Addictions Presentation

Oct 14, 2009Download as PPT, PDF0 likes228 views

This document discusses the code of ethics for registered nurses and a methadone program. The code of ethics outlines providing safe, compassionate, competent and ethical care, promoting health and well-being, and promoting and respecting informed decision-making. The document also mentions a methadone program.

Code of Ethics 1. Providing safe, compassionate, competent and ethical care 2. Promoting health and well-being 3. Promoting and respecting informed decision-making Code of Ethics for Registered Nurses (2008 Centennial Edition)

The document provides a summary of the opening scenes of the movie "IT" by Stephen King. It describes the opening credits with creepy music setting an ominous tone. Next, it shows black and white photos from an old album zooming out to reveal the main child characters. The last photo is of an old movie theater advertising retail space. The scene then changes to a backyard where a little girl is playing with her doll during a storm. She sees a clown hiding behind the laundry who says "Hi" with a friendly smile that then turns into a menacing snarl. The camera zooms in on the girl's face changing from curiosity to shock and fear as the music intensifies, then fades to black.

addiction ppt without moviepharho

Photovoice N4165pharho

This document discusses mental health and addiction from the perspective of lived experiences. It touches on several themes: the challenges of navigating health services and support systems; the stigma and discrimination faced by those with mental illness; the complex interplay between various social and biological determinants of health; and the importance of holistic, client-centered care that promotes wellness. Quotes from clients emphasize the difficulties of isolation, instability, and overwhelming symptoms, as well as the role of coping skills, treatment, and hope in recovery.

Mind The Gap Picspharho

This document discusses living with mental illness and the challenges that come with it. The author feels their life is in pieces, invisible in a crowd despite being surrounded by people. Living with a mental illness is described as an uphill battle, with feelings of isolation and like everything is a crisis without the support of friends and family who can provide peace of mind and prevent deeper isolation.

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addictionspharho

Addictions Presentationpharho

addictionspharho

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This document summarizes a research article about the implications of mindfulness for substance abuse and addiction. Some key points: - Mindfulness originated in Buddhism but has been adapted for use in Western psychology to help with various conditions. It involves paying non-judgmental attention to present-moment experiences. - Research shows mindfulness interventions may help with problems like stress, anxiety, depression, and pain. It is now included in treatments for substance abuse to help cope with urges to use. - However, the research is mixed on whether mindfulness alone can effectively treat substance abuse. Spirituality may be a more important factor and its relationship to mindfulness needs more study. - Addiction could be viewed as a "spiritual emergency

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This document discusses crisis management and self-soothing behaviors. It defines a crisis as a stressful or traumatic event that one wants resolved immediately in the short term. It explains that during a crisis, if the problem cannot be solved right away or made worse, one needs to use crisis survival skills to survive the crisis without making it worse. These crisis survival skills involve using self-soothing behaviors that involve the five senses of vision, hearing, smell, taste and touch to calm and comfort oneself.

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This document discusses crisis management and provides strategies for surviving a crisis without making the situation worse. It defines a crisis as a stressful or traumatic event that one wants resolved immediately in the short term. It recommends using distracting behaviors to survive a crisis when the problem cannot be solved right away or getting worse is a risk. These distracting behaviors include engaging in activities, contributing to others, making comparisons, focusing on emotions or sensations, putting thoughts away, and focusing on different thoughts. The document advises evaluating coping strategies used after a crisis to determine what worked and how to improve for next time.

