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�ݺ�ߣshows by User: parasuramwaddar1 / http://www.slideshare.net/images/logo.gif �ݺ�ߣshows by User: parasuramwaddar1 / Wed, 27 Apr 2022 06:01:44 GMT �ݺ�ߣShare feed for �ݺ�ߣshows by User: parasuramwaddar1 duct dependent heart lesions by Dr Parashuram Waddar 2021 ppt, for Pediatrics /slideshow/duct-dependent-heart-lesions-by-dr-parashuram-waddar-2021-ppt-for-pediatrics/251673838 ductdependentheartlesionsbydrparashuramwaddar2021-220427060144
Duct dependent heart lesions by Dr Parasuram Waddar 2021. Ductal anatomy and duct dependent circulation. Critical congenital heart disease, etiology, presentation, diagnosis and managment. scenario Incidence rate of CHD is 8-9/1000 live births, nearly 1.8- 2lacs children are born with CHD each year in India. 78.9- 81.4% Institutional deliveries. 20% of births in India occur at home, and the infant is likely to die before the critical, ductus-dependent CHD is diagnosed. Fortunately, the rate of hospital deliveries have increased due to several incentivized schemes by the Govt of India. Ductus-dependent CHD may still escape detection as babies are often discharged earlier. Predischarge screening of newborns by pulse oximetry, which may pick up these CHDs, is often not practiced, especially in rural & semi-urban centers. Lack of follow up care. Delay in referral results in poor outcomes as co-morbidities may have already set in. The risks of developing hypothermia and hypoglycemia during long, unsupervised transport further adds to the already serious condition of the infants with CHD. Of these, nearly 60,000 to 90,000 suffer from critical CHD requiring early intervention. Approximately 10% of present infant mortality in India may be accounted for by CHD alone. Lack of awareness & delay in diagnosis is biggest obstacle. Frontline health workers & primary caregivers are not sensitized to the problem of CHD. PDA- Short circuit channel between the pulmonary artery and the aorta in the fetus, which bypasses the lungs to distribute oxygen received through the placenta from the mother’s blood. It normally closes once the baby is born and the lungs inflate, separating the pulmonary and systemic circulations, thus converting parallel circulation into series. Functional closure of the ductus arteriosus occurs within 10-15 hours after birth in healthy infants born at term. This occurs by abrupt contraction of the medial smooth muscular wall of the ductus arteriosus. Multiple factors are responsible for the closure of ductus arteriosus. Ex- Po2, GA, PGE2 , etc. Increase in the partial pressure of oxygen (PO2) from 25mmHg(in utero) to 50mmHg after lung expansion is the strongest stimulus. Decrease in PGE2. Anatomic closure completes by end 2-3 weeks. Starting of ductus closure is the cause for deterioration in these lesions. Definition of DDHL- These are critical congenital heart disease (cCHD), in which the permeability of the ductus arteriosus is mandatory in order to maintain systemic and pulmonary perfusion after birth. These are most important d/d for newborns who are going to collapse in and around day3. Critical congenital heart disease (cCHD) is the most common reason for acute cardiac failure in the neonatal period Incidence- 25% of all CHDs, nearly 25% mortality in first year life. The distribution of cCHD differs from the distribution of CHDs in general. Left sided heart obstructions have the largest share w]]>
Duct dependent heart lesions by Dr Parasuram Waddar 2021. Ductal anatomy and duct dependent circulation. Critical congenital heart disease, etiology, presentation, diagnosis and managment. scenario Incidence rate of CHD is 8-9/1000 live births, nearly 1.8- 2lacs children are born with CHD each year in India. 78.9- 81.4% Institutional deliveries. 