AlzheimersDisease-Ryantriplettnew.ppt
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Alzheimer's disease is a progressive brain disorder that destroys memory and thinking skills. It is the most common cause of dementia among older adults. The main risk factor is increasing age, and symptoms usually appear after age 65. Alzheimer's damages and kills brain cells, leading to the shrinking of areas of the brain. This results in problems with memory, thinking, and behavior severe enough to affect work and daily living. The three main changes in the brain include loss of connections between neurons, the buildup of amyloid plaques, and tangles within neurons made of the tau protein. Currently, there is no cure for Alzheimer's and treatment aims to temporarily slow the worsening of dementia symptoms.
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Alzheimer's disease is a progressive brain disorder that destroys memory and thinking skills. It is the most common cause of dementia among older adults. The main risk factor is increasing age, and symptoms usually appear after age 65. Alzheimer's causes brain cells to degenerate and die, leading to impaired memory, thinking, and behavior. There is no cure for Alzheimer's, but medications and care strategies can temporarily slow the worsening of symptoms. Researchers are working to better understand the causes and develop more effective treatments.Alzheimer's disease 
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Alzheimer's disease is a progressive brain disorder that destroys memory and thinking skills. It is the most common cause of dementia among older adults. The main risk factor is age, with most cases occurring in people over 65 years of age. Pathologically, it is characterized by amyloid plaques and neurofibrillary tangles in the brain that lead to the loss of connections between neurons and ultimately the death of neurons. Currently, there is no cure for Alzheimer's, and available treatments can only temporarily slow the worsening of dementia symptoms. Researchers are studying new therapies targeting the underlying causes and progression of the disease.Major neuropathological conditions
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- Symptoms start with mild memory loss but progress to include problems with thinking, reasoning, language and perception as brain regions are damaged.
Alzheimer
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Alzheimer's disease is an irreversible progressive brain disorder that causes brain cells to degenerate and die. It is the most common cause of dementia, which is a continuous decline in thinking, behavioral, and social skills that disrupts independent functioning. The disease is named after Dr. Alois Alzheimer, who first identified characteristic brain changes of abnormal clumps and tangled bundles of fibers in a woman who had died of an unusual mental illness. Alzheimer's progresses through mild, moderate, and severe stages, and causes memory loss, mood changes, difficulty communicating, and other cognitive declines. While the exact causes are unknown, age and genetics are major risk factors. Current treatments cannot cure the disease but aim to slow symptoms and maintain quality
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This document provides information about Alzheimer's disease, including:
- It is a progressive brain disorder that causes degeneration and death of brain cells, resulting in cognitive decline and impaired functioning.
- The causes are not entirely known but include age, family history, genetic factors like Down's syndrome, abnormal protein deposits in the brain, and other risk factors.
- It was first identified by Dr. Alois Alzheimer in 1901 from examining the brain of a woman with unusual mental symptoms including memory loss and behavioral changes.
- Diagnosis involves mental status tests, neuropsychological testing, imaging scans, and other medical exams to evaluate cognitive abilities and rule out other conditions.
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Alzheimer's disease is a progressive brain disorder that causes memory loss and cognitive decline. It was first described by Alois Alzheimer in 1906 after examining brain tissue from a deceased patient. The disease results from the buildup of beta-amyloid plaques and tau protein tangles in the brain, which damage and kill neurons. Risk factors include age, family history, and genetic factors. There is no cure for Alzheimer's, but medications and caregiving can temporarily ease symptoms.Alzheimer's Disease and its pathophysiology
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Dementia is a general term for the decline in mental ability severe enough to interfere with daily life. Alzheimer's disease is the most common cause of dementia, accounting for 50-70% of cases. It is a neurodegenerative disease characterized by plaque and tangles in the brain that damage synapses and neural transmission. Symptoms of Alzheimer's disease include progressive memory loss, cognitive decline, and personality changes.Alzheimers presentation...........