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Title: Sensory Receptors | Comprehensive Physiology Lecture by Dr. Faiza (MBBS, FCPS Physiology, CHPE, MPH, MBA) Description: This in-depth lecture on Sensory Receptors, delivered by Dr. Faiza, Assistant Professor of Physiology, is designed for undergraduate and postgraduate medical students, particularly those studying MBBS, BDS, or allied health sciences. It provides a comprehensive overview of the physiology of sensory receptors, integrating foundational concepts with clinical relevance. Key Topics Covered: Classification of sensory receptors based on stimulus type, location, and structure Mechanism of sensory transduction: from stimulus to receptor potential and generation of action potential Differences between tonic (slow-adapting) and phasic (rapid-adapting) receptors Detailed comparison of major mechanoreceptors: structure, location, adaptation, and innervation Modality, intensity, location, and duration as principles of sensory coding Labeled Line Principle and Lateral Inhibition: how the brain identifies and localizes stimuli Clinical insights into receptor adaptation, two-point discrimination, and rate receptors Summary of graded vs action potentials and their roles in encoding stimulus strength Overview of afferent fiber types and nerve fiber classification Why Watch? Conceptual clarity supported by well-organized diagrams and comparative tables Designed for high-yield retention with clinical applications and exam relevance Ideal for students preparing for university exams, FCPS/MD entrance tests, and health sciences educators revisiting core concepts Created by a well-qualified educator with diverse expertise: MBBS (Best Graduate, AIMC Lahore), FCPS Physiology, CHPE, DHPE (STMU), MPH, and MBA Target Audience: MBBS and BDS students Postgraduate Physiology and Basic Sciences trainees Nursing and Allied Health students

DEMENTIA || degenerative neurologiclal dissorderAnurag Joseph

�ݺ�ߣ 1: Title �ݺ�ߣ Title: Understanding Dementia: Definition, Types, Diagnosis, Pathophysiology, Management, and Nursing Care Plan Subtitle: A comprehensive guide for nursing students �ݺ�ߣ 2: Definition of Dementia Definition: Dementia is a general term for a decline in cognitive ability severe enough to interfere with daily life. Characteristics: Memory loss Difficulty with communication and reasoning Progressive and irreversible decline Affects behavior, thinking, and emotions �ݺ�ߣ 3: Types of Dementia Alzheimer's Disease: Most common form Characterized by memory loss, confusion, and changes in behavior Vascular Dementia: Caused by reduced blood flow to the brain (stroke or series of strokes) Symptoms: Impaired judgment, memory loss Lewy Body Dementia: Hallucinations, movement problems, and cognitive decline Frontotemporal Dementia: Affects personality, behavior, and language, often at a younger age �ݺ�ߣ 4: Diagnosis of Dementia Methods of Diagnosis: Clinical Assessment: Detailed patient history (family, cognitive changes) Cognitive Testing (e.g., MMSE): Mini-Mental State Examination to assess cognitive function Neuroimaging (CT/MRI): Identifies structural brain changes Laboratory Tests: Rule out other causes (e.g., thyroid disorders, vitamin deficiencies) �ݺ�ߣ 5: Pathophysiology of Dementia Neurodegenerative Processes: Alzheimer��s Disease: Amyloid plaques and neurofibrillary tangles damage brain cells Vascular Dementia: Reduced blood supply due to blockages or mini-strokes Cognitive Decline: Impaired communication between neurons Brain regions affected: hippocampus, frontal lobe, temporal lobe Progressive Nature: Loss of neurons and synaptic connections over time, leading to increased functional decline �ݺ�ߣ 6: Management of Dementia Pharmacological Treatment: Cholinesterase Inhibitors: (e.g., Donepezil) for Alzheimer's NMDA Antagonists: (e.g., Memantine) for moderate-to-severe Alzheimer��s Antipsychotics: For managing severe behavioral symptoms (caution with side effects) Antidepressants: For mood stabilization Non-Pharmacological Treatment: Cognitive Stimulation Therapy (CST): Stimulate memory and cognitive skills Behavioral Therapy: For managing aggression or depression Social Interaction & Routine Activities: Help with orientation and reduce anxiety Physical Activity: Promotes brain health and overall well-being �ݺ�ߣ 7: Nursing Care Plan for Dementia Assessment: Monitor cognitive function, mood, and behavior changes Evaluate risk for falls, malnutrition, and skin integrity Assess the need for caregiver support and education Nursing Diagnoses: Impaired Memory Risk for Injury Disturbed Thought Processes Caregiver Role Strain Goals: Maximize cognitive function as much as possible Maintain safety and prevent injury Improve quality of life for the patient and caregiver Interventions: Promote a structured daily routine to reduce confusion Provide cognitive stimulation

Silent Signals_ Recognizing Early Markers in Hematologic Malignancies.pptxzeelm1995

