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CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC
ENZYME STUDIES, ECG MONITORING, HOLTER
MONITORING, STRESS STUDY,
ECHOCARDIOGRAM, CORONARY ANGIOGRAPHY
PRESENTED BY
SATHESHWARI N
II YEAR MSC (N)
THE HEART
Heart is hollow four chamber, muscular pump organ
 Pericardium outer layer
 Pericardial sac in between parietal and visceral
layer ,containing 5-20cc of fluid, protects myocardium
 Myocardium makes up the walls of the heart chambers 
left ventricle is 5-10 times thicker than right side
 Inner endocardium are the cardiac valves and blood
vessels.
 The intraventricular and intraatrial septa separates the
right and left chambers
CONTD
 Atrioventricular valves- tricuspid and mitral valves
 Semilunar valves- pulmonic and aortic valves
VASCULATURE
Right coronary artery, left coronary artery, great cardiac
veins, anterior cardiac veins, besian veins
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
CONDUCTION SYSTEM
ELECTRICAL CONDUCTION PROPERITIES
 Automaticity ( pace maker ability)
 Conductivity (Each cell has the ability to conduct impulses
to tnext cell)
 Contractility (ability to contract)
 Irritability (each cell has the ability to contrct on its own)
BLOOD FLOW IN THE HEART
BLOOD PRESSURE
 BP is determined by the cardiac output, the velocity, the
resistance of the blood vessels.
 Systolic- initial force of contraction of heart
 Diastolic-pressure of blood vessels after initial force
 Pulse pressure- difference between systolic and diastolic
BP
AUTONOMIC NERVOUS SYSTEM
 Control of HR, CO, blood vessels and the amount of
blood volume
 Sympathetic secretes norepinephrine and innervate
cardiac plexus and increase rate of SA node
 Parasympathetic secretes acetylcholine and innervates
cardiac plexus leads to decrease of SA node rate
 Both regulates normal heart rate and blood pressure
CARDIAC ASSESSMENT:
HISTORY AND CHIEF COMPLAINTS
 Prior history collection the nurse should know the data of
the patient
 Read old charts or summary : previous admission, current
and past drugs, reason of admission, social support,
allergies, discharge information, chronic medical
problems
 Ask symptoms of present illness
 Maintain a soothing conversation
 Dont rush the patient to answer
 Ask the patient to describe illness finally
 Record response and can ask more details if necessary
 Ask for specific cardiac conditions
 Find out any cardiac drugs patient is taking
 Assess for any use of alcohol or nicotin
 Assess the social factors
 Record the findings
ASSESSMENT
INSPECTION
Explain the procedure to the patient
Provide privacy and undress the patient
Sit upright to inspect thorax
Inspect posture and symmetry, color of skin, deformities
of bone, the neck, face and eyes
Breathing pattern to be noted
Aware of cyanosis
Central cyanosis -lips mouth and conjunctiva indicates
heart disease and poor oxygenation
Peripheral cyanosis- lips, ear lobes, nail buds
PMI (point of maximum intensity): ask to lay on left side
and assess skin color of thorax
EYES: Arcus senilis- light gray ring surrounding iris
common in older patient. If in younger patient indicate
lipid metabolism disorder.
Xanthelasma- yellowish plaques on skin surrounding the
eyes ,also in elbow indicate hypercholesterolemia
PALPATION
SKIN: frank edema ,puffiness, pitting edema
+1,+2,+3,+4.(feet, ankle, face , sacrum, trunk, abdomen)
BREATHING: lay hands on chest at different location ,
feel -respiration pattern , rib elevation, precordial pulses
ARTERIES: apical HR, radial, carotid, brachial, femoral,
popliteal, posterior tibialis, dorsalis pedis pulses. Check
pulse alternans for radial pulse
CAROTID ARTERY: plateau pulse (slow rise, slow
collapsed- aortic stenosis), decrease amplitude, bounding
pulse (HT, thyrotoxicosis)
0= absent
+1= diminished
+2= normal
+3= full pulse or slight increase
+4= bounding pulse
CENTRAL VENOUS PRESSURE: ask the patient to sit in bed then
lean backward at 45 degree angle, relax for few seconds. Look for
internal jugular vein , visible pulsation at the level of sternal notch,
- Pulsation > 3 cm shows elevation of CVP- indicate right heart
failure
- Normal CVP= 5 to 12 cm H2O , measured by placing a catheter
into large vein and attach to manometer
- Hepato jugular reflex- placing the hand in right upper quadrant of
abdomen with firm pressure for one full minute and observe jugular
vein. If its pulsation is high then CVP is high
CLUBBING OF FINGERS AND TOES: diffuse, bulbous
enlargement of the finger and toe tips. Nails appear shiny and
curves downward , loss of normal angulations due prolonged
hypoxemia
JUGULAR VEINS:
- Venous pulsation feels by gentle pressure
- Carotid pulse requires firm pressure
- Venous pulse descends upon inspiration and rises in
expiration, usually collapse in sitting position
- Carotid pulse unaffected
Venous pulse
HAIR: feel the consistency and texture of hair
- Very fine hair shafts  hyperthyroidism
- Very coarse hair shafts- hypothyroidism
- Both have adverse effect on cardiovascular system
PERCUSSION
On precordial area of chest percussion-listening for
resonant sound indicates normal tissue beneath the
fingers
Lung tissue- resonant, semi hollow , medium pitched
sound will be flat or dull in pitch
ASCULTATION
 lub and dub two major sounds called as S1 and S2
respectively
The time between S1 and S2 is shorter than the time
between S2 and beginning of next cycle (S1 of the next
beat)
The time between S1 and S2 corresponds systolic
pressure of cardiac cycle
Additional heart sounds S3 and S4. S3- early rapid
diastolic filling of ventricles. S4 last heart sound
S1- closing of mitral and tricuspid valve
S2- closing of aortic and pulmonic valves
Physiological or pathological spitting is the valves closed
in different timings, one valve closes slightly slower than
another valve.