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An X-ray generator is a crucial device used in medical imaging, industry, and research to produce X-rays. It operates by accelerating electrons toward a metal target, generating X-ray radiation. Key components include the X-ray tube, transformer assembly, rectifier system, and high-tension circuits. Various types, such as single-phase, three-phase, constant potential, and high-frequency generators, offer different efficiency levels. High-frequency generators are the most advanced, providing stable, high-quality imaging with minimal radiation exposure. X-ray generators play a vital role in diagnostics, security screening, and industrial testing while requiring strict radiation safety measures.physiology 1 T3T4 & Jaundice & capillary circulation 愕悗悋.pptx
physiology 1 T3T4 & Jaundice & capillary circulation 愕悗悋.pptxamralmohammady27
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惺惆 悋惡 惠惡 悖 惠悋惡惠 悋
power point show
惺 悴惆悋 惘悋悴惺悸 愕惘惺悸 悸 悋悋惠忰悋
悋 惆惘 惺 忰悋悴悸 惺悋
愆惘悋 惆惠惘悸 悋 惺 惠悴惺悸 悖愕悧悸 悋惡Pharm test bank- 12th lehne pharmacology nursing class
Pharm test bank- 12th lehne pharmacology nursing classkoxoyav221
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A pediatric nursing course is designed to prepare nursing students to provide specialized care for infants, children, and adolescents. The course integrates developmental, physiological, and psychological aspects of pediatric health and illness, emphasizing family-centered care. Below is a detailed breakdown of what you can expect in a pediatric nursing course:
1. Course Overview
Focuses on growth and development, health promotion, and disease prevention.
Covers common pediatric illnesses and conditions.
Emphasizes family dynamics, cultural competence, and ethical considerations in pediatric care.
Integrates clinical skills, including medication administration, assessment, and communication with children and families.
2. Key Topics Covered
A. Growth and Development
Neonates (0-28 days): Reflexes, feeding patterns, thermoregulation.
Infants (1 month - 1 year): Milestones, immunization schedule, nutrition.
Toddlers (1-3 years): Language development, toilet training, injury prevention.
Preschoolers (3-5 years): Cognitive and social development, school readiness.
School-age children (6-12 years): Psychosocial development, peer relationships.
Adolescents (13-18 years): Puberty, identity formation, risk-taking behaviors.
B. Pediatric Assessment
Head-to-toe assessment in children (differences from adults).
Vital signs (normal ranges vary by age).
Pain assessment using age-appropriate scales (FLACC, Wong-Baker, Numeric).
C. Pediatric Disease Conditions
Respiratory disorders: Asthma, bronchiolitis, pneumonia, cystic fibrosis.
Cardiac conditions: Congenital heart defects, Kawasaki disease.
Neurological disorders: Seizures, meningitis, cerebral palsy.
Gastrointestinal disorders: GERD, pyloric stenosis, intussusception.
Endocrine conditions: Diabetes mellitus type 1, congenital hypothyroidism.
Hematologic disorders: Sickle cell anemia, hemophilia, leukemia.
Infectious diseases: Measles, mumps, rubella, chickenpox.
Mental health concerns: Autism spectrum disorder, ADHD, eating disorders.
D. Pediatric Pharmacology
Medication administration (oral, IV, IM, subcutaneous).
Weight-based dosing calculations (mg/kg).
Common pediatric medications (antibiotics, analgesics, vaccines).
Parenteral nutrition and fluid management.
E. Pediatric Emergency & Critical Care
Pediatric Advanced Life Support (PALS) basics.
Recognizing signs of deterioration (early vs. late signs).
Shock, dehydration, respiratory distress management.
F. Family-Centered Care & Communication
Parental involvement in care decisions.
Therapeutic communication with children at different developmental stages.
Cultural considerations in pediatric care.
G. Ethical and Legal Issues in Pediatric Nursing
Informed consent for minors.
Mandatory reporting of abuse and neglect.
Palliative care and end-of-life considerations in pediatrics.
3. Clinical Component
Hands-on experience in pediatric hospital units, clinics, or community settings.