Silent Signals: Recognizing Early Markers in Hematologic Malignancies is a clinical presentation designed to raise awareness among healthcare professionals about the often-overlooked early indicators of blood cancers such as chronic lymphocytic leukemia (CLL), acute myeloid leukemia (AML), and multiple myeloma (MM). These malignancies can begin with subtle, nonspecific symptoms��often mistaken for benign conditions��leading to delayed diagnoses and suboptimal patient outcomes. This presentation walks the audience through the critical importance of early detection in hematologic malignancies. It begins by establishing the clinical and emotional impact of delayed diagnoses, highlighting that up to 30% of blood cancers may initially present with vague symptoms like fatigue, recurrent infections, or mild lab abnormalities. These ��silent signals�� are frequently dismissed or misattributed, emphasizing the need for clinicians to maintain a high index of suspicion. Through a structured four-part approach, the slides explore: The Stakes of Early Detection Understand why timing matters. This section provides key statistics on survival rates and emotional burden, and it contrasts benign explanations with red-flag symptoms, enabling clinicians to differentiate between common ailments and potential warning signs of malignancy. Lab Markers That Matter Subtle shifts in routine blood work can be crucial. Clinicians are guided to recognize how markers like elevated LDH, low hemoglobin, or abnormal WBC differentials may indicate deeper hematologic pathology. A simplified diagnostic pathway is provided to help connect clinical presentation to appropriate testing and specialist referral. A Hidden Myeloma: Case Spotlight A real-world case of a 55-year-old male patient illustrates how early symptoms��persistent fatigue, bone pain, and recurrent infections��were initially attributed to benign causes. The turning point came from diligent testing and interpretation of lab results (including SPEP and bone marrow biopsy), which led to a diagnosis 8 months earlier than the average. The outcome? A measurable clinical improvement, including very good partial response (VGPR) after prompt treatment initiation. Key Learnings for Practice This section summarizes actionable takeaways: think beyond the usual when symptoms linger, repeat and investigate unexplained lab anomalies, and ensure coordinated care between GPs, hematologists, and lab services. Clinician collaboration is not just helpful��it��s vital in reducing time to diagnosis and improving patient outcomes. Whether you're a general practitioner, internist, or early-career specialist, this presentation equips you with the insights needed to better recognize the earliest signs of hematologic malignancies. The ultimate goal? Reduce diagnostic delay, initiate timely intervention, and improve survival and quality of life for patients with blood cancers. Author: Zeel Mehta

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Musculoskeletal involvement in children is often challenging, with a wide range of benign conditions, such as growing pain, to severe systemic diseases. Spondylo-epiphyseal dysplasia (SED) is an autosomal recessive disease of the skeletal system that often mimics juvenile idiopathic arthritis (JIA) with no response to anti-rheumatic therapy. Case - A young male, 22 years of age, presented with a history of pain in his upper and lower back and multiple small and large joints since five years of age. He denied any morning stiffness or increased nocturnal pain. Previously, he was treated outside as juvenile idiopathic arthritis with methotrexate, sulfasalazine and injection Adalimumab. However, his mother denied any symptomatic improvement of his pain. On examination, his growth was stunted with significant kyphoscoliosis. (Figure 1 ) He had an antalgic gait with exaggerated lumbar lordosis. He had mild tenderness in both hips, knees and ankles with flexion contracture and bony swelling at bilateral proximal & distal interphalangeal joints (PIP and DIP), referred to as camptodactyly. (Figure 2 ) His cervical and lumbar movement was severely restricted, with bilateral hip movement restricted. Laboratory investigation showed normal ESR CRP. Rheumatoid factor was low titre positive, anti-CCP antibody and HLA-B27 by PCR were negative. His skeletal radiographic images showed platyspondyly in the thoracolumbar spine, epiphyseal widening in MCP and IP joints with ovoid carpal bones (Figure 3), platyspondyly in the thoracolumbar spine (Figure 4) , hip joint sclerosis . (Figure 5 ) His diagnosis was reconsidered as spondyloepiphyseal dysplasia (SED) . He was started with extensive physiotherapy and symptomatic pain relief treatment and noticed improvement in his pain and quality of life. Brief discussion - SED predominantly involves articular cartilage, which results in joint stiffness and epiphyseal enlargement. Typically starting at 2-8 years of age, they manifest with pain, stiffness and swelling of extremity joints. (1) On examination, the swelling is predominantly bony without any evidence of inflammation. Over time, the large joints and the spine get involved, causing significant joint contractures, gait abnormality, kyphoscoliosis, abnormal posture and poor quality of life. There are reported cases where SED have been misdiagnosed as JIA with no response to immunomodulatory treatment. (1,2) SED's radiological features differ from JIA, with platyspondyly being an early finding in the skeletal image, epiphyseal enlargement, and lack of destructive joint erosions. Though SED is considered a rare entity, one large study by Dalal et al. (3) reported 35 cases of SED from 25 unrelated families with 11 different homozygous mutations and one instance of compound heterozygosity in the WISP3 gene. It proves that this entity is not very uncommon in India.

