端端舝shows by User: romissaasaleh / http://www.slideshare.net/images/logo.gif 端端舝shows by User: romissaasaleh / Fri, 16 Aug 2024 12:21:14 GMT 端端舝Share feed for 端端舝shows by User: romissaasaleh Different techniques and Methods of DNA and RNA Isolation.pdf /slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pdf/271053979 dna-240816122115-8e61e69d
Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. ]]>

Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. ]]>
Fri, 16 Aug 2024 12:21:14 GMT /slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pdf/271053979 romissaasaleh@slideshare.net(romissaasaleh) Different techniques and Methods of DNA and RNA Isolation.pdf romissaasaleh Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/dna-240816122115-8e61e69d-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool.
Different techniques and Methods of DNA and RNA Isolation.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Different techniques and Methods of DNA and RNA Isolation.pptx /slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pptx/271053932 dna-240816121750-223ca4d5
Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. ]]>

Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. ]]>
Fri, 16 Aug 2024 12:17:50 GMT /slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pptx/271053932 romissaasaleh@slideshare.net(romissaasaleh) Different techniques and Methods of DNA and RNA Isolation.pptx romissaasaleh Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/dna-240816121750-223ca4d5-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Nucleic acids (DNA or RNA) are the hereditary molecules that harbor biological instructions for making each species unique. This genetic material is passed on from one generation to another at the time of reproduction. Each organism contains multiple molecules of DNA per cell. In eukaryotes, DNA is found localized in a particular region of the cell known as the nucleus. This DNA is packaged tightly in the form of chromosomes. The complete set of nuclear DNA of an organism is called a genome. Extraction or isolation of DNA is an initial step for various molecular biology, genetics, and recombinant DNA technology applications. It is one of the basic steps to study a genome or identify gene sequences in a heterogeneous gene pool.
Different techniques and Methods of DNA and RNA Isolation.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pptx /slideshow/tumor-microenvironment-underlying-secretes-and-its-impact-on-cancer-progression-pptx/270316886 tumor-240718120254-c893d341
The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. ]]>

The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. ]]>
Thu, 18 Jul 2024 12:02:53 GMT /slideshow/tumor-microenvironment-underlying-secretes-and-its-impact-on-cancer-progression-pptx/270316886 romissaasaleh@slideshare.net(romissaasaleh) Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pptx romissaasaleh The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/tumor-240718120254-c893d341-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system.
Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pdf /slideshow/tumor-microenvironment-underlying-secretes-and-its-impact-on-cancer-progression-pdf/270316846 tumor-240718120026-f0c1a019
The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. ]]>

The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. ]]>
Thu, 18 Jul 2024 12:00:26 GMT /slideshow/tumor-microenvironment-underlying-secretes-and-its-impact-on-cancer-progression-pdf/270316846 romissaasaleh@slideshare.net(romissaasaleh) Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pdf romissaasaleh The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/tumor-240718120026-f0c1a019-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> The TME refers to the cellular environment in which tumors or cancer stem cells exist. Cancer stem cells are cells in a tumor with the abilities to self-renew and drive tumorigenesis [8]. Previous studies have isolated unique cancer stem cells in samples from patients with breast, hematopoietic, colon, lung, and brain cancers [1,2,5]. These cells help improve the understanding of the TME [8,9], but pose significant challenges in the diagnosis and management of cancer. The TME encompasses the surrounding immune cells, blood vessels, extracellular matrix (ECM), fibroblasts, lymphocytes, bone marrow-derived inflammatory cells, and signaling molecules [9,10]. Interactions between malignant and nonmalignant cells create a TME that affects cancer development and progression [4,5]. The nonmalignant cells in the TME often play a protumorigenic function at all phases of carcinogenesis by stimulating uncontrolled cell proliferation [6每8]. In contrast, malignant cells invade healthy tissues and spread to other body parts through the lymphatic or circulatory system.
Tumor microenvironment: Underlying Secretes and its impact on cancer progression.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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cell cycle: Phases, control and analysis.pptx /slideshow/cell-cycle-phases-control-and-analysis-pptx/270213724 cellcycle-240712185026-7bb90294
A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete, and interphase takes up the remaining 23 hours ]]>

