterycalcification-1-201014165605-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Conduction system abnormalities after transcatheter aortic valve replacement mechanism, prediction, and management. /slideshow/conduction-system-abnormalities-after-transcatheter-aortic-valve-replacement-mechanism-prediction-and-management/238876282 conductionsystemabnormalitiesaftertranscatheteraorticvalvereplacementmechanismpredictionandmanagemen-201014164706
Aortic stenosis (AS) is a common form of valvular heart disease, the global burden of which continues to increase. Untreated, severe symptomatic AS carries a high mortality rate. Initially performed in patients deemed unsuitable for surgery, and then advancing to become an option for patients with high, intermediate, and now low operative risk, TAVR has revolutionized the treatment of symptomatic severe AS . TAVR is noninferior to surgical aortic valve replacement (SAVR) with regard to mortality at 1 year ]]>
Aortic stenosis (AS) is a common form of valvular heart disease, the global burden of which continues to increase. Untreated, severe symptomatic AS carries a high mortality rate. Initially performed in patients deemed unsuitable for surgery, and then advancing to become an option for patients with high, intermediate, and now low operative risk, TAVR has revolutionized the treatment of symptomatic severe AS . TAVR is noninferior to surgical aortic valve replacement (SAVR) with regard to mortality at 1 year ]]> Wed, 14 Oct 2020 16:47:06 GMT /slideshow/conduction-system-abnormalities-after-transcatheter-aortic-valve-replacement-mechanism-prediction-and-management/238876282 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Conduction system abnormalities after transcatheter aortic valve replacement mechanism, prediction, and management. NAJEEBULLAHSOFI Aortic stenosis (AS) is a common form of valvular heart disease, the global burden of which continues to increase. Untreated, severe symptomatic AS carries a high mortality rate. Initially performed in patients deemed unsuitable for surgery, and then advancing to become an option for patients with high, intermediate, and now low operative risk, TAVR has revolutionized the treatment of symptomatic severe AS . TAVR is noninferior to surgical aortic valve replacement (SAVR) with regard to mortality at 1 year <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/conductionsystemabnormalitiesaftertranscatheteraorticvalvereplacementmechanismpredictionandmanagemen-201014164706-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Aortic stenosis (AS) is a common form of valvular heart disease, the global burden of which continues to increase. Untreated, severe symptomatic AS carries a high mortality rate. Initially performed in patients deemed unsuitable for surgery, and then advancing to become an option for patients with high, intermediate, and now low operative risk, TAVR has revolutionized the treatment of symptomatic severe AS . TAVR is noninferior to surgical aortic valve replacement (SAVR) with regard to mortality at 1 year
Conduction system abnormalities after transcatheter aortic valve replacement mechanism, prediction, and management. from NAJEEB ULLAH SOFI
]]> 83 0 https://cdn.slidesharecdn.com/ss_thumbnails/conductionsystemabnormalitiesaftertranscatheteraorticvalvereplacementmechanismpredictionandmanagemen-201014164706-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Cardiac MRI /slideshow/cardiac-mri-238876273/238876273 cardiacmri-201014164524
Unlike other modalities, MRI offers the capability to modulate both the emitted and received signals so that a multitude of tissue characteristics can be examined and differentiated without the need to change scanner hardware. As a result, from a single imaging session, one could obtain a wealth of information regarding cardiac function and morphology, myocardial perfusion & viability, hemodynamics, large vessel anatomy. CMR is now considered the gold standard for the assessment of regional and global systolic function, myocardial infarction (MI) and viability, and the assessment of congenital heart disease. ]]>
Unlike other modalities, MRI offers the capability to modulate both the emitted and received signals so that a multitude of tissue characteristics can be examined and differentiated without the need to change scanner hardware. As a result, from a single imaging session, one could obtain a wealth of information regarding cardiac function and morphology, myocardial perfusion & viability, hemodynamics, large vessel anatomy. CMR is now considered the gold standard for the assessment of regional and global systolic function, myocardial infarction (MI) and viability, and the assessment of congenital heart disease. ]]> Wed, 14 Oct 2020 16:45:24 GMT /slideshow/cardiac-mri-238876273/238876273 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Cardiac MRI NAJEEBULLAHSOFI Unlike other modalities, MRI offers the capability to modulate both the emitted and received signals so that a multitude of tissue characteristics can be examined and differentiated without the need to change scanner hardware. As a result, from a single imaging session, one could obtain a wealth of information regarding cardiac function and morphology, myocardial perfusion & viability, hemodynamics, large vessel anatomy. CMR is now considered the gold standard for the assessment of regional and global systolic function, myocardial infarction (MI) and viability, and the assessment of congenital heart disease. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cardiacmri-201014164524-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Unlike other modalities, MRI offers the capability to modulate both the emitted and received signals so that a multitude of tissue characteristics can be examined and differentiated without the need to change scanner hardware. As a result, from a single imaging session, one could obtain a wealth of information regarding cardiac function and morphology, myocardial perfusion &amp; viability, hemodynamics, large vessel anatomy. CMR is now considered the gold standard for the assessment of regional and global systolic function, myocardial infarction (MI) and viability, and the assessment of congenital heart disease.
