LBBB + RBBB

Oct 18, 2016Download as PPT, PDF11 likes1,512 views

kashif Anwer

LBBB+RBBB

Cardiac arrhythmias are abnormalities in the heart's rhythm. There are two main types: bradycardia, a slow heart rate, and tachycardia, a fast heart rate. Various arrhythmias are described including sinus bradycardia, heart block, atrial fibrillation, atrial flutter, AV nodal reentry tachycardia, ventricular fibrillation, and ventricular tachycardia. Treatment depends on the type of arrhythmia and may include medication, cardioversion, ablation, or pacemaker implantation. Diagnosis involves ECG, echocardiogram, blood tests, and other cardiac tests. Lifestyle changes and avoiding arrhythmia triggers can help management.

2nd part ECG basics PR interval and heart blockDr Salah Mabrouk Khallaf

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This document discusses PR interval and heart block. It defines the PR interval as the time from the beginning of the P wave to the beginning of the QRS complex, normally ranging from 120-200ms. Abnormal PR intervals can be shortened or prolonged. A shortened PR interval may indicate preexcitation syndromes like WPW syndrome or AV junctional rhythms. A prolonged PR interval suggests different degrees of heart block, from first degree block with a constant delay, to second degree blocks like Mobitz I and II, and third degree or complete heart block where there is no relationship between P waves and QRS complexes.

ECG: Ventricular Premature BeatsStanley Medical College, Department of Medicine

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1. The ECG shows a 45-year-old diabetic man with episodes of palpitations who presents with ventricular trigeminy. 2. Ventricular trigeminy is characterized by ventricular premature beats occurring after every two normal sinus beats with a compensatory pause. 3. Further investigation with Holter monitoring is recommended to better characterize the ventricular arrhythmia and assess for underlying cardiac disease.

ECG -QRS complexPROFESSOR DR. MD. TOUFIQUR RAHMAN

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Repolarization syndromeshttps://aiimsbhubaneswar.nic.in/

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Early repolarization (ER), consisting of a J-point elevation, notching or slurring of the terminal portion of the R wave (J wave), and tall/symmetric T wave, is a common finding on the 12-lead electrocardiogram. For decades, it has been considered as benign, barring sporadic case reports and basic electrophysiology research that suggested a critical role of the J wave in the pathogenesis of idiopathic ventricular fibrillation (VF). In 2007-2008, a high prevalence of ER in patients with idiopathic VF was reported and subsequent studies reinforced the results. This PPT describes the current state of knowledge concerning ER syndrome associated with sudden cardiac death.

3rd part ECG Basics QRS complex Dr Salah Mabrouk KhallafDr Salah Mabrouk Khallaf

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This document provides an overview of QRS complexes and abnormalities seen on electrocardiograms (ECGs). It defines the components of the QRS complex and discusses causes of low or high voltage QRS complexes. Specific conditions that can cause left or right ventricular hypertrophy are described. Various conduction abnormalities are also summarized, including right and left bundle branch blocks, fascicular blocks, and bifascicular blocks. Causes of wide QRS complexes like hyperkalemia and certain drugs are mentioned. The document aims to educate on interpreting and analyzing QRS complexes on ECGs.

Arrhythmiashttps://aiimsbhubaneswar.nic.in/

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Right and left ventricular hypertrophyRawalpindi Medical College

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Left ventricular hypertrophy is an increase in the mass of the left ventricle that can be caused by hypertension, hypertrophic cardiomyopathy, aortic stenosis, or athletic training. It is defined on an ECG as increased voltages in certain leads. Risk factors include age, gender, high blood pressure, obesity, and genetic factors. If left untreated, LVH can lead to heart failure, arrhythmias, heart attack, or sudden cardiac death. Right ventricular hypertrophy is the enlargement of the right ventricle and can be caused by pulmonary hypertension, congenital heart defects, or lung diseases. Both LVH and RVH are diagnosed using ECG criteria and can cause chest pain, palpitations

AV BlockssruthiMeenaxshiSR

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This document discusses various types of conduction abnormalities and atrioventricular (AV) block. It defines AV block as a delay or interruption in conduction from the atria to the ventricles. The types of AV block discussed are first, second, and third degree block. Second degree block is further classified into Mobitz type I (Wenckebach) and type II. Causes and indications for pacing are also summarized. Case examples are presented to demonstrate different types of AV block.

Cardiac rhythm disordersLih Yin Chong

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The document provides guidance on cardiac arrest and arrhythmia management. The key steps are to check for responsiveness, call for help, open the airway, give rescue breaths if needed, check the pulse and perform chest compressions if pulseless. CPR is used to keep the patient alive until cardioversion. Ventricular fibrillation is treated with immediate defibrillation followed by CPR and epinephrine. Atrial fibrillation is typically managed by controlling the heart rate and long-term anticoagulation to prevent stroke.