Performing assessments and interventions under supervision.
Case study discStability of Dosage Forms as per ICH Guidelines
Stability of Dosage Forms as per ICH GuidelinesKHUSHAL CHAVAN
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This presentation covers the stability testing of pharmaceutical dosage forms according to ICH guidelines (Q1A-Q1F). It explains the definition of stability, various testing protocols, storage conditions, and evaluation criteria required for regulatory submissions. Key topics include stress testing, container closure systems, stability commitment, and photostability testing. The guidelines ensure that pharmaceutical products maintain their identity, purity, strength, and efficacy throughout their shelf life. This resource is valuable for pharmaceutical professionals, researchers, and regulatory experts.Hingula.ppt- Rasadravya parichaya- NCISM
Hingula.ppt- Rasadravya parichaya- NCISMShri Shivayogeeshwar Rural Ayurvedic Medical college, INCHAL,
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Hingula(Cinnabar)
Synonyms - Churna Parada, Darada, Mleccha, Rasagarbha,Rasodbhava,
Shukatunda, Hamsapada, Lohagna
Mineralogical Identification
Chemical Formula- HgS
Chemical name- mercury sulfide
Nature Crystalline
Colour- Bright Red
Crystal- Trigonal.
Clevage- -
Diaphaneity- Transparent to Translucent
Fracture- Uneven
Tenacity- Brittle
Lustre- Vitreous
Streak- Red
Sp. Gravity- 8 to 8.1
Hardness- 2 to 2.5
Sources
- It is found near volcanic region and near mercury mines(extracted).
- It is associated with other minerals like Stibnite, pyrite, realgar, calcite etc.
- Almaden (Spain), California, Texas.
Types
1) Charmara
2) Shukhatunda
3) Hamsapada ( Ap 2/69)
1) Khanija
2) Kritrima (RT 9/4)
Grahya ad Agrahyata
爐爐爐 爐爛爐伍爐 爐朽ぐ爛爐爐鉦き: 爐爛爐劇ぃ 爐伍爐爐爛爐項ぐ: |
爐爐項爐爛爐朽ぞ爐迦 爐爐鉦ぐ爐爛爐萎爐爛 爐項た爐爐爛爐: 爐謹爐萎爐劇爐 爐爐爛爐爐む || RT 9/3
Doshas
Andya( Blindness)
Kshinata
Klama
Bhrama
Moha
Prameha ( AP 2/73)
Shodhana
1) Grahya Hingula ChurnaArdraka rasa bhavana(7)--Shuddhahingula (RT 9/12)
Marana
Other Processing Techniques
Satwa Patana
-Shuddha HingulaAdhaha patana/Urdhwa patana -Satva (Parada) (RRS 3/144)
Yogas
Hinguleshwar rasa
Anandabhairava Rasa,
Mrutunjaya Rasa,
Tribhuvana keerti Rasa, etc.
Research Updates
A CONCEPTUAL REVIEW ON HINGULA (CINNABAR- HgS)
https://ijapr.in/index.php/ijapr/article/view/1237
EXTRACTION OF PARAD FROM HINGULA, A TRADITIONAL AYURVEDIC METHOD
https://www.researchgate.net/publication/323601866_EXTRACTION_OF_PARAD_FROM_HINGULA_A_TRADITIONAL_AYURVEDIC_METHOD
DrMahantesh.B.Rudrapuri,
M.D.(Ayu) FAGE , PGDYT
HOD Department of Rasa Shastra&BhaishajyaKalpana
Shri Shivayogeeshwar Rural Ayurvedic Medical College,
INCHAL 591 102, Dist: Belgaum
Mob: 9972710790
HER2-Targeting Therapy in HER2+ MBC With and Without CNS Metastases: Selectio...
