�ݺ�ߣshows by User: rezahei

�ݺ�ߣshows by User: rezahei / http://www.slideshare.net/images/logo.gif �ݺ�ߣshows by User: rezahei / Mon, 05 Jun 2017 07:16:41 GMT �ݺ�ߣShare feed for �ݺ�ߣshows by User: rezahei Autonomic system and Autonomic Pharmacology /slideshow/autonomic-system-and-autonomic-pharmacology/76647596 autonomicsystem-170605071641
Autonomic Nervous System & Autonomic Pharmacology Sympathetic: ADRENERGIC Central: EPINEPHRINE Peripheral: NOREPINEPHRINE Parasympathetic: CHOLINERGIC Acetylcholine ]]>
Autonomic Nervous System & Autonomic Pharmacology Sympathetic: ADRENERGIC Central: EPINEPHRINE Peripheral: NOREPINEPHRINE Parasympathetic: CHOLINERGIC Acetylcholine ]]> Mon, 05 Jun 2017 07:16:41 GMT /slideshow/autonomic-system-and-autonomic-pharmacology/76647596 rezahei@slideshare.net(rezahei) Autonomic system and Autonomic Pharmacology rezahei Autonomic Nervous System & Autonomic Pharmacology Sympathetic: ADRENERGIC Central: EPINEPHRINE Peripheral: NOREPINEPHRINE Parasympathetic: CHOLINERGIC Acetylcholine <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/autonomicsystem-170605071641-thumbnail.jpg?width=120&height=120&fit=bounds" /> Autonomic Nervous System & Autonomic Pharmacology Sympathetic: ADRENERGIC Central: EPINEPHRINE Peripheral: NOREPINEPHRINE Parasympathetic: CHOLINERGIC Acetylcholine

Autonomic system and Autonomic Pharmacology from Reza Heidari

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0 Cardiovascular pharmacology /slideshow/cardiovascular-pharmacology-76647530/76647530 cardiovascularpharmacology-170605071338
Cardiovascular pharmacology Cardiovascular (=Circulatory) system – heart and blood vessels Arteries – transport blood to tissues Capillaries – sites of exchange, fluid O2, CO2, nutrients etc. Venules – collect blood from capillaries Veins – transport blood back to heart Blood moves within vessels – higher pressure to lower pressure Resistance to flow depends on vessel diameter, length and viscosity of blood ]]>
Cardiovascular pharmacology Cardiovascular (=Circulatory) system – heart and blood vessels Arteries – transport blood to tissues Capillaries – sites of exchange, fluid O2, CO2, nutrients etc. Venules – collect blood from capillaries Veins – transport blood back to heart Blood moves within vessels – higher pressure to lower pressure Resistance to flow depends on vessel diameter, length and viscosity of blood ]]> Mon, 05 Jun 2017 07:13:38 GMT /slideshow/cardiovascular-pharmacology-76647530/76647530 rezahei@slideshare.net(rezahei) Cardiovascular pharmacology rezahei Cardiovascular pharmacology Cardiovascular (=Circulatory) system – heart and blood vessels Arteries – transport blood to tissues Capillaries – sites of exchange, fluid O2, CO2, nutrients etc. Venules – collect blood from capillaries Veins – transport blood back to heart Blood moves within vessels – higher pressure to lower pressure Resistance to flow depends on vessel diameter, length and viscosity of blood <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cardiovascularpharmacology-170605071338-thumbnail.jpg?width=120&height=120&fit=bounds" /> Cardiovascular pharmacology Cardiovascular (=Circulatory) system – heart and blood vessels Arteries – transport blood to tissues Capillaries – sites of exchange, fluid O2, CO2, nutrients etc. Venules – collect blood from capillaries Veins – transport blood back to heart Blood moves within vessels – higher pressure to lower pressure Resistance to flow depends on vessel diameter, length and viscosity of blood

Cardiovascular pharmacology from Reza Heidari

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0 Mechanisms of Hepatotoxicity /slideshow/mechanisms-of-hepatotoxicity/76647473 mechanismsofhepatotoxicity-170605071048
The liver plays a key role in detoxifying harmful substances that you may eat, drink, inhale or rub on your skin. Toxic hepatitis is liver inflammation that occurs when your liver is damaged by toxic chemicals, drugs or certain poisonous mushrooms. ]]>
The liver plays a key role in detoxifying harmful substances that you may eat, drink, inhale or rub on your skin. Toxic hepatitis is liver inflammation that occurs when your liver is damaged by toxic chemicals, drugs or certain poisonous mushrooms. ]]> Mon, 05 Jun 2017 07:10:48 GMT /slideshow/mechanisms-of-hepatotoxicity/76647473 rezahei@slideshare.net(rezahei) Mechanisms of Hepatotoxicity rezahei The liver plays a key role in detoxifying harmful substances that you may eat, drink, inhale or rub on your skin. Toxic hepatitis is liver inflammation that occurs when your liver is damaged by toxic chemicals, drugs or certain poisonous mushrooms. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/mechanismsofhepatotoxicity-170605071048-thumbnail.jpg?width=120&height=120&fit=bounds" /> The liver plays a key role in detoxifying harmful substances that you may eat, drink, inhale or rub on your skin. Toxic hepatitis is liver inflammation that occurs when your liver is damaged by toxic chemicals, drugs or certain poisonous mushrooms.