Sick Sinus SyndromeAyesha Bukhari

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Sick sinus syndrome is a group of abnormalities that prevent the sinoatrial node from performing its pacemaking function of the heart. It is characterized by sinus node dysfunction resulting in an atrial rate that is inappropriate for physiological needs. Common causes include ischemia, fibrosis, drugs, or infiltrative disorders. Symptoms include syncope, palpitations, and dizziness. Treatment of recurrent symptomatic bradycardia or prolonged pauses requires a permanent pacemaker.

Myocardial infarction (MI) ecg localisationMalleswara rao Dangeti

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This document discusses various non-coronary causes of ST-elevation on electrocardiograms (ECGs) including ventricular aneurysms, pericarditis, early repolarization patterns, left ventricular hypertrophy, left bundle branch block, hypothermia, cardioversion, intraventricular hemorrhage, hyperkalemia, Brugada pattern, type 1C antiarrhythmic drugs, hypercalcemia, pulmonary embolism, hypothermia, myocarditis, and tumor invasion of the left ventricle. It then discusses left ventricular aneurysms, early repolarization, acute pericarditis, hyperkalemia, hypothermia, increased intracranial pressure, Brugada syndrome, Tak

Ventricular tachycardiaApollo Hospitals

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Ventricular tachycardia is a fast heart rhythm originating from the ventricles with a rate over 100 bpm. It is classified based on duration (sustained vs non-sustained), morphology (monomorphic, polymorphic, sinusoidal), and symptoms. Causes include structural heart disease, electrolyte abnormalities, drugs, and prolonged QT interval. Diagnosis involves ECG criteria showing ventricular origin. Treatment depends on hemodynamic stability and may include antiarrhythmic drugs, implantable cardioverter-defibrillator, catheter ablation, or surgery. Recurrent ventricular tachycardia is managed long term with devices, drugs, and treatment of underlying causes.

Lvh & rvhNiyaz Mohammed

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This document discusses left ventricular hypertrophy (LVH) and right ventricular hypertrophy (RVH). It defines LVH as an increase in left ventricle mass due to increased wall thickness or cavity size. There are two types of LVH - systolic overload from conditions like hypertension which compromise the left ventricle during systole, and diastolic overload from things like valvular diseases which compromise it during diastole. The document outlines ECG criteria for diagnosing LVH including Sokolov-Lyon and Cornell voltage criteria. It also discusses RVH manifestations on ECG like right axis deviation, tall R waves in right precordial leads, and an S1S2S3 pattern.

Arrythmia ratheeshRatheeshkrishnakripa

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This document defines and describes various types of arrhythmias. It begins by defining arrhythmia as any change from the normal sequence of electrical impulses in the heart. Various cardiac and non-cardiac conditions can cause arrhythmias. The document then describes several specific types of arrhythmias in detail, including sinus bradycardia, sick sinus syndrome, sinus tachycardia, premature atrial contractions, atrial flutter, atrial fibrillation, junctional tachycardia, paroxysmal supraventricular tachycardia, premature ventricular contractions, ventricular fibrillation, pulseless electrical activity, asystole, and various types of heart block. Treatment options are provided for each type

ECG, step by step approach (Updated)Kerolus Shehata

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Sinus node dysfunctionsruthiMeenaxshiSR

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This document discusses sinus node dysfunction (SND), which refers to dysfunction of the sinoatrial node that can cause various electrocardiogram abnormalities like sinus bradycardia, sinus pauses, and inadequate heart rate response to activity. Common causes of SND include sinus node fibrosis, medications that depress sinus node function, infiltrative diseases, inflammatory diseases, and sinus node artery disease. The document recommends permanent pacing for patients with SND who experience symptomatic bradycardia or pauses, as well as those with chronotropic incompetence. It describes various ECG patterns that can occur in SND such as sinus bradycardia, sinus pause/arrest, sinus node exit block, and chronotropic incompetence.

Dr Vivek Baliga - Diastolic heart failure - A complete overviewDr Vivek Baliga

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Wolff–Parkinson–White syndromeWayne Adighibenma

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Wolff-Parkinson-White syndrome is caused by an abnormal accessory electrical pathway between the atria and ventricles that can bypass the AV node and allow rapid conduction, potentially causing palpitations, dizziness and other symptoms; the condition is usually asymptomatic but can cause tachyarrhythmias due to orthodromic or antidromic conduction along the accessory pathway; treatment involves catheter ablation to destroy the accessory pathway or medications to control the heart rate during arrhythmias.