To auscultate make the patient relax comfortably, and
ascultate the areas
Auscultation land marks
MURMURS: abnormal heart sound due turbulent or rapid
blood flow via heart, greater blood vessels and heart
valves.
-It is a forward blood flow of constricted valve or dilated
chamber
-and also backflow of blood in incompetent valve- rushing
or swooshing sound
-record timing , characteristics (loudness, intensity, pitch,
quality), location and radiation of murmur
GALLOPS: S3 and S4 are generally low pitched sounds
and are heard best by using bell in the stethoscope. S3
ventricular gallop- sound of blood prematurely rushing into
ventricle
CLICKS: extra sounds heard in mitral valve prolapse,
aortic stenosis, prosthetic heart valve. Opening snaps
caused by mitral / tricuspid stenosis
RUBS: visceral and parietal layer of pericardium rub
together due to inflammation in uremic pericarditis, MI,
inflammatory condition
HEART SOUNDS
 Systolic murmur- between S1 and S2
 Pansystolic or holosystolic murmur- throughout the
systolic S1 to S2 phase
 Diastolic murmur- between S2 and S1
 holodiastolic murmur- throughout the systolic S2 to S1
phase
 Murmurs  early systolic, mid systolic, early diastolic, mid
diastolic, late systolic, late diastolic
 Mitral stenosis  loud S1
 Bundle branch block- split S1 sound
 Pulmonic stenosis, ASD  split S2 sound
 Hypertension  loud S2 sound
 Chart the basic information  HR, rate , rhythm, intensity,
abnormal sounds, location, loudness , pitch
CARDIAC ENZYME STUDIES
Used since mid 20th
century with MI suspects
Troponin is widely used  elevation within 2-3 hours of MI
Creatinine kinase  elevation in 6-12 hours
Elevation of enzymes interpreted with ECG findings
INDICATIONS
 ACS
 Routinely following PCI
 CABG
 MI
TROPONINS T AND I
 Troponins are protein released from myocytes in
irreversible myocardial damage
 Troponin dependent on infarct size
 Lower level troponin assay used to know non ST elevated
MI
 Troponin level increases within 3-12 hours from the onset
of damage, peak at 24- 48 hours, return to baseline 5-14
days.
 Normal troponin= 0 and 0.4 ng/ml
 Troponin should be measured at presentation and again
10-12 hrs from the onset of symptoms
 Elevated troponin without ACS  CHF, Pulmonary
embolism, sepsis, CKD, myocarditis, aortic dissection ,
often higher after thrombolytic therapy
CREATINE KINASE
 CK MB increases with 3-12 hours, reach peak within 24
hours, return to baseline 48-72 hrs
 Normal value 22- 198 U/L
MYOGLOBIN
 Found in cardiac and skeletal muscle
 Released more rapidly than other enzymes i.e. within 2
hours
 Normal value-= 25-72 ng/ml
NATRIURETRIC PEPTIDES
 ACS shows elevated level B type natriuretic peptide BNP
 NORMAL < 100 micograms/ ml
OTHERS
 Leucocytosis peaks 2-4 days of MI and returns normal
within one week
 Increased C Reactive Protein CRP . Normal <10 mg/ L
 ESR values rises within 3 days and elevated for week.
Normal 0-22mm/hr
FUTURE DEVELOPMENT
 Heart type fatty acid binding protein
 Mid regional pro atrial natriuretic peptide
 ST2
 Growth differentiation factor 15
 C terminal pro- endothelia 1
ECG MONITORING
ECG meaning
 Electro- electricity
 Cardio- heart
 Graph- to write
 It is a trans thoracic interpretation of electrical activity of
the heart overtime captured and externally recorded by
skin electrodes
 The device used is ELECTROCARDIOGRAPH
INDICATIONS
 Detect electrolyte imbalance
 Conduction abnormalities
 Ischemic heart disease
 Non cardiac diseases( eg. pulmonary embolism ,
hypothermia)
ECG TIME LINE
 1872- Alexander Muirhead- attach wires to patient wrist
 1887- Augustus publishes first human ECG, heartbeat
projected on photographic plate
 1893-Einthoven introduce electrocardiogram
 1895- distinguish 5 deflection P,Q,R,S,T
 1902- first ECG record on string galvanometer
 1912- addresses equilateral triangle formed by standard
leads I, II, III called Einthovens triangle
 1924- Einthoven won Noble prize
DEPOLARIZATION
 During the rise of potential the membrane becomes
permeable to sodium ions and potential rise to positive
direction.