Mechanisms of Hepatotoxicity from Reza Heidari

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0 Eicosanoids an NSAID Drugs /slideshow/eicosanoids-an-nsaid-drugs/76647266 eicosanoids-drheidari-170605070019
The eicosanoids are oxygenation products of polyunsaturated long-chain fatty acids. They are ubiquitous in the animal kingdom and are also found—together with their precursors—in a variety of plants. They constitute a very large family of compounds that are highly potent and display an extraordinarily wide spectrum of biologic activity. Because of their biologic activity, the eicosanoids, their specific receptor antagonists and enzyme inhibitors, and their plant and fish oil precursors have great therapeutic potential.]]>
The eicosanoids are oxygenation products of polyunsaturated long-chain fatty acids. They are ubiquitous in the animal kingdom and are also found—together with their precursors—in a variety of plants. They constitute a very large family of compounds that are highly potent and display an extraordinarily wide spectrum of biologic activity. Because of their biologic activity, the eicosanoids, their specific receptor antagonists and enzyme inhibitors, and their plant and fish oil precursors have great therapeutic potential.]]> Mon, 05 Jun 2017 07:00:19 GMT /slideshow/eicosanoids-an-nsaid-drugs/76647266 rezahei@slideshare.net(rezahei) Eicosanoids an NSAID Drugs rezahei The eicosanoids are oxygenation products of polyunsaturated long-chain fatty acids. They are ubiquitous in the animal kingdom and are also found—together with their precursors—in a variety of plants. They constitute a very large family of compounds that are highly potent and display an extraordinarily wide spectrum of biologic activity. Because of their biologic activity, the eicosanoids, their specific receptor antagonists and enzyme inhibitors, and their plant and fish oil precursors have great therapeutic potential. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/eicosanoids-drheidari-170605070019-thumbnail.jpg?width=120&height=120&fit=bounds" /> The eicosanoids are oxygenation products of polyunsaturated long-chain fatty acids. They are ubiquitous in the animal kingdom and are also found—together with their precursors—in a variety of plants. They constitute a very large family of compounds that are highly potent and display an extraordinarily wide spectrum of biologic activity. Because of their biologic activity, the eicosanoids, their specific receptor antagonists and enzyme inhibitors, and their plant and fish oil precursors have great therapeutic potential.

Eicosanoids an NSAID Drugs from Reza Heidari

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0 Anticoagulants pharmacology /slideshow/anticoagulants-pharmacology/76647148 anticoagulants-pharmacology-170605065437
Katzung Anticoagulants Hemostasis refers to the finely regulated dynamic process of maintaining the fluidity of the blood, repairing vascular injury, and limiting blood loss while avoiding vessel occlusion (thrombosis) and inadequate perfusion of vital organs. Either extreme excessive bleeding or thrombosis—represents a breakdown of the hemostatic mechanism. Common causes of dysregulated hemostasis include hereditary or acquired defects in the clotting mechanism and secondary effects of infection or cancer. The drugs used to inhibit thrombosis and to limit abnormal bleeding are the subjects of this chapter. ]]>
Katzung Anticoagulants Hemostasis refers to the finely regulated dynamic process of maintaining the fluidity of the blood, repairing vascular injury, and limiting blood loss while avoiding vessel occlusion (thrombosis) and inadequate perfusion of vital organs. Either extreme excessive bleeding or thrombosis—represents a breakdown of the hemostatic mechanism. Common causes of dysregulated hemostasis include hereditary or acquired defects in the clotting mechanism and secondary effects of infection or cancer. The drugs used to inhibit thrombosis and to limit abnormal bleeding are the subjects of this chapter. ]]> Mon, 05 Jun 2017 06:54:37 GMT /slideshow/anticoagulants-pharmacology/76647148 rezahei@slideshare.net(rezahei) Anticoagulants pharmacology rezahei Katzung Anticoagulants Hemostasis refers to the finely regulated dynamic process of maintaining the fluidity of the blood, repairing vascular injury, and limiting blood loss while avoiding vessel occlusion (thrombosis) and inadequate perfusion of vital organs. Either extreme excessive bleeding or thrombosis—represents a breakdown of the hemostatic mechanism. Common causes of dysregulated hemostasis include hereditary or acquired defects in the clotting mechanism and secondary effects of infection or cancer. The drugs used to inhibit thrombosis and to limit abnormal bleeding are the subjects of this chapter. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/anticoagulants-pharmacology-170605065437-thumbnail.jpg?width=120&height=120&fit=bounds" /> Katzung Anticoagulants Hemostasis refers to the finely regulated dynamic process of maintaining the fluidity of the blood, repairing vascular injury, and limiting blood loss while avoiding vessel occlusion (thrombosis) and inadequate perfusion of vital organs. Either extreme excessive bleeding or thrombosis—represents a breakdown of the hemostatic mechanism. Common causes of dysregulated hemostasis include hereditary or acquired defects in the clotting mechanism and secondary effects of infection or cancer. The drugs used to inhibit thrombosis and to limit abnormal bleeding are the subjects of this chapter.

