This document provides guidance on performing a general examination of the musculoskeletal system. It outlines examination principles and specific techniques for assessing the skin, nails, soft tissues, and individual joints. Key steps include observing appearance, palpating for warmth, swelling or deformity, and assessing both active and passive range of motion. Common abnormal findings associated with various rheumatologic conditions are described. The overall examination sequence and areas to evaluate are also outlined.
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2-General Examination of nursing MSS.pptx
1. General Examination of the
Musculoskeletal System
Prepared by Dr. Ruqaya Al-Kathiry
Head of the Medical Department in UST
Medicine is learned at
the bedside
and not in the classroom
Sir William Osler (1849-1919)
3. Dynamic tests are difficult to describe in pictures & text so ask an experienced clinician
to check your technique. Practise examining as many joints as possible to become
familiar with NL appearances & ranges of movement.
General Principles: Ask Look Feel Move
After taking the Hx observe, palpate & move.
Always expose the joint above & below the one in question.
Compare one limb with the opposite side.
Examine the overall appearance for pallor, rash, skin tightening & hair changes.
Look at the skin, SC tissues & bony outline of each area.
Before palpating, ask the pt which area is painful or tender.
Feel for warmth, swelling, stability & deformity.
Assess if a deformity is reducible or fixed.
Assess active before passive movement. Do not cause the pt additional pain.
In suspected systemic dis, examine all joints and fully examine all systems.
Use standard terminology to describe joint limb positions & movement. Always
describe movements from the neutral position. Commonly used terms are:
o Flexion: bending at a joint from the neutral position
o Extension: straightening a joint back to the neutral position
o Hyperextension: movement beyond the NL neutral position because of a torn
lig. or underlying ligamentous laxity, e.g. Ehlers-Danlos $
PHYSICAL EXAMINATION
4. o Adduction: movement towards the midline of the body (finger adduction is
movement towards the axis of the limb)
o Abduction: movement away from the midline.
Describe the position of a limb because of joint/bone deformity:
o Valgus: the distal part deviates away from the midline
o Varus: the distal part deviates towards the midline.
Equipment:
You need a:
Tape measure
Tendon hammer
Goniometer (protractor for measuring the range of joint movement)
Stethoscope
Blocks for assessing leg length discrepancy
General Examination:
Skin, nail & soft tissue
ABNL Findings:
Skin & related struct. are the most common sites of ass. lesions.
Psoriasis may be hidden (U, natal cleft, scalp). Nail changes:
fine or coarse pits in nail, onycholysis, thickening and ridging.
Rash of SLE is induced by UV light exposure.
5. Small, dark red vasculitic spots (due to capillary infarcts) occur
in RA, SLE & PAN These indicate active dis. Common sites:
nail folds, finger and toe tips & other pressure areas.
Raynauds phenomenon: episodic ischaemia of the fingers
ppt. by stimuli as cold, pain & stress. There is a typical
progression of colour changes: blanching (white) followed by
cyanosis (blue) & reactive hyperaemia (red) ass. dysaesthesia
(altered sensation) & pain. It is common in healthy individuals
but is a freq. in systemic sclerosis & SLE.
Systemic sclerosis: thickened, tight skin produces a ch.ch. facial appearance. In the
hands, flexion contractures, calcium deposits in the finger pulps & tissue ischaemia
leading to ulceration. Telangiectasias of systemic sclerosis are purplish, blanch with
pressure & are most common on the hands & face.
Beaking of the nose & taut skin around the mouth
Calcium deposits ulcerating through the skin
Reactive arthritis has extra-art. features & is ass. with skin & nail changes similar to
those of psoriasis, together with conjunctivitis, circinate balanitis (painless superficial
ulcers on the prepuce & glans), urethritis & superficial mouth ulcers.
6. Nodules:
S.C nodules in RA: most commonly occur on the extensor
surface of the forearm. They are firm & non-tender, felt
at sites of pressure or friction (sacrum or Achilles tendon).
Multiple small nodules can occur in the hands & are part.
ass. with MTX therapy. These are strongly ass. with a +ve RF
& can occur at other sites (lungs).
Bony nodules in OA: affect the hand & are smaller &
harder than rheumatoid nodules. They occur on the lat.
aspects of the interphalangeal (IP) joints.
o DIP joints Heberdens nodes.
o PIP joints Bouchards nodes.
Gouty tophi: firm, white, irregular SC crystal collections
(monosodium urate monohydrate). Common sites:
olecranon bursa, helix of the ear & extensor aspects of
the fingers, hands, knees & toes. The overlying skin may
ulcerate, discharge crystals & become secondarily
infected.
Heberdens nodes
Bouchards nodes
Eyes: affected in many MSS conditions.
Dry Eyes: Sj旦grens $, RA & other CTD.
Blue Sclerae: certain types of osteogenesis imperfecta
& scleromalacia of RA.
8. Examination Sequence:
Perform Schirmers tear test to Dx keratoconjunctivitis sicca.
Hook a small strip of notched blotting paper ~40 mm long over the lower eyelid
while the pt looks upwards. The notch is ~5 mm from one end of the strip & is where
the strip is bent over the eyelid.
Ask the pt to close the eye.
Wait for exactly 5mins, then remove the strip.
Measure the distance that tears travel down the strip with a millimetre rule:
>15mm = NL
515 mm = equivocal
<5 mm = ABNL
ABNL findings
Conjunctivitis reactive arthritis.
tear production with dry eyes (keratoconjunctivitis sicca) conjunctivitis &
blepharitis (inflam. of the eyelids) Sj旦grens $ & as 2ary changes in RA & other CTD.
Scleritis & episcleritis RA & psoriatic arthritis.
Ant. uveitis (iritis) ~25% of pts with ankylosing spondylitis & reactive arthritis but
is asymp. in juvenile idiopathic arthritis (JIA), so ophthalmological assessment is
essential if JIA is suspected.
The sclerae are blue in certain types of osteogenesis imperfecta & in the
scleromalacia of long-standing RA.
9. Medicine is
learned at the
bedside
and not in the
classroom
Sir William Osler (1849-1919)
THANK YOU FOR YOUR ATTENDANCE