 Depolarization is followed by muscle contraction
REPOLARIZATION
 The sodium channel close and there is a rapid diffusion of
K ions into exterior, re establishing resting membrane
potential.
 Repolarization is followed by muscle relaxation
.
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
U wave
RECORDING OF ECG
ECG LEAD PLACEMENT
.
MAKING ECG REPORTING
 ECG strip should be correctly labelled with patient data
and lead markings, should contain
- Heart rate
- Rhythm
- Various conduction intervals
- QRS complex, ST segment , T wave
- Cardiac axis
- Any abnormal wave
HEART RATE CALCULATION
 Sinus tachycardia HR> 100 b/ mt
 Sinus bradycardia HR< 60 b/mt
 Rhythm controlled by sinus node at a rate of 60-100 b/mt
Disturbance of SA node
HR 250-350 b/ mt
Rapid and uncordinated ventricular depolarization
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
.
ST ELEVATION
Acute myocardial infarction
Pericarditis
Left Ventricular Hypertrophy
Left Bundle Branch Block
hyperkalemia
ST DEPRESSION
Ischemic heart disease
Hypokalemia
Secondary ST segment changes with conduction abnormalities
o RBBB,
o LBBB,
o WPW
o hypokalemia
ABNORMAL ECG FINDINGS
Myocardial Ischaemia
 T inversion, ST depression, U wave inversion
Acute Myocardial Infarction
 Hyperacute T waves, ST elevation, new LBBB, TWI, Q waves
Pericarditis
 ST elevation, PR depression
Pericardial Effusion
 Sinus tachycardia, low QRS voltage, QRS alternans
Hyperkalaemia
 Tented T waves, long PR, wide QRS, short QT
Hypokalaemia
 PR prolongation, ST depression, flat T waves, U waves
Hypercalcaemia
 Shortened QT interval, J waves
Hypocalcaemia
 Prolonged QT interval
Brugada Syndrome
Wolff-Parkinson-White Syndrome
 Short PR interval, delta wave, wide QRS
Congenital Long QT Syndrome
Arrhythmogenic Right Ventricular Dysplasia
 Epsilon waves in V1-V3; localised QRS widening and TWI
Catecholaminergic Polymorphic VT
Polymorphic VT with alternating QRS morphology
Early Repolarisation Syndrome
 J-point elevation, J-point notching
Sodium Channel Blockade
 QRS widening, ventricular arrhythmias
Digoxin Toxicity
 Scooped ST depression, T wave flattening / inversion
Pulmonary Embolism
 Sinus tachycardia, RBBB, S1 Q3 T3
Acute Stroke
 ST depression, prolonged QT, T wave inversion
Hypothermia
 Movement artefact, Osborn waves, bradyarrhythmias
HOLTER MONITORING
 The Holter monitor study is a type of
ambulatory electrocardiographic (ECG) monitoring. It is named
for Norman J. Holter, the physicist chiefly responsible for its
invention in the 1950s.
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
 Holter monitor study is to analyze the
electrical activity of the heart outside of the clinical setting that
is, as a person goes about his or her normal daily activities.
 When a person has a Holter monitor study, they wear the
monitoring device for either 24 or 48 hours, and the ECG
recorded during this time is subsequently analyzed for any
cardiac arrhythmias that might have occurred during the
monitoring period, as well as for any signs of cardiac ischemia.
PURPOSES
 if a person is suspected to have an arrhythmia, it is important to
diagnose the precise nature of the arrhythmia in order to decide
 The odds of capturing one of these rare or fleeting arrhythmias
while a doctor happens to be recording a standard ECG (which
records the heart rhythm for only 12 seconds), is quite small.
 The Holter monitor was developed to address the problem of
diagnosing infrequent or fleeting cardiac arrhythmias. By
recording each and every heartbeat that occurs over a
prolonged period of time, while a person goes about their
activities of daily life
WHEN SHOULD DONE
Who have transient episodes
 Syncope
 Unexplained light headedness
 palpitations
RISK AND CONTRA INDICATION
 Free of risk, except skin irritation due to electrodes
 It can used for longer weeks to be implement
BEFORE THE TEST
 24-48hrs period
 Patient is not travelling, not doing water activities, not
making body sweat
 Done in outside clinic by technician
 A comfortable loose clothing should be wear
 Can eat and drink normally prior to test
 Bath before the study
DURING THE TEST
 Men should be shaved to remove hairs to attach patch
 Instruct the patient dos and donts
 Keep diary with him
 Normal routine can be followed
 Note the time he experience symptoms
 He will return to lab to remove holter
AFTER THE PROCEDURE
 Can meet the doctor within one or two weeks
 Doctor plan about further treatment
INTERPRETATION
 Many people (most people, in fact) have occasional, benign
types of arrhythmias that do not cause any symptoms at all.