20% of births in India occur at home, and the infant is likely to die before the critical, ductus-dependent CHD is diagnosed. Fortunately, the rate of hospital deliveries have increased due to several incentivized schemes by the Govt of India. Ductus-dependent CHD may still escape detection as babies are often discharged earlier. Predischarge screening of newborns by pulse oximetry, which may pick up these CHDs, is often not practiced, especially in rural & semi-urban centers. Lack of follow up care. Delay in referral results in poor outcomes as co-morbidities may have already set in. The risks of developing hypothermia and hypoglycemia during long, unsupervised transport further adds to the already serious condition of the infants with CHD. Of these, nearly 60,000 to 90,000 suffer from critical CHD requiring early intervention. Approximately 10% of present infant mortality in India may be accounted for by CHD alone. Lack of awareness & delay in diagnosis is biggest obstacle. Frontline health workers & primary caregivers are not sensitized to the problem of CHD. PDA- Short circuit channel between the pulmonary artery and the aorta in the fetus, which bypasses the lungs to distribute oxygen received through the placenta from the mother’s blood. It normally closes once the baby is born and the lungs inflate, separating the pulmonary and systemic circulations, thus converting parallel circulation into series. Functional closure of the ductus arteriosus occurs within 10-15 hours after birth in healthy infants born at term. This occurs by abrupt contraction of the medial smooth muscular wall of the ductus arteriosus. Multiple factors are responsible for the closure of ductus arteriosus. Ex- Po2, GA, PGE2 , etc. Increase in the partial pressure of oxygen (PO2) from 25mmHg(in utero) to 50mmHg after lung expansion is the strongest stimulus. Decrease in PGE2. Anatomic closure completes by end 2-3 weeks. Starting of ductus closure is the cause for deterioration in these lesions. Definition of DDHL- These are critical congenital heart disease (cCHD), in which the permeability of the ductus arteriosus is mandatory in order to maintain systemic and pulmonary perfusion after birth. These are most important d/d for newborns who are going to collapse in and around day3. Critical congenital heart disease (cCHD) is the most common reason for acute cardiac failure in the neonatal period Incidence- 25% of all CHDs, nearly 25% mortality in first year life. The distribution of cCHD differs from the distribution of CHDs in general. Left sided heart obstructions have the largest share w]]> Wed, 27 Apr 2022 06:01:44 GMT /slideshow/duct-dependent-heart-lesions-by-dr-parashuram-waddar-2021-ppt-for-pediatrics/251673838 parasuramwaddar1@slideshare.net(parasuramwaddar1) duct dependent heart lesions by Dr Parashuram Waddar 2021 ppt, for Pediatrics parasuramwaddar1 Duct dependent heart lesions by Dr Parasuram Waddar 2021. Ductal anatomy and duct dependent circulation. Critical congenital heart disease, etiology, presentation, diagnosis and managment. scenario Incidence rate of CHD is 8-9/1000 live births, nearly 1.8- 2lacs children are born with CHD each year in India. 78.9- 81.4% Institutional deliveries. 20% of births in India occur at home, and the infant is likely to die before the critical, ductus-dependent CHD is diagnosed. Fortunately, the rate of hospital deliveries have increased due to several incentivized schemes by the Govt of India. Ductus-dependent CHD may still escape detection as babies are often discharged earlier. Predischarge screening of newborns by pulse oximetry, which may pick up these CHDs, is often not practiced, especially in rural & semi-urban centers. Lack of follow up care. Delay in referral results in poor outcomes as co-morbidities may have already set in. The risks of developing hypothermia and hypoglycemia during long, unsupervised transport further adds to the already serious condition of the infants with CHD. Of these, nearly 60,000 to 90,000 suffer from critical CHD requiring early intervention. Approximately 10% of present infant mortality in India may be accounted for by CHD alone. Lack of awareness & delay in diagnosis is biggest obstacle. Frontline health workers & primary caregivers are not sensitized to the problem of CHD. PDA- Short circuit channel between the pulmonary artery and the aorta in the fetus, which bypasses the lungs to distribute oxygen received through the placenta from the mother’s blood. It normally closes once the baby is born and the lungs inflate, separating the pulmonary and systemic circulations, thus converting parallel circulation into series. Functional closure of the ductus arteriosus occurs within 10-15 hours after birth in