A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete, and interphase takes up the remaining 23 hours ]]>
Fri, 12 Jul 2024 18:50:26 GMT /slideshow/cell-cycle-phases-control-and-analysis-pptx/270213724 romissaasaleh@slideshare.net(romissaasaleh) cell cycle: Phases, control and analysis.pptx romissaasaleh A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete, and interphase takes up the remaining 23 hours <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cellcycle-240712185026-7bb90294-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete, and interphase takes up the remaining 23 hours
cell cycle: Phases, control and analysis.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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cell cycle: Phases, control and analysis.pdf /slideshow/cell-cycle-phases-control-and-analysis-pdf/270213683 cellcycle-240712184741-b9d751fb
A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete and interphase takes up the remaining 23 hours ]]>

A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete and interphase takes up the remaining 23 hours ]]>
Fri, 12 Jul 2024 18:47:41 GMT /slideshow/cell-cycle-phases-control-and-analysis-pdf/270213683 romissaasaleh@slideshare.net(romissaasaleh) cell cycle: Phases, control and analysis.pdf romissaasaleh A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete and interphase takes up the remaining 23 hours <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cellcycle-240712184741-b9d751fb-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> A typical eukaryotic cell cycle contains several distinct phases, which progress in an orderly fashion〞a phase cannot commence without completion of the previous one. The four phases of the cell cycle are G1 (G for gap), S (synthesis), G2, and M (mitosis) phases (Fig. 8.1). The G1, S, and G2 phases collectively make up the interphase. The DNA content of a cell in the G1 phase is 2N (N is the number of chromosomes), also known as diploid, whereas the DNA content of a cell in the G2 phase is 4N (tetraploid). The DNA content of a cell in the S phase varies between 2N and 4N, depending on the stage of replication. The M phase is in turn comprised of two processes: mitosis, in which the cell*s chromosomes are equally divided between the two daughter cells, and cytokinesis (or cell division), in which the cytoplasm of the cell divides in half to form two distinct daughter cells. Typically, the amount of time required for a single-cell cycle in actively proliferating human cells in culture is 24 hours. Of these, the M phase takes approximately 1 hour to complete and interphase takes up the remaining 23 hours
cell cycle: Phases, control and analysis.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pdf /slideshow/magnetic-resonance-imaging-basic-concepts-and-its-applications-in-dental-field-pdf/270119817 mri-240708085839-55e6bb8f
MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. ]]>

MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. ]]>
Mon, 08 Jul 2024 08:58:39 GMT /slideshow/magnetic-resonance-imaging-basic-concepts-and-its-applications-in-dental-field-pdf/270119817 romissaasaleh@slideshare.net(romissaasaleh) Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pdf romissaasaleh MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/mri-240708085839-55e6bb8f-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer.
Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pptx /slideshow/magnetic-resonance-imaging-basic-concepts-and-its-applications-in-dental-field-pptx/270119779 mri-240708085622-42814609
MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. ]]>

MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. ]]>
Mon, 08 Jul 2024 08:56:22 GMT /slideshow/magnetic-resonance-imaging-basic-concepts-and-its-applications-in-dental-field-pptx/270119779 romissaasaleh@slideshare.net(romissaasaleh) Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pptx romissaasaleh MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/mri-240708085622-42814609-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> MRI is a non-invasive method of mapping the internal structure and certain aspects of function within the body. It uses nonionizing electromagnetic radiation and appears to be without exposure-related hazard. It employs radio frequency (RF) radiation in the presence of carefully controlled magnetic fields in order to produce high quality cross-sectional images of the body in any plane. The MR Image is constructed by placing the patient inside a large magnet, which induces a relatively strong External magnetic field. This causes the nuclei of many atoms in the body, including Hydrogen, to align them with the magnetic field and later application of RF signal, Energy is released from the body, detected and used to construct the MR image by Computer.
Magnetic Resonance Imaging: Basic Concepts and its applications in Dental Field.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Cell biology: basics and knowledge study.pdf /slideshow/cell-biology-basics-and-knowledge-study-pdf/270019803 cellbiology-240702124028-d8fdb024
A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. ]]>