Seeing such an arrhythmia on the Holter report, without
simultaneous symptoms, indicates that this arrhythmia
is not causing a problem, and (usually) does not require any
treatment of further evaluation.
 On the other hand, when symptoms are well-correlated with a
cardiac arrhythmia, thats an arrhythmia that is causing a
problem (at the very least, it is producing symptoms), and that
deserves to be addressed.
 The doctor may also discuss with the other results shown on
the Holter monitor report, including the maximum, minimum,
and average heart rate, the total number of
premature atrial complexes (PACs) and
premature ventricular complexes (PVCs) that the person may
have had, and any episodes of possible ischemia.
 If a person is having a Holter study to look for silent ischemia,
finding strong signs of ischemia on the test will likely lead
either to further testing (perhaps with a nuclear stress test or a
cardiac catheterization), or to a change in your anti-ischemia
therapy.
STRESS TEST
 An exercise stress test is used to determine how well the
individual heart responds during times when its working its
hardest.
 During the test, the person will be asked to exercise typically
on a treadmill while he hooked up to an
electrocardiogram (EKG) machine. This allows the doctor to
monitor the individual heart rate.
 The exercise stress test is also referred to as an exercise test or
treadmill test.
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
PURPOSE
 Determine person heart receives enough oxygen
 Order for people experiencing chest pain
 Determine the level of health
 To know the risk of heart disease
RISK
RARE RISK
Chest pain
Collapsing
Fainting
Heart attack
Irregular HR
PREPERATION
 Perform history collection and physical examination
 Assess for stiff joints
 Check blood sugar
 Wear loose dress
 NPO three hours before procedure
 Stop certain drugs
 Report any chest pains
DURING PROCEDURE
 Attached to EKG machine
 Start off walking slowly
 Watch for difficulties
POST PROCEDURE
 Give water and make patient rest
 If results reveal arrhythmias plan for cardiologist
consultation
ECHO
Echocardiography is a test that uses sound waves to produce
live images of the heart. The image is an echocardiogram. This
test allows the doctor to monitor how the heart and its valves
are functioning. The images can help them spot:
blood clots in the heart
fluid in the sac around the heart
problems with the aorta, which is the main artery connected
to the heart
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
USES
 To inspect heart valves , chambers
 Check hearts ability to pump
TYPES
 Transthoracic ECHO
 Transesophageal ECHO
 Stress ECHO
 Three dimensional ECHO
 Fetal ECHO
RISKS
 Very safe
 TRANSTHORACIC-discomfort of leads
 TRANSESOPHAGEAL- esophageal scrap
 Medication and exercise increases HR
PREPERATION
 TRANSESOPHAGEAL- 3hrs NPO
 STRESS ECHO- loose cloth
AFTER ECHO
 If any abnormalities found out , patient refers to
cardiologist for further treatment
CORONARY ANGIOGRAPHY
 Coronary Angiography is a procedure that uses a special dye
[contrast material ] and x- rays to see how contrast material
filled blood flows through the coronary arteries of the heart.
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
IMPORTANCE
 Coronary angiography is the gold standard for the evaluation of
coronary artery disease.
 It can be used to identify the exact location and severity of
coronary artery disease.
INDICATIONS
 Acute MI
 Unstable Angina
 Chronic stable angina
 Abnormal stress test
 Ventricular arrythmias
 Left ventricular dysfunction
 Valvular heart disease
 Pre operative coronary assessment
 Periodic follow up after cardiac
transplantation
CONTRA INDICATIONS
 Coagulopathy
 Active bleeding
 Malignant hypertension
 Acute or chronic renal failure
 Electrolyte imbalance
 Fever
 Active systemic infection
 Transient ischemic attack
BEFORE PROCEDURE
 Explain procedure and get consent
 Record vital signs
 Blood tests- BUN, Cr, PT,PTT,INR, Blood glucose level
 ECG must take
 Skin preparation
 IV access
 Remove ornaments
 Check puncture site for allergy and also pulsation
EQUIPMENTS
 Sterile gowns and gloves
 Sterile towels or drapes
 Gauze
 Syringes for heparin, lidocaine, saline flush
 Skin preparation solution
 Needles, scissors, hemostasis
 Catheters and wires
 Medications-fentanyl, heparin, lidocaine, contrast
ACCESS SITES
 Femoral artery
 Radial artery
 Brachial artery
PROCEDURE
 Done with cardiac catheterization
 Will give mild sedation
 Patient lies on X ray table, may be tilted
 Xray cameras move around
 Electrodes on chest monitor HR
 A small amount hair is shaved at access site
 A small incision is made for easy insertion of catheter
 Dye is injected through the catheter- patient feels flushing
and warmth
 As dye moves through can visualize blockages
 Based on report plan for further treatment
 The total procedure take an hour to complete
RISKS
Heart attack
Stroke
Injury to the catheterized artery
Irregular heart rhythms (arrhythmias)
Allergic reactions to the dye or medications used during
the procedure
Kidney damage
Excessive bleeding
Infection
POST PROCEDURE
 Remove the sheath.