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? Title: Anemia �C Types, Causes, Pathophysiology, and Clinical Features | Dr. Faiza ? Presented by: Dr. Faiza Assistant Professor of Physiology MBBS (Best Graduate, AIMC Lahore) FCPS Physiology ICMT, CHPE, DHPE (STMU) MPH (GC University, Faisalabad) MBA (Virtual University of Pakistan) ? This presentation is a comprehensive overview of Anemia, designed for medical students and healthcare professionals. It covers: ? Learning Objectives: Classification of anemias: Microcytic, Macrocytic, Normocytic Differentiation based on etiology, lab findings, and clinical presentation Unique features of hereditary and acquired anemias (e.g., thalassemia, sickle cell disease) Diagnostic approaches and pathophysiological mechanisms Impact of anemia on circulatory function An overview of polycythemia vera vs secondary polycythemia ? Includes detailed tables, RBC indices, blood picture interpretation, and mnemonics for easier recall. ? Ideal for: MBBS and BDS students FCPS Part 1 candidates Educators revising core physiology Clinical practitioners updating their concepts ? Topics Covered: Iron Deficiency Anemia Thalassemia (Alpha & Beta) Sickle Cell Anemia Megaloblastic Anemia (Vitamin B12 & Folate) Aplastic Anemia Acute Blood Loss Hemolytic Anemias (Hereditary Spherocytosis, G6PD Deficiency) Circulatory changes in anemia Primary & Secondary Polycythemia

Understanding Acute and Chronic Renal Failure: Pathophysiology, Etiology, Cli...Dr Aman Suresh Tharayil

This presentation provides a comprehensive overview of Acute Renal Failure (ARF) and Chronic Renal Failure (CRF), covering definitions, causes, pathophysiology, signs and symptoms, and stages of disease progression. It is designed for B.Pharm 2nd semester students, pharmacy educators, and healthcare learners seeking a solid foundation in renal pathophysiology. The slides explore key differences between ARF and CRF, highlighting pre-renal, intra-renal, and post-renal causes, and the progression of chronic renal disease from early decline in renal reserve to end-stage kidney failure. Ideal for use in academic lectures, self-learning, or clinical revision.

Grazi - Personalizing surgical strategy in HPB surgery.pptxGian Luca Grazi

The growing need to introduce guidelines for the optimal management of various pathological conditions has highlighted that only a certain percentage of patients can be correctly classified in these guidelines. Liver, pancreas and biliary tract diseases that require surgical treatment present this problem. The anatomical variables of individual patients and diseases that develop in unpredictable ways make it necessary to personalize treatment. This scientific presentation analyzes the modern tools available to the medical community for the surgical treatment of hepato-biliary-pancreatic tumors. The potential of using "big data" is illustrated, in particular with the help of artificial intelligence, the application of 3D reconstruction models and virtual reality, and the use of vital dyes during minimally invasive surgical interventions. Finally, it is emphasized that in daily clinical practice not all patients are treated following international guidelines. There are situations, such as the treatment of hepatocellular carcinoma, in which a significant portion of patients are treated with alternative approaches, even though these may yield inferior results compared to more aggressive but effective treatments. This lecture was presented at the 142nd Congress of the German Society of Surgery (DCK 2025), held at the Congress Center Munich from 18 to 20 March 2025, in the session HPB International, with the original title "Personalizing surgical strategy in HPB surgery".

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