Cardiac MRI from NAJEEB ULLAH SOFI
]]> 675 0 https://cdn.slidesharecdn.com/ss_thumbnails/cardiacmri-201014164524-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 CCM in CHF: FIX-HF-5C Study /slideshow/ccm-in-chf-fixhf5c-study/238876131 ccminchf-201014163158
Cardiac contractility modulation (CCM) is an electrical device-based approach developed for the treatment of CHF with reduced and midrange ejection fractions (EFs). CCM signals are non-excitatory electrical signals applied during the cardiac absolute refractory period that enhance the strength of cardiac muscular contraction ]]>
Cardiac contractility modulation (CCM) is an electrical device-based approach developed for the treatment of CHF with reduced and midrange ejection fractions (EFs). CCM signals are non-excitatory electrical signals applied during the cardiac absolute refractory period that enhance the strength of cardiac muscular contraction ]]> Wed, 14 Oct 2020 16:31:58 GMT /slideshow/ccm-in-chf-fixhf5c-study/238876131 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) CCM in CHF: FIX-HF-5C Study NAJEEBULLAHSOFI Cardiac contractility modulation (CCM) is an electrical device-based approach developed for the treatment of CHF with reduced and midrange ejection fractions (EFs). CCM signals are non-excitatory electrical signals applied during the cardiac absolute refractory period that enhance the strength of cardiac muscular contraction <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/ccminchf-201014163158-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Cardiac contractility modulation (CCM) is an electrical device-based approach developed for the treatment of CHF with reduced and midrange ejection fractions (EFs). CCM signals are non-excitatory electrical signals applied during the cardiac absolute refractory period that enhance the strength of cardiac muscular contraction
CCM in CHF: FIX-HF-5C Study from NAJEEB ULLAH SOFI
]]> 95 0 https://cdn.slidesharecdn.com/ss_thumbnails/ccminchf-201014163158-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 LMCA : Evolution of management GABG to PCI /slideshow/lmca-evolution-of-management-gabg-to-pci/238876040 lmca-201014162659
At the bifurcation, the shear forces peak at the carina, creating areas of high endothelial shear stress. The development of atherosclerosis in the LMCA has been linked to flow haemodynamics, with atherosclerotic plaques described at areas of low endothelial shear stress in the lateral wall of the bifurcation, opposite to the carina. Conversely, the carina is often free from disease, probably owing to the protective effect of high shear stress against plaque formation. The length of the LMCA also influences stenosis location and morphology. In short LMCA (<10 mm), lesions develop more frequently near the ostium than in the bifurcation (55% versus 38%), whereas in long arteries, lesions develop predominantly near the bifurcation (ostium 18% versus bifurcation 77%). Furthermore, ostial lesions more frequently have negative remodelling, larger luminal areas, and less calcium than distal lesions. ]]>
At the bifurcation, the shear forces peak at the carina, creating areas of high endothelial shear stress. The development of atherosclerosis in the LMCA has been linked to flow haemodynamics, with atherosclerotic plaques described at areas of low endothelial shear stress in the lateral wall of the bifurcation, opposite to the carina. Conversely, the carina is often free from disease, probably owing to the protective effect of high shear stress against plaque formation. The length of the LMCA also influences stenosis location and morphology. In short LMCA (<10 mm), lesions develop more frequently near the ostium than in the bifurcation (55% versus 38%), whereas in long arteries, lesions develop predominantly near the bifurcation (ostium 18% versus bifurcation 77%). Furthermore, ostial lesions more frequently have negative remodelling, larger luminal areas, and less calcium than distal lesions. ]]> Wed, 14 Oct 2020 16:26:59 GMT /slideshow/lmca-evolution-of-management-gabg-to-pci/238876040 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) LMCA : Evolution of management GABG to PCI NAJEEBULLAHSOFI At the bifurcation, the shear forces peak at the carina, creating areas of high endothelial shear stress. The development of atherosclerosis in the LMCA has been linked to flow haemodynamics, with atherosclerotic plaques described at areas of low endothelial shear stress in the lateral wall of the bifurcation, opposite to the carina. Conversely, the carina is often free from disease, probably owing to the protective effect of high shear stress against plaque formation. The length of the LMCA also influences stenosis location and morphology. In short LMCA (<10 mm), lesions develop more frequently near the ostium than in the bifurcation (55% versus 38%), whereas in long arteries, lesions develop predominantly near the bifurcation (ostium 18% versus bifurcation 77%). Furthermore, ostial lesions more frequently have negative remodelling, larger luminal areas, and less calcium than distal lesions. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/lmca-201014162659-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> At the bifurcation, the shear forces peak at the carina, creating areas of high endothelial shear stress. The development of atherosclerosis in the LMCA has been linked to flow haemodynamics, with atherosclerotic plaques described at areas of low endothelial shear stress in the lateral wall of the bifurcation, opposite to the carina. Conversely, the carina is often free from disease, probably owing to the protective effect of high shear stress against plaque formation. The length of the LMCA also influences stenosis location and morphology. In short LMCA (&lt;10 mm), lesions develop more frequently near the ostium than in the bifurcation (55% versus 38%), whereas in long arteries, lesions develop predominantly near the bifurcation (ostium 18% versus bifurcation 77%). Furthermore, ostial lesions more frequently have negative remodelling, larger luminal areas, and less calcium than distal lesions.
LMCA : Evolution of management GABG to PCI from NAJEEB ULLAH SOFI
]]> 108 0 https://cdn.slidesharecdn.com/ss_thumbnails/lmca-201014162659-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Renal artery stenosis /slideshow/renal-artery-stenosis-238873100/238873100 ras-201014133043
Renal artery stenosis is the leading cause of secondary hypertension and may lead to : Resistant (refractory) hypertension, Progressive decline in renal function, and Cardiac destabilization syndromes (Flash pulmonary edema, recurrent heart failure, or acute coronary syndromes) ]]>
Renal artery stenosis is the leading cause of secondary hypertension and may lead to : Resistant (refractory) hypertension, Progressive decline in renal function, and Cardiac destabilization syndromes (Flash pulmonary edema, recurrent heart failure, or acute coronary syndromes) ]]> Wed, 14 Oct 2020 13:30:43 GMT /slideshow/renal-artery-stenosis-238873100/238873100 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Renal artery stenosis NAJEEBULLAHSOFI Renal artery stenosis is the leading cause of secondary hypertension and may lead to : Resistant (refractory) hypertension, Progressive decline in renal function, and Cardiac destabilization syndromes (Flash pulmonary edema, recurrent heart failure, or acute coronary syndromes) <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/ras-201014133043-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Renal artery stenosis is the leading cause of secondary hypertension and may lead to : Resistant (refractory) hypertension, Progressive decline in renal function, and Cardiac destabilization syndromes (Flash pulmonary edema, recurrent heart failure, or acute coronary syndromes)
Renal artery stenosis from NAJEEB ULLAH SOFI
]]> 1955 0 https://cdn.slidesharecdn.com/ss_thumbnails/ras-201014133043-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 InStent Resetenosis: An Algorithmic Approach to Diagnosis and Treatment /slideshow/instent-resetenosis-an-algorithmic-approach-to-diagnosis-and-treatment/238872844 isr-201014131448
BMS were developed to mitigate elastic recoil and negative remodeling, but they remain prone to NIH. DES were developed to prevent NIH, and these devices (especially first-generation DES) can be accompanied by delayed reendothelialization, which has been associated with stent thrombosis. Even in the contemporary era of percutaneous coronary intervention using drug-eluting stents, ISR remains a common problem, occurring in 5% to 20% of cases, depending on several patient and lesion characteristics. The cumulative rates of DES failure have created a major clinical problem so that > 10% of all PCIs done in the United States are to treat ISR, and the number of ISR interventions appears to be increasing year over year ]]>