 Tight dressing should be done.
 Continous monitoring of vital signs, saturation.
 Check for any bleeding, hematoma.
 Check the pedal pulse.
 Instruct to avoid flexing or hyperextending the affected
extremityfor 12- 24 hours.
 Unless contraindicated encourage oral fluids.
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx
CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx

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CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME.pptx

  • 2. CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC ENZYME STUDIES, ECG MONITORING, HOLTER MONITORING, STRESS STUDY, ECHOCARDIOGRAM, CORONARY ANGIOGRAPHY PRESENTED BY SATHESHWARI N II YEAR MSC (N)
  • 3. THE HEART Heart is hollow four chamber, muscular pump organ Pericardium outer layer Pericardial sac in between parietal and visceral layer ,containing 5-20cc of fluid, protects myocardium Myocardium makes up the walls of the heart chambers left ventricle is 5-10 times thicker than right side Inner endocardium are the cardiac valves and blood vessels. The intraventricular and intraatrial septa separates the right and left chambers
  • 4. CONTD Atrioventricular valves- tricuspid and mitral valves Semilunar valves- pulmonic and aortic valves VASCULATURE Right coronary artery, left coronary artery, great cardiac veins, anterior cardiac veins, besian veins
  • 7. ELECTRICAL CONDUCTION PROPERITIES Automaticity ( pace maker ability) Conductivity (Each cell has the ability to conduct impulses to tnext cell) Contractility (ability to contract) Irritability (each cell has the ability to contrct on its own)
  • 8. BLOOD FLOW IN THE HEART
  • 9. BLOOD PRESSURE BP is determined by the cardiac output, the velocity, the resistance of the blood vessels. Systolic- initial force of contraction of heart Diastolic-pressure of blood vessels after initial force Pulse pressure- difference between systolic and diastolic BP
  • 10. AUTONOMIC NERVOUS SYSTEM Control of HR, CO, blood vessels and the amount of blood volume Sympathetic secretes norepinephrine and innervate cardiac plexus and increase rate of SA node Parasympathetic secretes acetylcholine and innervates cardiac plexus leads to decrease of SA node rate Both regulates normal heart rate and blood pressure
  • 11. CARDIAC ASSESSMENT: HISTORY AND CHIEF COMPLAINTS Prior history collection the nurse should know the data of the patient Read old charts or summary : previous admission, current and past drugs, reason of admission, social support, allergies, discharge information, chronic medical problems Ask symptoms of present illness Maintain a soothing conversation Dont rush the patient to answer Ask the patient to describe illness finally
  • 12. Record response and can ask more details if necessary Ask for specific cardiac conditions Find out any cardiac drugs patient is taking Assess for any use of alcohol or nicotin Assess the social factors Record the findings
  • 13. ASSESSMENT INSPECTION Explain the procedure to the patient Provide privacy and undress the patient Sit upright to inspect thorax Inspect posture and symmetry, color of skin, deformities of bone, the neck, face and eyes Breathing pattern to be noted Aware of cyanosis
  • 14. Central cyanosis -lips mouth and conjunctiva indicates heart disease and poor oxygenation Peripheral cyanosis- lips, ear lobes, nail buds PMI (point of maximum intensity): ask to lay on left side and assess skin color of thorax EYES: Arcus senilis- light gray ring surrounding iris common in older patient. If in younger patient indicate lipid metabolism disorder. Xanthelasma- yellowish plaques on skin surrounding the eyes ,also in elbow indicate hypercholesterolemia
  • 15. PALPATION SKIN: frank edema ,puffiness, pitting edema +1,+2,+3,+4.(feet, ankle, face , sacrum, trunk, abdomen) BREATHING: lay hands on chest at different location , feel -respiration pattern , rib elevation, precordial pulses ARTERIES: apical HR, radial, carotid, brachial, femoral, popliteal, posterior tibialis, dorsalis pedis pulses. Check pulse alternans for radial pulse
  • 16. CAROTID ARTERY: plateau pulse (slow rise, slow collapsed- aortic stenosis), decrease amplitude, bounding pulse (HT, thyrotoxicosis) 0= absent +1= diminished +2= normal +3= full pulse or slight increase +4= bounding pulse
  • 17. CENTRAL VENOUS PRESSURE: ask the patient to sit in bed then lean backward at 45 degree angle, relax for few seconds. Look for internal jugular vein , visible pulsation at the level of sternal notch, - Pulsation > 3 cm shows elevation of CVP- indicate right heart failure - Normal CVP= 5 to 12 cm H2O , measured by placing a catheter into large vein and attach to manometer - Hepato jugular reflex- placing the hand in right upper quadrant of abdomen with firm pressure for one full minute and observe jugular vein. If its pulsation is high then CVP is high
  • 18. CLUBBING OF FINGERS AND TOES: diffuse, bulbous enlargement of the finger and toe tips. Nails appear shiny and curves downward , loss of normal angulations due prolonged hypoxemia JUGULAR VEINS: - Venous pulsation feels by gentle pressure - Carotid pulse requires firm pressure - Venous pulse descends upon inspiration and rises in expiration, usually collapse in sitting position - Carotid pulse unaffected
