Robotic Assisted Lung Bilobectomy is Performed for First Time
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The surgery was performed on a 63-year-old female patient who was an active smoker with a history of coughing up traces of blood, a common symptom of lung cancer.
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Title: Regulation of Tubular Reabsorption A Comprehensive Overview
Description:
This lecture provides a detailed and structured explanation of the mechanisms regulating tubular reabsorption in the kidneys. It explores how different physiological and hormonal factors influence glomerular filtration and reabsorption rates, ensuring fluid and electrolyte balance in the body.
Who Should Read This?
This presentation is designed for:
鏝 Medical Students (MBBS, BDS, Nursing, Allied Health Sciences) preparing for physiology exams.
鏝 Medical Educators & Professors looking for structured teaching material.
鏝 Healthcare Professionals (doctors, nephrologists, and physiologists) seeking a refresher on renal physiology.
鏝 Postgraduate Students & Researchers in the field of medical sciences and physiology.
What Youll Learn:
Local Regulation of Tubular Reabsorption
鏝 Glomerulo-Tubular Balance its mechanism and clinical significance
鏝 Net reabsorptive forces affecting peritubular capillaries
鏝 Role of peritubular hydrostatic and colloid osmotic pressures
Hormonal Regulation of Tubular Reabsorption
鏝 Effects of Aldosterone, Angiotensin II, ADH, and Natriuretic Peptides
鏝 Clinical conditions like Addisons disease & Conn Syndrome
鏝 Mechanisms of pressure natriuresis and diuresis
Nervous System Regulation
鏝 Sympathetic Nervous System activation and its effects on sodium reabsorption
Clinical Correlations & Case Discussions
鏝 How renal regulation is altered in hypertension, hypotension, and proteinuria
鏝 Comparison of Glomerulo-Tubular Balance vs. Tubulo-Glomerular Feedback
This presentation provides detailed diagrams, flowcharts, and calculations to enhance understanding and retention. Whether you are studying, teaching, or practicing medicine, this lecture will serve as a valuable resource for mastering renal physiology.
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#Physiology #RenalPhysiology #TubularReabsorption #GlomeruloTubularBalance #HormonalRegulation #MedicalEducation #Nephrology
Hingula.ppt- Rasadravya parichaya- NCISM
Hingula.ppt- Rasadravya parichaya- NCISMShri Shivayogeeshwar Rural Ayurvedic Medical college, INCHAL,
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Hingula(Cinnabar)
Synonyms - Churna Parada, Darada, Mleccha, Rasagarbha,Rasodbhava,
Shukatunda, Hamsapada, Lohagna
Mineralogical Identification
Chemical Formula- HgS
Chemical name- mercury sulfide
Nature Crystalline
Colour- Bright Red
Crystal- Trigonal.
Clevage- -
Diaphaneity- Transparent to Translucent
Fracture- Uneven
Tenacity- Brittle
Lustre- Vitreous
Streak- Red
Sp. Gravity- 8 to 8.1
Hardness- 2 to 2.5
Sources
- It is found near volcanic region and near mercury mines(extracted).
- It is associated with other minerals like Stibnite, pyrite, realgar, calcite etc.
- Almaden (Spain), California, Texas.
Types
1) Charmara
2) Shukhatunda
3) Hamsapada ( Ap 2/69)
1) Khanija
2) Kritrima (RT 9/4)
Grahya ad Agrahyata
爐爐爐 爐爛爐伍爐 爐朽ぐ爛爐爐鉦き: 爐爛爐劇ぃ 爐伍爐爐爛爐項ぐ: |
爐爐項爐爛爐朽ぞ爐迦 爐爐鉦ぐ爐爛爐萎爐爛 爐項た爐爐爛爐: 爐謹爐萎爐劇爐 爐爐爛爐爐む || RT 9/3
Doshas
Andya( Blindness)
Kshinata
Klama
Bhrama
Moha
Prameha ( AP 2/73)
Shodhana
1) Grahya Hingula ChurnaArdraka rasa bhavana(7)--Shuddhahingula (RT 9/12)
Marana
Other Processing Techniques
Satwa Patana
-Shuddha HingulaAdhaha patana/Urdhwa patana -Satva (Parada) (RRS 3/144)
Yogas
Hinguleshwar rasa
Anandabhairava Rasa,
Mrutunjaya Rasa,
Tribhuvana keerti Rasa, etc.