BMS were developed to mitigate elastic recoil and negative remodeling, but they remain prone to NIH. DES were developed to prevent NIH, and these devices (especially first-generation DES) can be accompanied by delayed reendothelialization, which has been associated with stent thrombosis. Even in the contemporary era of percutaneous coronary intervention using drug-eluting stents, ISR remains a common problem, occurring in 5% to 20% of cases, depending on several patient and lesion characteristics. The cumulative rates of DES failure have created a major clinical problem so that > 10% of all PCIs done in the United States are to treat ISR, and the number of ISR interventions appears to be increasing year over year ]]> Wed, 14 Oct 2020 13:14:48 GMT /slideshow/instent-resetenosis-an-algorithmic-approach-to-diagnosis-and-treatment/238872844 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) InStent Resetenosis: An Algorithmic Approach to Diagnosis and Treatment NAJEEBULLAHSOFI BMS were developed to mitigate elastic recoil and negative remodeling, but they remain prone to NIH. DES were developed to prevent NIH, and these devices (especially first-generation DES) can be accompanied by delayed reendothelialization, which has been associated with stent thrombosis. Even in the contemporary era of percutaneous coronary intervention using drug-eluting stents, ISR remains a common problem, occurring in 5% to 20% of cases, depending on several patient and lesion characteristics. The cumulative rates of DES failure have created a major clinical problem so that > 10% of all PCIs done in the United States are to treat ISR, and the number of ISR interventions appears to be increasing year over year <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/isr-201014131448-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> BMS were developed to mitigate elastic recoil and negative remodeling, but they remain prone to NIH. DES were developed to prevent NIH, and these devices (especially first-generation DES) can be accompanied by delayed reendothelialization, which has been associated with stent thrombosis. Even in the contemporary era of percutaneous coronary intervention using drug-eluting stents, ISR remains a common problem, occurring in 5% to 20% of cases, depending on several patient and lesion characteristics. The cumulative rates of DES failure have created a major clinical problem so that &gt; 10% of all PCIs done in the United States are to treat ISR, and the number of ISR interventions appears to be increasing year over year
InStent Resetenosis: An Algorithmic Approach to Diagnosis and Treatment from NAJEEB ULLAH SOFI
]]> 187 0 https://cdn.slidesharecdn.com/ss_thumbnails/isr-201014131448-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 Perioperative cardiac assesment and interventions /slideshow/perioperative-cardiac-assesment-and-interventions/238460088 perioperativecardiacassesmentandinterventions-200912053935
Noncardiac surgery (NCS) is associated with a considerable risk of adverse cardiac events among individuals with coronary artery or aortic valve disease]]>
Noncardiac surgery (NCS) is associated with a considerable risk of adverse cardiac events among individuals with coronary artery or aortic valve disease]]> Sat, 12 Sep 2020 05:39:35 GMT /slideshow/perioperative-cardiac-assesment-and-interventions/238460088 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) Perioperative cardiac assesment and interventions NAJEEBULLAHSOFI Noncardiac surgery (NCS) is associated with a considerable risk of adverse cardiac events among individuals with coronary artery or aortic valve disease <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/perioperativecardiacassesmentandinterventions-200912053935-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Noncardiac surgery (NCS) is associated with a considerable risk of adverse cardiac events among individuals with coronary artery or aortic valve disease
Perioperative cardiac assesment and interventions from NAJEEB ULLAH SOFI
]]> 125 0 https://cdn.slidesharecdn.com/ss_thumbnails/perioperativecardiacassesmentandinterventions-200912053935-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 PAD & Lower Extremity Interventions /slideshow/pad-lower-extremity-interventions/238460043 padlowerextremityinterventions-200912053222
PAD can be diagnosed in asymptomatic individuals by a combination of physical examination and simple, noninvasive Doppler ultrasonography to measure the ankle–brachial index]]>