  • 20. HAIR: feel the consistency and texture of hair - Very fine hair shafts hyperthyroidism - Very coarse hair shafts- hypothyroidism - Both have adverse effect on cardiovascular system
  • 21. PERCUSSION On precordial area of chest percussion-listening for resonant sound indicates normal tissue beneath the fingers Lung tissue- resonant, semi hollow , medium pitched sound will be flat or dull in pitch
  • 22. ASCULTATION lub and dub two major sounds called as S1 and S2 respectively The time between S1 and S2 is shorter than the time between S2 and beginning of next cycle (S1 of the next beat) The time between S1 and S2 corresponds systolic pressure of cardiac cycle Additional heart sounds S3 and S4. S3- early rapid diastolic filling of ventricles. S4 last heart sound
  • 23. S1- closing of mitral and tricuspid valve S2- closing of aortic and pulmonic valves Physiological or pathological spitting is the valves closed in different timings, one valve closes slightly slower than another valve. To auscultate make the patient relax comfortably, and ascultate the areas
  • 25. MURMURS: abnormal heart sound due turbulent or rapid blood flow via heart, greater blood vessels and heart valves. -It is a forward blood flow of constricted valve or dilated chamber -and also backflow of blood in incompetent valve- rushing or swooshing sound -record timing , characteristics (loudness, intensity, pitch, quality), location and radiation of murmur
  • 26. GALLOPS: S3 and S4 are generally low pitched sounds and are heard best by using bell in the stethoscope. S3 ventricular gallop- sound of blood prematurely rushing into ventricle CLICKS: extra sounds heard in mitral valve prolapse, aortic stenosis, prosthetic heart valve. Opening snaps caused by mitral / tricuspid stenosis RUBS: visceral and parietal layer of pericardium rub together due to inflammation in uremic pericarditis, MI, inflammatory condition
  • 27. HEART SOUNDS Systolic murmur- between S1 and S2 Pansystolic or holosystolic murmur- throughout the systolic S1 to S2 phase Diastolic murmur- between S2 and S1 holodiastolic murmur- throughout the systolic S2 to S1 phase Murmurs early systolic, mid systolic, early diastolic, mid diastolic, late systolic, late diastolic Mitral stenosis loud S1
  • 28. Bundle branch block- split S1 sound Pulmonic stenosis, ASD split S2 sound Hypertension loud S2 sound Chart the basic information HR, rate , rhythm, intensity, abnormal sounds, location, loudness , pitch
  • 29. CARDIAC ENZYME STUDIES Used since mid 20th century with MI suspects Troponin is widely used elevation within 2-3 hours of MI Creatinine kinase elevation in 6-12 hours Elevation of enzymes interpreted with ECG findings
  • 30. INDICATIONS ACS Routinely following PCI CABG MI
  • 31. TROPONINS T AND I Troponins are protein released from myocytes in irreversible myocardial damage Troponin dependent on infarct size Lower level troponin assay used to know non ST elevated MI Troponin level increases within 3-12 hours from the onset of damage, peak at 24- 48 hours, return to baseline 5-14 days. Normal troponin= 0 and 0.4 ng/ml
  • 32. Troponin should be measured at presentation and again 10-12 hrs from the onset of symptoms Elevated troponin without ACS CHF, Pulmonary embolism, sepsis, CKD, myocarditis, aortic dissection , often higher after thrombolytic therapy
  • 33. CREATINE KINASE CK MB increases with 3-12 hours, reach peak within 24 hours, return to baseline 48-72 hrs Normal value 22- 198 U/L
  • 34. MYOGLOBIN Found in cardiac and skeletal muscle Released more rapidly than other enzymes i.e. within 2 hours Normal value-= 25-72 ng/ml
  • 35. NATRIURETRIC PEPTIDES ACS shows elevated level B type natriuretic peptide BNP NORMAL < 100 micograms/ ml
  • 36. OTHERS Leucocytosis peaks 2-4 days of MI and returns normal within one week Increased C Reactive Protein CRP . Normal <10 mg/ L ESR values rises within 3 days and elevated for week. Normal 0-22mm/hr
  • 37. FUTURE DEVELOPMENT Heart type fatty acid binding protein Mid regional pro atrial natriuretic peptide ST2 Growth differentiation factor 15 C terminal pro- endothelia 1
  • 38. ECG MONITORING ECG meaning Electro- electricity Cardio- heart Graph- to write
  • 39. It is a trans thoracic interpretation of electrical activity of the heart overtime captured and externally recorded by skin electrodes The device used is ELECTROCARDIOGRAPH
  • 40. INDICATIONS Detect electrolyte imbalance Conduction abnormalities Ischemic heart disease Non cardiac diseases( eg. pulmonary embolism , hypothermia)
  • 41. ECG TIME LINE 1872- Alexander Muirhead- attach wires to patient wrist 1887- Augustus publishes first human ECG, heartbeat projected on photographic plate 1893-Einthoven introduce electrocardiogram 1895- distinguish 5 deflection P,Q,R,S,T 1902- first ECG record on string galvanometer 1912- addresses equilateral triangle formed by standard leads I, II, III called Einthovens triangle 1924- Einthoven won Noble prize
  • 42. DEPOLARIZATION During the rise of potential the membrane becomes permeable to sodium ions and potential rise to positive direction. Depolarization is followed by muscle contraction
  • 43. REPOLARIZATION The sodium channel close and there is a rapid diffusion of K ions into exterior, re establishing resting membrane potential. Repolarization is followed by muscle relaxation
  • 44. .