Research Updates
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https://ijapr.in/index.php/ijapr/article/view/1237
EXTRACTION OF PARAD FROM HINGULA, A TRADITIONAL AYURVEDIC METHOD
https://www.researchgate.net/publication/323601866_EXTRACTION_OF_PARAD_FROM_HINGULA_A_TRADITIONAL_AYURVEDIC_METHOD
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M.D.(Ayu) FAGE , PGDYT
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Methods to enhance drug solubility (Buffers, Co-solvents, Surfactants, Complexation, Solid Dispersions)
Advanced approaches (Polymorphism, Salt Formation, Co-crystallization, Prodrugs)
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Differentiate histology and cytology
Overview on tissue types
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Overview on common Histological Techniques:
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o Grossing
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Title: Regulation of Tubular Reabsorption A Comprehensive Overview
Description:
This lecture provides a detailed and structured explanation of the mechanisms regulating tubular reabsorption in the kidneys. It explores how different physiological and hormonal factors influence glomerular filtration and reabsorption rates, ensuring fluid and electrolyte balance in the body.
Who Should Read This?
This presentation is designed for:
鏝 Medical Students (MBBS, BDS, Nursing, Allied Health Sciences) preparing for physiology exams.
鏝 Medical Educators & Professors looking for structured teaching material.
鏝 Healthcare Professionals (doctors, nephrologists, and physiologists) seeking a refresher on renal physiology.
鏝 Postgraduate Students & Researchers in the field of medical sciences and physiology.
What Youll Learn:
Local Regulation of Tubular Reabsorption
鏝 Glomerulo-Tubular Balance its mechanism and clinical significance
鏝 Net reabsorptive forces affecting peritubular capillaries
鏝 Role of peritubular hydrostatic and colloid osmotic pressures
Hormonal Regulation of Tubular Reabsorption
鏝 Effects of Aldosterone, Angiotensin II, ADH, and Natriuretic Peptides
鏝 Clinical conditions like Addisons disease & Conn Syndrome
鏝 Mechanisms of pressure natriuresis and diuresis
Nervous System Regulation
鏝 Sympathetic Nervous System activation and its effects on sodium reabsorption
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鏝 Comparison of Glomerulo-Tubular Balance vs. Tubulo-Glomerular Feedback
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油
Hingula(Cinnabar)
Synonyms - Churna Parada, Darada, Mleccha, Rasagarbha,Rasodbhava,
Shukatunda, Hamsapada, Lohagna
Mineralogical Identification
Chemical Formula- HgS
Chemical name- mercury sulfide
Nature Crystalline
Colour- Bright Red
Crystal- Trigonal.
Clevage- -
Diaphaneity- Transparent to Translucent
Fracture- Uneven
Tenacity- Brittle
Lustre- Vitreous
Streak- Red
Sp. Gravity- 8 to 8.1
Hardness- 2 to 2.5
Sources
- It is found near volcanic region and near mercury mines(extracted).
- It is associated with other minerals like Stibnite, pyrite, realgar, calcite etc.
- Almaden (Spain), California, Texas.