PAD can be diagnosed in asymptomatic individuals by a combination of physical examination and simple, noninvasive Doppler ultrasonography to measure the ankle–brachial index]]> Sat, 12 Sep 2020 05:32:22 GMT /slideshow/pad-lower-extremity-interventions/238460043 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) PAD & Lower Extremity Interventions NAJEEBULLAHSOFI PAD can be diagnosed in asymptomatic individuals by a combination of physical examination and simple, noninvasive Doppler ultrasonography to measure the ankle–brachial index <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/padlowerextremityinterventions-200912053222-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> PAD can be diagnosed in asymptomatic individuals by a combination of physical examination and simple, noninvasive Doppler ultrasonography to measure the ankle–brachial index
PAD & Lower Extremity Interventions from NAJEEB ULLAH SOFI
]]> 379 0 https://cdn.slidesharecdn.com/ss_thumbnails/padlowerextremityinterventions-200912053222-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 New Heart Failure modalities: HIS Bundle Pacing & Cardiac Contractility Modulation /slideshow/new-heart-failure-modalities-his-bundle-pacing-cardiac-contractility-modulation/238460028 newhfmodalitieshbpccm-200912052843
His bundle pacing activates the ventricles via the native His-Purkinje system, resulting in true physiological pacing, and, therefore, is a promising alternate site for pacing in bradycardia and traditional CRT indications in cases where it can overcome left bundle branch block CCM signals do not elicit a new contraction; rather, they influence the biology of the failing myocardium]]>
His bundle pacing activates the ventricles via the native His-Purkinje system, resulting in true physiological pacing, and, therefore, is a promising alternate site for pacing in bradycardia and traditional CRT indications in cases where it can overcome left bundle branch block CCM signals do not elicit a new contraction; rather, they influence the biology of the failing myocardium]]> Sat, 12 Sep 2020 05:28:43 GMT /slideshow/new-heart-failure-modalities-his-bundle-pacing-cardiac-contractility-modulation/238460028 NAJEEBULLAHSOFI@slideshare.net(NAJEEBULLAHSOFI) New Heart Failure modalities: HIS Bundle Pacing & Cardiac Contractility Modulation NAJEEBULLAHSOFI His bundle pacing activates the ventricles via the native His-Purkinje system, resulting in true physiological pacing, and, therefore, is a promising alternate site for pacing in bradycardia and traditional CRT indications in cases where it can overcome left bundle branch block CCM signals do not elicit a new contraction; rather, they influence the biology of the failing myocardium <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/newhfmodalitieshbpccm-200912052843-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> His bundle pacing activates the ventricles via the native His-Purkinje system, resulting in true physiological pacing, and, therefore, is a promising alternate site for pacing in bradycardia and traditional CRT indications in cases where it can overcome left bundle branch block CCM signals do not elicit a new contraction; rather, they influence the biology of the failing myocardium
New Heart Failure modalities: HIS Bundle Pacing & Cardiac Contractility Modulation from NAJEEB ULLAH SOFI
]]> 163 0 https://cdn.slidesharecdn.com/ss_thumbnails/newhfmodalitieshbpccm-200912052843-thumbnail.jpg?width=120&height=120&fit=bounds presentation Black http://activitystrea.ms/schema/1.0/post http://activitystrea.ms/schema/1.0/posted 0 https://public.slidesharecdn.com/v2/images/profile-picture.png https://cdn.slidesharecdn.com/ss_thumbnails/transcathetertherapiesforcongenitalheartdisease-201126150201-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/transcatheter-therapies-for-congenital-heart-disease-239503506/239503506 Transcatheter therapie... https://cdn.slidesharecdn.com/ss_thumbnails/cardiactroponinelevationinpatientswithoutaspecificdiagnosis-201014171101-thumbnail.jpg?width=320&height=320&fit=bounds NAJEEBULLAHSOFI/cardiac-troponin-elevation-in-patients-without-a-specific-diagnosis-238876629 Cardiac troponin eleva... https://cdn.slidesharecdn.com/ss_thumbnails/dualantiplatelettherapydurationbasedonischemicandbleedingrisksaftercoronarystenting-201014170648-thumbnail.jpg?width=320&height=320&fit=bounds NAJEEBULLAHSOFI/dual-antiplatelet-therapy-duration-based-on-ischemic-and-bleeding-risks-after-coronary-stenting Dual antiplatelet ther...