HER2-Targeting Therapy in HER2+ MBC With and Without CNS Metastases: Selectio...PVI, PeerView Institute for Medical Education
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Chair and Presenters Sara A. Hurvitz, MD, FACP, Carey K. Anders, MD, FASCO, and Vyshak Venur, MD, discuss metastatic HER2-positive breast cancer in this CME/NCPD/CPE/AAPA/IPCE activity titled Fine-Tuning the Selection and Sequencing of HER2-Targeting Therapies in HER2-Positive MBC With and Without CNS Metastases: Expert Guidance on How to Individualize Therapy Based on Latest Evidence, Disease Features, Treatment Characteristics, and Patient Needs and Preferences. For the full presentation, downloadable Practice Aids, and complete CME/NCPD/CPE/AAPA/IPCE information, and to apply for credit, please visit us at https://bit.ly/4f8sUs7. CME/NCPD/CPE/AAPA/IPCE credit will be available until March 2, 2026.Hingula.ppt- Rasadravya parichaya- NCISM
Hingula.ppt- Rasadravya parichaya- NCISMShri Shivayogeeshwar Rural Ayurvedic Medical college, INCHAL,
油
HER2-Targeting Therapy in HER2+ MBC With and Without CNS Metastases: Selectio...
HER2-Targeting Therapy in HER2+ MBC With and Without CNS Metastases: Selectio...PVI, PeerView Institute for Medical Education
油
AlzheimersDisease-Ryantriplettnew.ppt
- 1. Alzheimers Disease By: Ryan Triplett
- 2. Alzheimers The deterioration of intellectual capabilities, memory, judgment, and personality to the extent that daily functioning and quality of life are seriously impaired. Generally occurs in the elderly impairing brain function, which can lead to dementia. Named for German neurologist Alois Alzheimer in 1907.
- 3. Statistics 4 million or more cases in U.S. 100,000 die each year. 4th major leading cause of death in U.S. 65 or older when symptoms can begin
- 4. Clinical Features Loss of short-term memory and ability to create memories Concentration on past Loss of time Communication diminishes Personality changes Delusions Become immobilized and uncomprehending Death due to respiratory failure 65 and up disease lasts 8-20 years 65 and down disease lasts 5-10 years disease more rapid
- 5. Histological Analysis 3 distinctive neuropathological features 1. Devastating losses of synapses and neurons within hippocampus and entorhinal cortex. 2. Dense spherical structures, called senile plaques (SP), prevalent outside the neurons of the hippocampus and other regions of the brain. 3. Aggregations of fibrils (Neurofibrillary tangles, NFT) accumulate within cell bodies and dendritic processes of the neurons of the hippocampus, neurocortex, entorhinal cortex, and other brain parts.
- 6. Pictures of Brain Degradation
- 8. Senile Plaques Densely packed fibrous structures called amyloid bodies. Consists of mainly protein, 4-kDa peptide. Many isoforms make up the amyloid proteins. Amyloid precursor protein another source of which can lead to AD. Functional role unknown.
- 9. Neurofibrillary Tangles Consist of helical filaments called PHF. Intertwined protein strands made of tau protein molecules. Formation of NFTs not unique to AD. Found in many other neurodegenerative disorders with dementias, affecting the brain. Scientists believe that the amyloid proteins lead to the formation of the neurofibrillary tangles, and both can lead to AD symptoms.
- 10. Diagnosis Only definitive way is to use brain scans (CT) to see plaques or tangles in brain tissue. Tests used to exclude other diseases.
- 11. Treatments No treatment can prevent Alzheimers Drugs for early stages Cognax, Aricept, Exelon, or Razadyne Severe stages Memantine (Namenda) Medicines used to control symptoms, allow caregivers to provide easier care.
- 12. Research Neuroimaging Finding damaged parts of the brain Alzheimers Genetics Anti-oxidants Ginkgo biloba Using to stimulate memory Estrogen Tested for levels found in AD patients, which are women
- 13. References Pasternak, J. Jack. Introduction to Molecular Genetics. 2nd edition.2005. pg. 403-408. www.bic.ucs.edu/images/alz3d.jpg www.ahaf.org www.nia.nih.gov/Alzheimers/Publications