healthy infants born at term. This occurs by abrupt contraction of the medial smooth muscular wall of the ductus arteriosus. Multiple factors are responsible for the closure of ductus arteriosus. Ex- Po2, GA, PGE2 , etc. Increase in the partial pressure of oxygen (PO2) from 25mmHg(in utero) to 50mmHg after lung expansion is the strongest stimulus. Decrease in PGE2. Anatomic closure completes by end 2-3 weeks. Starting of ductus closure is the cause for deterioration in these lesions. Definition of DDHL- These are critical congenital heart disease (cCHD), in which the permeability of the ductus arteriosus is mandatory in order to maintain systemic and pulmonary perfusion after birth. These are most important d/d for newborns who are going to collapse in and around day3. Critical congenital heart disease (cCHD) is the most common reason for acute cardiac failure in the neonatal period Incidence- 25% of all CHDs, nearly 25% mortality in first year life. The distribution of cCHD differs from the distribution of CHDs in general. Left sided heart obstructions have the largest share w <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/ductdependentheartlesionsbydrparashuramwaddar2021-220427060144-thumbnail.jpg?width=120&height=120&fit=bounds" /><br> Duct dependent heart lesions by Dr Parasuram Waddar 2021. Ductal anatomy and duct dependent circulation. Critical congenital heart disease, etiology, presentation, diagnosis and managment. scenario Incidence rate of CHD is 8-9/1000 live births, nearly 1.8- 2lacs children are born with CHD each year in India. 78.9- 81.4% Institutional deliveries. 20% of births in India occur at home, and the infant is likely to die before the critical, ductus-dependent CHD is diagnosed. Fortunately, the rate of hospital deliveries have increased due to several incentivized schemes by the Govt of India. Ductus-dependent CHD may still escape detection as babies are often discharged earlier. Predischarge screening of newborns by pulse oximetry, which may pick up these CHDs, is often not practiced, especially in rural & semi-urban centers. Lack of follow up care. Delay in referral results in poor outcomes as co-morbidities may have already set in. The risks of developing hypothermia and hypoglycemia during long, unsupervised transport further adds to the already serious condition of the infants with CHD. Of these, nearly 60,000 to 90,000 suffer from critical CHD requiring early intervention. Approximately 10% of present infant mortality in India may be accounted for by CHD alone. Lack of awareness & delay in diagnosis is biggest obstacle. Frontline health workers & primary caregivers are not sensitized to the problem of CHD. PDA- Short circuit channel between the pulmonary artery and the aorta in the fetus, which bypasses the lungs to distribute oxygen received through the placenta from the mother’s blood. It normally closes once the baby is born and the lungs inflate, separating the pulmonary and systemic circulations, thus converting parallel circulation into series. Functional closure of the ductus arteriosus occurs within 10-15 hours after birth in healthy infants born at term. This occurs by abrupt contraction of the medial smooth muscular wall of the ductus arteriosus. Multiple factors are responsible for the closure of ductus arteriosus. Ex- Po2, GA, PGE2 , etc. Increase in the partial pressure of oxygen (PO2) from 25mmHg(in utero) to 50mmHg after lung expansion is the strongest stimulus. Decrease in PGE2. Anatomic closure completes by end 2-3 weeks. Starting of ductus closure is the cause for deterioration in these lesions. Definition of DDHL- These are critical congenital heart disease (cCHD), in which the permeability of the ductus arteriosus is mandatory in order to maintain systemic and pulmonary perfusion after birth. These are most important d/d for newborns who are going to collapse in and around day3. Critical congenital heart disease (cCHD) is the most common reason for acute cardiac failure in the neonatal period Incidence- 25% of all CHDs, nearly 25% mortality in first year life. The distribution of cCHD differs from the distribution of CHDs in general. Left sided heart obstructions have the largest share w

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