Narrow QRS Tachycardiahttps://aiimsbhubaneswar.nic.in/

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Tachycardias are broadly categorized based upon the width of the QRS complex on the electrocardiogram (ECG). A narrow QRS complex (<120 milliseconds) reflects rapid activation of the ventricles via the normal His-Purkinje system, which in turn suggests that the arrhythmia originates above or within the His bundle (ie, a supraventricular tachycardia). The site of origin may be in the sinus node, the atria, the atrioventricular (AV) node, the His bundle, or some combination of these sites. A widened QRS (≥120 milliseconds) occurs when ventricular activation is abnormally slow. The most common reason that a QRS is widened is because the arrhythmia originates below the His bundle in the bundle branches, Purkinje fibers, or ventricular myocardium (eg, ventricular tachycardia). Alternatively, a supraventricular arrhythmia can produce a widened QRS if there are either pre-existing or rate-related abnormalities within the His-Purkinje system (eg, supraventricular tachycardia with aberrancy), or if conduction occurs over an accessory pathway. Thus, wide QRS complex tachycardias may be either supraventricular or ventricular in origin.

Basics of ECG.ppt dr.k.subramanyamAdarsh

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This document provides an overview of electrocardiography (ECG), including how an ECG works, the basics of recording an ECG, ECG leads, normal ECG waveforms and intervals, interpreting an ECG, common abnormalities, and how to report an ECG. It discusses topics such as the cardiac conduction system, Einthoven's triangle, the 12-lead ECG, determining heart rate and axis, normal sinus rhythm, P waves, QRS complex, ST segment, T waves, and the QT interval.

ECG Wellen's sign and Widow maker sign presentationmeducationdotnet

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Arrhythmia Shany Thomas

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1. Dysrhythmias are disorders of heart rhythm caused by abnormalities in the heart's electrical conduction system. 2. Common dysrhythmias include sinus tachycardia/bradycardia, premature atrial contractions, atrial fibrillation/flutter, supraventricular tachycardia, ventricular tachycardia/fibrillation, heart blocks, and bundle branch blocks. 3. Dysrhythmias can be caused by underlying conditions like myocardial infarction, electrolyte imbalances, drug toxicity, and more. Management involves treating the underlying cause and using medications, cardioversion, or pacemakers depending on the specific dysrhythmia.

TachyarrhythmiaSMSRAZA

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This document provides an overview of evaluating and treating different types of tachycardia, including: 1) It discusses evaluating the patient's hemodynamic stability, history, and ECG to determine the characteristics and cause of the tachycardia. 2) It describes differentiating between narrow and wide complex tachycardias, and the differential diagnoses for each, including sinus tachycardia, atrial fibrillation, AV nodal reentrant tachycardia, and ventricular tachycardia. 3) It provides guidance on therapies for different tachycardias, such as electrical or chemical cardioversion, rate control, and ablation. The importance of correctly diagnosing wide complex tachycard

Arrhythmia Ashwini Somayaji

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This document defines and discusses cardiac arrhythmia, including abnormal heart rhythms such as bradycardia and tachycardia. It outlines the causes of arrhythmia such as ischemia, mechanical injury, and electrolyte imbalances. Symptoms can include palpitations, syncope, chest pain, and shortness of breath. The document then covers the normal cardiac rhythm and the mechanisms that can lead to arrhythmia, such as enhanced pacemaker activity, afterdepolarizations, reentry circuits, and changes in automaticity. Finally, it discusses the classification and treatment of arrhythmias with various drug classes.

SUPRAVENTRICULAR TACHYCARDIA - SVTPinkesh Parmar

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This document discusses several types of supraventricular tachycardia (SVT), including their definitions, pathophysiology, diagnosis, and treatment. It covers sinus tachycardia, sinus node reentry, atrial flutter, and atrial tachycardia. For each type, it describes the characteristic heart rate, P wave morphology, and mechanisms involving automaticity, reentry, or triggered activity. Treatment options discussed include medications, cardioversion, ablation, and stroke prophylaxis.

Premature Atrial complexesMohamed Salih Aziz

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The document discusses three types of atrial premature complexes: 1. Normally conducted complexes which have an early P wave that conducts normally with a PR interval that may be normal, increased, or decreased and a QRS similar to sinus rhythm. 2. Non-conducted complexes where an early P wave is not followed by a QRS and the P wave is often hidden in the T wave. 3. Complexes with aberrant conduction where the early P wave is followed by a QRS with an abnormal conduction pattern most commonly right bundle branch block but sometimes left bundle branch block.

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