A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. ]]>
Tue, 02 Jul 2024 12:40:28 GMT /slideshow/cell-biology-basics-and-knowledge-study-pdf/270019803 romissaasaleh@slideshare.net(romissaasaleh) Cell biology: basics and knowledge study.pdf romissaasaleh A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cellbiology-240702124028-d8fdb024-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell&#39;s boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers.
Cell biology: basics and knowledge study.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Cell biology: Basics and knowledge study.pptx /slideshow/cell-biology-basics-and-knowledge-study-pptx/270019692 cellbiology-240702123549-75806c45
A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. ]]>

A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. ]]>
Tue, 02 Jul 2024 12:35:49 GMT /slideshow/cell-biology-basics-and-knowledge-study-pptx/270019692 romissaasaleh@slideshare.net(romissaasaleh) Cell biology: Basics and knowledge study.pptx romissaasaleh A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell's boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cellbiology-240702123549-75806c45-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> A cell consists of three parts: the?cell membrane, the?nucleus, and, between the two, the?cytoplasm. Within the cytoplasm lie intricate arrangements of fine fibers and hundreds or even thousands of miniscule but distinct structures called organelles. Every cell in the body is enclosed by a cell (Plasma)?membrane. The cell membrane separates the material outside the cell, extracellular, from the material inside the cell,?intracellular. It maintains the integrity of a cell and controls passage of materials into and out of the cell. All materials within a cell must have access to the cell membrane (the cell&#39;s boundary) for the needed exchange. The cell membrane is a double layer of phospholipid molecules.?Proteins?in the cell membrane provide structural support, form channels for passage of materials, act as?receptor?sites, function as?carrier?molecules, and provide identification markers.
Cell biology: Basics and knowledge study.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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underlining mechanisms of autoimmunity.pptx /slideshow/underlining-mechanisms-of-autoimmunity-pptx/269923547 autoimmunity-240627110929-1821cf86
Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity ]]>

Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity ]]>
Thu, 27 Jun 2024 11:09:29 GMT /slideshow/underlining-mechanisms-of-autoimmunity-pptx/269923547 romissaasaleh@slideshare.net(romissaasaleh) underlining mechanisms of autoimmunity.pptx romissaasaleh Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/autoimmunity-240627110929-1821cf86-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity
underlining mechanisms of autoimmunity.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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underlining mechanisms of autoimmunity.pdf /slideshow/underlining-mechanisms-of-autoimmunity-pdf/269923483 autoimmunity-240627110600-c073ba63
Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity ]]>

Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity ]]>
Thu, 27 Jun 2024 11:06:00 GMT /slideshow/underlining-mechanisms-of-autoimmunity-pdf/269923483 romissaasaleh@slideshare.net(romissaasaleh) underlining mechanisms of autoimmunity.pdf romissaasaleh Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/autoimmunity-240627110600-c073ba63-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Autoimmune reactions reflect an imbalance between effector and regulatory immune responses, typically develop through stages of initiation and propagation, and often show phases of resolution (indicated by clinical remissions) and exacerbations (indicated by symptomatic flares). The fundamental underlying mechanism of autoimmunity is defective elimination and/or control of self-reactive lymphocytes. Studies in humans and experimental animal models are revealing the genetic and environmental factors that contribute to autoimmunity
underlining mechanisms of autoimmunity.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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sex hormones and periodontium &its impact on oral diseases.pptx /slideshow/sex-hormones-and-periodontium-its-impact-on-oral-diseases-pptx/269748742 sexhormones-240618173005-51b147bd
Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 ]]>

Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 ]]>
Tue, 18 Jun 2024 17:30:04 GMT /slideshow/sex-hormones-and-periodontium-its-impact-on-oral-diseases-pptx/269748742 romissaasaleh@slideshare.net(romissaasaleh) sex hormones and periodontium &its impact on oral diseases.pptx romissaasaleh Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/sexhormones-240618173005-51b147bd-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1
sex hormones and periodontium &its impact on oral diseases.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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sex hormones and periodontium and its impact on oral diseasesa.pdf /slideshow/sex-hormones-and-periodontium-and-its-impact-on-oral-diseasesa-pdf/269748635 sexhormones-240618172247-b6f26d4e
Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 ]]>

Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 ]]>
Tue, 18 Jun 2024 17:22:46 GMT /slideshow/sex-hormones-and-periodontium-and-its-impact-on-oral-diseasesa-pdf/269748635 romissaasaleh@slideshare.net(romissaasaleh) sex hormones and periodontium and its impact on oral diseasesa.pdf romissaasaleh Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1 <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/sexhormones-240618172247-b6f26d4e-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Hormones are specific regulatory molecules that have potent effects on the major determinants of the development and the integrity of the skeleton and oral cavity including periodontal tissues. It is clear that periodontal manifestations occur when an imbalance of these steroid hormones take place. The homeostasis of the periodontium involves complex multifactorial relationships, in which the endocrine system plays an important role. (1) Hormones are specific regulatory molecules that modulate reproduction, growth and development and the maintenance of internal environments as well as energy production, utilization and storage.1
sex hormones and periodontium and its impact on oral diseasesa.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pptx /slideshow/cancer-underlying-pathophysiological-mechanisms-and-novel-treatment-approaches-pptx/269665870 cancer-240613123853-c3b95b32
the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). ]]>

the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). ]]>
Thu, 13 Jun 2024 12:38:53 GMT /slideshow/cancer-underlying-pathophysiological-mechanisms-and-novel-treatment-approaches-pptx/269665870 romissaasaleh@slideshare.net(romissaasaleh) Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pptx romissaasaleh the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cancer-240613123853-c3b95b32-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008).
Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pdf /slideshow/cancer-underlying-pathophysiological-mechanisms-and-novel-treatment-approaches-pdf/269665809 cancer-240613123348-ca99768d
the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). ]]>

the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). ]]>
Thu, 13 Jun 2024 12:33:47 GMT /slideshow/cancer-underlying-pathophysiological-mechanisms-and-novel-treatment-approaches-pdf/269665809 romissaasaleh@slideshare.net(romissaasaleh) Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pdf romissaasaleh the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008). <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/cancer-240613123348-ca99768d-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> the cancer secretome has been described as including the extracellular matrix components and all the proteins that are released from a given type of cancer cells, such as growth factors, cytokines, adhesion molecules, shed receptors and proteases, and reflects the functionality of this cell type at a given time point (Kulasingam and Diamandis, 2008). Therefore, the cancer secretome includes proteins released from cancer cells, either with classical or non-classical secretory pathways, and corresponds to an important class of proteins that can act both locally and systemically (Kulasingam and Diamandis, 2008). Theoretically, the cancer secretome includes all the proteins that can be identified in the interstitial fluid of the tumor mass in vivo (Celis et al., 2005), however it is better conceptualized as the group of proteins identified with mass spectrometry in cancer cell line conditioned media (CM) in in vitro studies (Kulasingam and Diamandis, 2008).
Cancer: Underlying pathophysiological mechanisms and novel treatment approaches.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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blood每brain barrier and uses of blood components as diagnostic tools.pptx /slideshow/blood-brain-barrier-and-uses-of-blood-components-as-diagnostic-tools-pptx/269599452 bloodbrainbarriar-240610091458-575ce06c
. HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS ]]>

. HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS ]]>
Mon, 10 Jun 2024 09:14:58 GMT /slideshow/blood-brain-barrier-and-uses-of-blood-components-as-diagnostic-tools-pptx/269599452 romissaasaleh@slideshare.net(romissaasaleh) blood每brain barrier and uses of blood components as diagnostic tools.pptx romissaasaleh . HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/bloodbrainbarriar-240610091458-575ce06c-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> . HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS
blood碉捯ain barrier and uses of blood components as diagnostic tools.pptx from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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blood每brain barrier and uses of blood components as diagnostic tools .pdf /slideshow/blood-brain-barrier-and-uses-of-blood-components-as-diagnostic-tools-pdf/269599369 bloodbrainbarrier-240610090959-66000ccc
. HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS ]]>

. HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS ]]>
Mon, 10 Jun 2024 09:09:58 GMT /slideshow/blood-brain-barrier-and-uses-of-blood-components-as-diagnostic-tools-pdf/269599369 romissaasaleh@slideshare.net(romissaasaleh) blood每brain barrier and uses of blood components as diagnostic tools .pdf romissaasaleh . HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/bloodbrainbarrier-240610090959-66000ccc-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> . HIV-1-infected monocyte每macrophages traverse the BBB and enter the CNS throughout the course of HIV-1 disease. Once in the brain, both free virus and virus-infected cells are able to infect neighboring resident microglia and astrocytes and possibly other cell types. HIV-1-infected cells in both the periphery and the CNS give rise to elevated levels of viral proteins, including gp120, Tat, and Nef, and of host inflammatory mediators such as cytokines and chemokines. It has been shown that the viral proteins may act alone or in concert with host cytokines and chemokines, affecting the integrity of the BBB. The pathological end point of these interactions may facilitate a positive feedback loop resulting in increased penetration of HIV into the CNS
blood碉捯ain barrier and uses of blood components as diagnostic tools .pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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gingival hyperplasia :underlying Pathological mechanisms.pdf /slideshow/gingival-hyperplasia-underlying-pathological-mechanisms-pdf/269539425 gingivalhyperplasia-240606093150-8e8c58e7
Inflammatory gingival hyperplasia is an inflammatory restraint to local irritant correlating with the gingiva; the irritant could be microbial like plaque and calculus. Clinically present as deep red or bluish, considerably friable and fine with smooth glossy surface and commonly bleed easily [1]. These conditions are presented with the epithelial to mesenchymal transition (EMT), where the basal lamina show disruptions and epithelial cells migrate into connective tissue and change their phenotypes to fibroblast-like cells [12]. ]]>

Inflammatory gingival hyperplasia is an inflammatory restraint to local irritant correlating with the gingiva; the irritant could be microbial like plaque and calculus. Clinically present as deep red or bluish, considerably friable and fine with smooth glossy surface and commonly bleed easily [1]. These conditions are presented with the epithelial to mesenchymal transition (EMT), where the basal lamina show disruptions and epithelial cells migrate into connective tissue and change their phenotypes to fibroblast-like cells [12]. ]]>
Thu, 06 Jun 2024 09:31:50 GMT /slideshow/gingival-hyperplasia-underlying-pathological-mechanisms-pdf/269539425 romissaasaleh@slideshare.net(romissaasaleh) gingival hyperplasia :underlying Pathological mechanisms.pdf romissaasaleh Inflammatory gingival hyperplasia is an inflammatory restraint to local irritant correlating with the gingiva; the irritant could be microbial like plaque and calculus. Clinically present as deep red or bluish, considerably friable and fine with smooth glossy surface and commonly bleed easily [1]. These conditions are presented with the epithelial to mesenchymal transition (EMT), where the basal lamina show disruptions and epithelial cells migrate into connective tissue and change their phenotypes to fibroblast-like cells [12]. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/gingivalhyperplasia-240606093150-8e8c58e7-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Inflammatory gingival hyperplasia is an inflammatory restraint to local irritant correlating with the gingiva; the irritant could be microbial like plaque and calculus. Clinically present as deep red or bluish, considerably friable and fine with smooth glossy surface and commonly bleed easily [1]. These conditions are presented with the epithelial to mesenchymal transition (EMT), where the basal lamina show disruptions and epithelial cells migrate into connective tissue and change their phenotypes to fibroblast-like cells [12].
gingival hyperplasia :underlying Pathological mechanisms.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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cancer stem cell : challenge in cancer treatment.pdf /slideshow/cancer-stem-cell-challenge-in-cancer-treatment-pdf/269453989 oralcancerandcancerstemcell1-240601114544-829bca0a
Many evidences that CSCs also play a central role in the pathogenesis and progression of carcinomas of the head and neck (HNSCC), including OSCC,have been found. Early tissue culture studies showed that only a subpopulation of OSCC cells can form expanding tumor colonies, suggesting that human OSCC may contain some form of stem cells and it was subsequently shown that only a small subpopulation of the cells in OSCC corresponds to tumor-initiating cells. These finding are in accordance with CSCs concept (17,34) that the tumor mass is a mixture of (a) CSCs dividing themselves to feed the tumor's growth, b) transient amplifying cells that divide themselves a few times before maturing into (c) differentiated tumor cells that do not contribute to tumor growth (4). The isolation of CSCs from oral cancers has mainly been performed with the CD44 marker that was initially used to isolate breast cancer CSCs. ]]>