  • 49. .
  • 50. MAKING ECG REPORTING ECG strip should be correctly labelled with patient data and lead markings, should contain - Heart rate - Rhythm - Various conduction intervals - QRS complex, ST segment , T wave - Cardiac axis - Any abnormal wave
  • 52. Sinus tachycardia HR> 100 b/ mt Sinus bradycardia HR< 60 b/mt Rhythm controlled by sinus node at a rate of 60-100 b/mt
  • 55. Rapid and uncordinated ventricular depolarization
  • 57. .
  • 58. ST ELEVATION Acute myocardial infarction Pericarditis Left Ventricular Hypertrophy Left Bundle Branch Block hyperkalemia
  • 59. ST DEPRESSION Ischemic heart disease Hypokalemia
  • 60. Secondary ST segment changes with conduction abnormalities o RBBB, o LBBB, o WPW o hypokalemia
  • 61. ABNORMAL ECG FINDINGS Myocardial Ischaemia T inversion, ST depression, U wave inversion Acute Myocardial Infarction Hyperacute T waves, ST elevation, new LBBB, TWI, Q waves Pericarditis ST elevation, PR depression Pericardial Effusion Sinus tachycardia, low QRS voltage, QRS alternans
  • 62. Hyperkalaemia Tented T waves, long PR, wide QRS, short QT Hypokalaemia PR prolongation, ST depression, flat T waves, U waves Hypercalcaemia Shortened QT interval, J waves Hypocalcaemia Prolonged QT interval
  • 63. Brugada Syndrome Wolff-Parkinson-White Syndrome Short PR interval, delta wave, wide QRS Congenital Long QT Syndrome Arrhythmogenic Right Ventricular Dysplasia Epsilon waves in V1-V3; localised QRS widening and TWI Catecholaminergic Polymorphic VT Polymorphic VT with alternating QRS morphology Early Repolarisation Syndrome J-point elevation, J-point notching
  • 64. Sodium Channel Blockade QRS widening, ventricular arrhythmias Digoxin Toxicity Scooped ST depression, T wave flattening / inversion Pulmonary Embolism Sinus tachycardia, RBBB, S1 Q3 T3 Acute Stroke ST depression, prolonged QT, T wave inversion Hypothermia Movement artefact, Osborn waves, bradyarrhythmias
  • 65. HOLTER MONITORING The Holter monitor study is a type of ambulatory electrocardiographic (ECG) monitoring. It is named for Norman J. Holter, the physicist chiefly responsible for its invention in the 1950s.
  • 67. Holter monitor study is to analyze the electrical activity of the heart outside of the clinical setting that is, as a person goes about his or her normal daily activities.
  • 68. When a person has a Holter monitor study, they wear the monitoring device for either 24 or 48 hours, and the ECG recorded during this time is subsequently analyzed for any cardiac arrhythmias that might have occurred during the monitoring period, as well as for any signs of cardiac ischemia.
  • 69. PURPOSES if a person is suspected to have an arrhythmia, it is important to diagnose the precise nature of the arrhythmia in order to decide The odds of capturing one of these rare or fleeting arrhythmias while a doctor happens to be recording a standard ECG (which records the heart rhythm for only 12 seconds), is quite small.
  • 70. The Holter monitor was developed to address the problem of diagnosing infrequent or fleeting cardiac arrhythmias. By recording each and every heartbeat that occurs over a prolonged period of time, while a person goes about their activities of daily life
  • 71. WHEN SHOULD DONE Who have transient episodes Syncope Unexplained light headedness palpitations
  • 72. RISK AND CONTRA INDICATION Free of risk, except skin irritation due to electrodes It can used for longer weeks to be implement
  • 73. BEFORE THE TEST 24-48hrs period Patient is not travelling, not doing water activities, not making body sweat Done in outside clinic by technician A comfortable loose clothing should be wear Can eat and drink normally prior to test Bath before the study
  • 74. DURING THE TEST Men should be shaved to remove hairs to attach patch Instruct the patient dos and donts Keep diary with him Normal routine can be followed Note the time he experience symptoms He will return to lab to remove holter
  • 75. AFTER THE PROCEDURE Can meet the doctor within one or two weeks Doctor plan about further treatment
  • 76. INTERPRETATION Many people (most people, in fact) have occasional, benign types of arrhythmias that do not cause any symptoms at all. Seeing such an arrhythmia on the Holter report, without simultaneous symptoms, indicates that this arrhythmia is not causing a problem, and (usually) does not require any treatment of further evaluation.