Anticoagulants pharmacology from Reza Heidari

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0 Diuretics /slideshow/diuretics-72238716/72238716 diuretics-170216173843
Diuretics Pharmacology Katzung Abnormalities in fluid volume and electrolyte composition are common and important clinical disorders. Drugs that block specific transport functions of the renal tubules are valuable clinical tools in the treatment of these disorders. Although various agents that increase urine volume (diuretics) have been described since antiquity, it was not until 1937 that carbonic anhydrase inhibitors were first described and not until 1957 that a much more useful and powerful diuretic agent (chlorothiazide) became available. Technically, a “diuretic” is an agent that increases urine volume, whereas a “natriuretic” causes an increase in renal sodium excretion and an “aquaretic” increases excretion of solute-free water. Because natriuretics almost always also increase water excretion, they are usually called diuretics. Osmotic diuretics and antidiuretic hormone antagonists (see Agents That Alter Water Excretion) are aquaretics that are not directly natriuretic.]]>
Diuretics Pharmacology Katzung Abnormalities in fluid volume and electrolyte composition are common and important clinical disorders. Drugs that block specific transport functions of the renal tubules are valuable clinical tools in the treatment of these disorders. Although various agents that increase urine volume (diuretics) have been described since antiquity, it was not until 1937 that carbonic anhydrase inhibitors were first described and not until 1957 that a much more useful and powerful diuretic agent (chlorothiazide) became available. Technically, a “diuretic” is an agent that increases urine volume, whereas a “natriuretic” causes an increase in renal sodium excretion and an “aquaretic” increases excretion of solute-free water. Because natriuretics almost always also increase water excretion, they are usually called diuretics. Osmotic diuretics and antidiuretic hormone antagonists (see Agents That Alter Water Excretion) are aquaretics that are not directly natriuretic.]]> Thu, 16 Feb 2017 17:38:42 GMT /slideshow/diuretics-72238716/72238716 rezahei@slideshare.net(rezahei) Diuretics rezahei Diuretics Pharmacology Katzung Abnormalities in fluid volume and electrolyte composition are common and important clinical disorders. Drugs that block specific transport functions of the renal tubules are valuable clinical tools in the treatment of these disorders. Although various agents that increase urine volume (diuretics) have been described since antiquity, it was not until 1937 that carbonic anhydrase inhibitors were first described and not until 1957 that a much more useful and powerful diuretic agent (chlorothiazide) became available. Technically, a “diuretic” is an agent that increases urine volume, whereas a “natriuretic” causes an increase in renal sodium excretion and an “aquaretic” increases excretion of solute-free water. Because natriuretics almost always also increase water excretion, they are usually called diuretics. Osmotic diuretics and antidiuretic hormone antagonists (see Agents That Alter Water Excretion) are aquaretics that are not directly natriuretic. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/diuretics-170216173843-thumbnail.jpg?width=120&height=120&fit=bounds" /> Diuretics Pharmacology Katzung Abnormalities in fluid volume and electrolyte composition are common and important clinical disorders. Drugs that block specific transport functions of the renal tubules are valuable clinical tools in the treatment of these disorders. Although various agents that increase urine volume (diuretics) have been described since antiquity, it was not until 1937 that carbonic anhydrase inhibitors were first described and not until 1957 that a much more useful and powerful diuretic agent (chlorothiazide) became available. Technically, a “diuretic” is an agent that increases urine volume, whereas a “natriuretic” causes an increase in renal sodium excretion and an “aquaretic” increases excretion of solute-free water. Because natriuretics almost always also increase water excretion, they are usually called diuretics. Osmotic diuretics and antidiuretic hormone antagonists (see Agents That Alter Water Excretion) are aquaretics that are not directly natriuretic.

Diuretics from Reza Heidari

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0 https://cdn.slidesharecdn.com/profile-photo-rezahei-48x48.jpg?cb=1717470798 Toxicological Research http://www.ncbi.nlm.nih.gov/pubmed?cmd=historysearch&querykey=4 https://cdn.slidesharecdn.com/ss_thumbnails/autonomicsystem-170605071641-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/autonomic-system-and-autonomic-pharmacology/76647596 Autonomic system and A... https://cdn.slidesharecdn.com/ss_thumbnails/cardiovascularpharmacology-170605071338-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/cardiovascular-pharmacology-76647530/76647530 Cardiovascular pharmac... https://cdn.slidesharecdn.com/ss_thumbnails/mechanismsofhepatotoxicity-170605071048-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/mechanisms-of-hepatotoxicity/76647473 Mechanisms of Hepatoto...