Many evidences that CSCs also play a central role in the pathogenesis and progression of carcinomas of the head and neck (HNSCC), including OSCC,have been found. Early tissue culture studies showed that only a subpopulation of OSCC cells can form expanding tumor colonies, suggesting that human OSCC may contain some form of stem cells and it was subsequently shown that only a small subpopulation of the cells in OSCC corresponds to tumor-initiating cells. These finding are in accordance with CSCs concept (17,34) that the tumor mass is a mixture of (a) CSCs dividing themselves to feed the tumor's growth, b) transient amplifying cells that divide themselves a few times before maturing into (c) differentiated tumor cells that do not contribute to tumor growth (4). The isolation of CSCs from oral cancers has mainly been performed with the CD44 marker that was initially used to isolate breast cancer CSCs. ]]>
Sat, 01 Jun 2024 11:45:44 GMT /slideshow/cancer-stem-cell-challenge-in-cancer-treatment-pdf/269453989 romissaasaleh@slideshare.net(romissaasaleh) cancer stem cell : challenge in cancer treatment.pdf romissaasaleh Many evidences that CSCs also play a central role in the pathogenesis and progression of carcinomas of the head and neck (HNSCC), including OSCC,have been found. Early tissue culture studies showed that only a subpopulation of OSCC cells can form expanding tumor colonies, suggesting that human OSCC may contain some form of stem cells and it was subsequently shown that only a small subpopulation of the cells in OSCC corresponds to tumor-initiating cells. These finding are in accordance with CSCs concept (17,34) that the tumor mass is a mixture of (a) CSCs dividing themselves to feed the tumor's growth, b) transient amplifying cells that divide themselves a few times before maturing into (c) differentiated tumor cells that do not contribute to tumor growth (4). The isolation of CSCs from oral cancers has mainly been performed with the CD44 marker that was initially used to isolate breast cancer CSCs. <img style="border:1px solid #C3E6D8;float:right;" alt="" src="https://cdn.slidesharecdn.com/ss_thumbnails/oralcancerandcancerstemcell1-240601114544-829bca0a-thumbnail.jpg?width=120&amp;height=120&amp;fit=bounds" /><br> Many evidences that CSCs also play a central role in the pathogenesis and progression of carcinomas of the head and neck (HNSCC), including OSCC,have been found. Early tissue culture studies showed that only a subpopulation of OSCC cells can form expanding tumor colonies, suggesting that human OSCC may contain some form of stem cells and it was subsequently shown that only a small subpopulation of the cells in OSCC corresponds to tumor-initiating cells. These finding are in accordance with CSCs concept (17,34) that the tumor mass is a mixture of (a) CSCs dividing themselves to feed the tumor&#39;s growth, b) transient amplifying cells that divide themselves a few times before maturing into (c) differentiated tumor cells that do not contribute to tumor growth (4). The isolation of CSCs from oral cancers has mainly been performed with the CD44 marker that was initially used to isolate breast cancer CSCs.
cancer stem cell : challenge in cancer treatment.pdf from Romissaa ali Esmail/ faculty of dentistry/Al-Azhar university
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https://public.slidesharecdn.com/v2/images/profile-picture.png Romissaa has graduated from Al-Azhar University. Her grade was very good with an honor degree. She trained for approximately one year in her college at different departments as a house officer ※an intern§. Further, she worked at the Ministry of Health for about nine months as a dentist. Moreover, she worked at Al-Azhar University in Oral Medicine, Periodontology, Oral Diagnosis, and Dental Radiology Department for six years as an instructor, and then still working as a teaching assistant till now. She had completed her master*s degree in Oral Medicine, Periodontology, oral diagnosis, and dental Radiology Department at Al-Azhar University, Faculty of Dental Medicine for Girls, and nearly fini www.linkedin.com/in/romissaa-ali-146832156 https://cdn.slidesharecdn.com/ss_thumbnails/dna-240816122115-8e61e69d-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pdf/271053979 Different techniques a... https://cdn.slidesharecdn.com/ss_thumbnails/dna-240816121750-223ca4d5-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/different-techniques-and-methods-of-dna-and-rna-isolation-pptx/271053932 Different techniques a... https://cdn.slidesharecdn.com/ss_thumbnails/tumor-240718120254-c893d341-thumbnail.jpg?width=320&height=320&fit=bounds slideshow/tumor-microenvironment-underlying-secretes-and-its-impact-on-cancer-progression-pptx/270316886 Tumor microenvironment...