Types
1) Charmara
2) Shukhatunda
3) Hamsapada ( Ap 2/69)
1) Khanija
2) Kritrima (RT 9/4)
Grahya ad Agrahyata
爐爐爐 爐爛爐伍爐 爐朽ぐ爛爐爐鉦き: 爐爛爐劇ぃ 爐伍爐爐爛爐項ぐ: |
爐爐項爐爛爐朽ぞ爐迦 爐爐鉦ぐ爐爛爐萎爐爛 爐項た爐爐爛爐: 爐謹爐萎爐劇爐 爐爐爛爐爐む || RT 9/3
Doshas
Andya( Blindness)
Kshinata
Klama
Bhrama
Moha
Prameha ( AP 2/73)
Shodhana
1) Grahya Hingula ChurnaArdraka rasa bhavana(7)--Shuddhahingula (RT 9/12)
Marana
Other Processing Techniques
Satwa Patana
-Shuddha HingulaAdhaha patana/Urdhwa patana -Satva (Parada) (RRS 3/144)
Yogas
Hinguleshwar rasa
Anandabhairava Rasa,
Mrutunjaya Rasa,
Tribhuvana keerti Rasa, etc.
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https://ijapr.in/index.php/ijapr/article/view/1237
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M.D.(Ayu) FAGE , PGDYT
HOD Department of Rasa Shastra&BhaishajyaKalpana
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Mob: 9972710790
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Methods to enhance drug solubility (Buffers, Co-solvents, Surfactants, Complexation, Solid Dispersions)
Advanced approaches (Polymorphism, Salt Formation, Co-crystallization, Prodrugs)
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Overview on tissue types
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o Grossing
o Tissue processing
o Microtomy
o Staining
o Mounting
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Distribution of motion between other two joints.
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Eczema is a group of medical conditions which causes inflammation and irritation to skin.
It is also called as Dermatitis
Eczema is an itchy consisting of ill defined erythremotous patches. The skin surface is usually scaly and As time progress, constant scratching leads to thickened lichenified skin.
Several classifications of eczemas are available based on Etiology, Pattern and chronicity.
According to aetiology Eczema are classified as:
Endogenous eczema: Where constitutional factors predispose the patient to developing an eczema.
Family history (maternal h/o eczema) is often present
Strong genetic predisposition (Filaggrin gene mutations are often present).
Filaggrin is responsible for maintaining moisture in skin (hence all AD patients have dry skin.
Immunilogical factor-Th-2 disease, Type I hypersensitivity (hence serum IgE high)
e.g., Seborrheic dermatitis, Statis dermatitis, Nummular dermatitis, Dyshidrotic Eczema
Exogenous eczema: Where external stimuli trigger development of eczema,
e.g., Irritant dermatitis, Allergic Dermatitis, Neurodermatitis,
Combined eczema: When a combination of constitutional factors and extrinsic triggers are responsible for the development of eczema
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Extremes of Temperature
Irritants : Soaps, Detergents, Shower gels, Bubble baths and water
Stress
Infection either bacterial or viral,
Bacterial infections caused by Staphylococcus aureus and Streptococcus species.
Viral infections such as Herpes Simplex, Molluscum Contagiosum
Contact allergens
Inhaled allergens
Airborne allergens
Allergens include
Metals eg. Nickle, Cobalt
Neomycin, Topical ointment
Fragrance ingredients such as Balsam of Peru
Rubber compounds
Hair dyes for example p-Phenylediamine
Plants eg. Poison ivy .
Atopic Dermatitis : AD is a chronic, pruritic inflammatory skin disease characterized by itchy inflamed skin.
Allergic Dermatitis: A red itchy weepy reaction where the skin has come in contact with a substance That immune system recognizes as foreign substances.
Ex: Poison envy, Preservatives from creams and lotions.
Contact Irritant Dermatitis: A Localized reaction that include redness, itching and burning where the skin has come In contact with an allergen or with irritant such as acid, cleaning agent or chemical.
Dyshidrotic Eczema: Irritation of skin on the palms and soles by
clear deep blisters that itch and burn.
Clinical Features; Acute Eczema:- Acute eczema is characterized by an erythematous and edematous plaque, which is ill-defined and is surmounted by papules, vesicles, pustules and exudate that dries to form crusts. A subsiding eczematous plaque may be covered with scales.
Chronic Eczema:- Chronic eczema is characterized by lichenification, which is a triad of hyperpigmentation, thickening markings. The lesions are less exudative and more scaly. Flexural lesions may develop fissures.
Pruritus
Characteristic Rash
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