  • 77. On the other hand, when symptoms are well-correlated with a cardiac arrhythmia, thats an arrhythmia that is causing a problem (at the very least, it is producing symptoms), and that deserves to be addressed.
  • 78. The doctor may also discuss with the other results shown on the Holter monitor report, including the maximum, minimum, and average heart rate, the total number of premature atrial complexes (PACs) and premature ventricular complexes (PVCs) that the person may have had, and any episodes of possible ischemia.
  • 79. If a person is having a Holter study to look for silent ischemia, finding strong signs of ischemia on the test will likely lead either to further testing (perhaps with a nuclear stress test or a cardiac catheterization), or to a change in your anti-ischemia therapy.
  • 80. STRESS TEST An exercise stress test is used to determine how well the individual heart responds during times when its working its hardest. During the test, the person will be asked to exercise typically on a treadmill while he hooked up to an electrocardiogram (EKG) machine. This allows the doctor to monitor the individual heart rate. The exercise stress test is also referred to as an exercise test or treadmill test.
  • 82. PURPOSE Determine person heart receives enough oxygen Order for people experiencing chest pain Determine the level of health To know the risk of heart disease
  • 84. PREPERATION Perform history collection and physical examination Assess for stiff joints Check blood sugar Wear loose dress NPO three hours before procedure Stop certain drugs Report any chest pains
  • 85. DURING PROCEDURE Attached to EKG machine Start off walking slowly Watch for difficulties POST PROCEDURE Give water and make patient rest If results reveal arrhythmias plan for cardiologist consultation
  • 86. ECHO Echocardiography is a test that uses sound waves to produce live images of the heart. The image is an echocardiogram. This test allows the doctor to monitor how the heart and its valves are functioning. The images can help them spot: blood clots in the heart fluid in the sac around the heart problems with the aorta, which is the main artery connected to the heart
  • 88. USES To inspect heart valves , chambers Check hearts ability to pump
  • 89. TYPES Transthoracic ECHO Transesophageal ECHO Stress ECHO Three dimensional ECHO Fetal ECHO
  • 90. RISKS Very safe TRANSTHORACIC-discomfort of leads TRANSESOPHAGEAL- esophageal scrap Medication and exercise increases HR
  • 91. PREPERATION TRANSESOPHAGEAL- 3hrs NPO STRESS ECHO- loose cloth
  • 92. AFTER ECHO If any abnormalities found out , patient refers to cardiologist for further treatment
  • 93. CORONARY ANGIOGRAPHY Coronary Angiography is a procedure that uses a special dye [contrast material ] and x- rays to see how contrast material filled blood flows through the coronary arteries of the heart.
  • 96. IMPORTANCE Coronary angiography is the gold standard for the evaluation of coronary artery disease. It can be used to identify the exact location and severity of coronary artery disease.
  • 97. INDICATIONS Acute MI Unstable Angina Chronic stable angina Abnormal stress test Ventricular arrythmias
  • 98. Left ventricular dysfunction Valvular heart disease Pre operative coronary assessment Periodic follow up after cardiac transplantation
  • 99. CONTRA INDICATIONS Coagulopathy Active bleeding Malignant hypertension Acute or chronic renal failure Electrolyte imbalance
  • 100. Fever Active systemic infection Transient ischemic attack
  • 101. BEFORE PROCEDURE Explain procedure and get consent Record vital signs Blood tests- BUN, Cr, PT,PTT,INR, Blood glucose level ECG must take Skin preparation IV access Remove ornaments Check puncture site for allergy and also pulsation
  • 102. EQUIPMENTS Sterile gowns and gloves Sterile towels or drapes Gauze Syringes for heparin, lidocaine, saline flush
  • 103. Skin preparation solution Needles, scissors, hemostasis Catheters and wires Medications-fentanyl, heparin, lidocaine, contrast
  • 104. ACCESS SITES Femoral artery Radial artery Brachial artery
  • 105. PROCEDURE Done with cardiac catheterization Will give mild sedation Patient lies on X ray table, may be tilted Xray cameras move around Electrodes on chest monitor HR A small amount hair is shaved at access site A small incision is made for easy insertion of catheter
  • 106. Dye is injected through the catheter- patient feels flushing and warmth As dye moves through can visualize blockages Based on report plan for further treatment The total procedure take an hour to complete
  • 107. RISKS Heart attack Stroke Injury to the catheterized artery Irregular heart rhythms (arrhythmias) Allergic reactions to the dye or medications used during the procedure Kidney damage Excessive bleeding Infection
  • 108. POST PROCEDURE Remove the sheath. Tight dressing should be done. Continous monitoring of vital signs, saturation. Check for any bleeding, hematoma. Check the pedal pulse. Instruct to avoid flexing or hyperextending the affected extremityfor 12- 24 hours. Unless contraindicated encourage oral fluids.