Heart is hollow four chamber, muscular pump organ
Pericardium outer layer
Pericardial sac in between parietal and visceral layer ,containing 5-20cc of fluid, protects myocardium
Myocardium makes up the walls of the heart chambers left ventricle is 5-10 times thicker than right side
Inner endocardium are the cardiac valves and blood vessels.
The intraventricular and intraatrial septa separates the right and left chambers
Atrioventricular valves- tricuspid and mitral valves
Semilunar valves- pulmonic and aortic valves
VASCULATURE
Right coronary artery, left coronary artery, great cardiac veins, anterior cardiac veins, besian veins
Automaticity ( pace maker ability)
Conductivity (Each cell has the ability to conduct impulses to tnext cell)
Contractility (ability to contract)
Irritability (each cell has the ability to contrct on its own)
BP is determined by the cardiac output, the velocity, the resistance of the blood vessels.
Systolic- initial force of contraction of heart
Diastolic-pressure of blood vessels after initial force
Pulse pressure- difference between systolic and diastolic BP
Control of HR, CO, blood vessels and the amount of blood volume
Sympathetic secretes norepinephrine and innervate cardiac plexus and increase rate of SA node
Parasympathetic secretes acetylcholine and innervates cardiac plexus leads to decrease of SA node rate
Both regulates normal heart rate and blood pressure
Prior history collection the nurse should know the data of the patient
Read old charts or summary : previous admission, current and past drugs, reason of admission, social support, allergies, discharge information, chronic medical problems
Ask symptoms of present illness
Maintain a soothing conversation
Dont rush the patient to answer
Ask the patient to describe illness finally
Record response and can ask more details if necessary
Ask for specific cardiac conditions
Find out any cardiac drugs patient is taking
Assess for any use of alcohol or nicotin
Assess the social factors
Record the findings
INSPECTION
Explain the procedure to the patient
Provide privacy and undress the patient
Sit upright to inspect thorax
Inspect posture and symmetry, color of skin, deformities of bone, the neck, face and eyes
Breathing pattern to be noted
Aware of cyanosis
Central cyanosis -lips mouth and conjunctiva indicates heart disease and poor oxygenation
Peripheral cyanosis- lips, ear lobes, nail buds
PMI (point of maximum intensity): ask to lay on left side and assess skin color of thorax
EYES: Arcus senilis- light gray ring surrounding iris common in older patient. If in younger patient indicate lipid metabolism disorder.
Xanthelasma- yellowish plaques on skin surrounding the eyes ,also in elbow indicate hypercholesterolemia
PALPATION
SKIN: frank edema ,puffiness, pitting edema +1,+2,+3,+4.(feet, ankle, face , sacrum, trunk, abdomen)
BREATHING: lay hands on chest at different location , feel -respiration pattern , rib elevation, precordial pulses
ARTERIES: apical
2. CARDIAC ASSESSMENT, HEART SOUNDS, CARDIAC
ENZYME STUDIES, ECG MONITORING, HOLTER
MONITORING, STRESS STUDY,
ECHOCARDIOGRAM, CORONARY ANGIOGRAPHY
PRESENTED BY
SATHESHWARI N
II YEAR MSC (N)
3. THE HEART
Heart is hollow four chamber, muscular pump organ
Pericardium outer layer
Pericardial sac in between parietal and visceral
layer ,containing 5-20cc of fluid, protects myocardium
Myocardium makes up the walls of the heart chambers
left ventricle is 5-10 times thicker than right side
Inner endocardium are the cardiac valves and blood
vessels.
The intraventricular and intraatrial septa separates the
right and left chambers
4. CONTD
Atrioventricular valves- tricuspid and mitral valves
Semilunar valves- pulmonic and aortic valves
VASCULATURE
Right coronary artery, left coronary artery, great cardiac
veins, anterior cardiac veins, besian veins
7. ELECTRICAL CONDUCTION PROPERITIES
Automaticity ( pace maker ability)
Conductivity (Each cell has the ability to conduct impulses
to tnext cell)
Contractility (ability to contract)
Irritability (each cell has the ability to contrct on its own)
9. BLOOD PRESSURE
BP is determined by the cardiac output, the velocity, the
resistance of the blood vessels.
Systolic- initial force of contraction of heart
Diastolic-pressure of blood vessels after initial force
Pulse pressure- difference between systolic and diastolic
BP
10. AUTONOMIC NERVOUS SYSTEM
Control of HR, CO, blood vessels and the amount of
blood volume
Sympathetic secretes norepinephrine and innervate
cardiac plexus and increase rate of SA node
Parasympathetic secretes acetylcholine and innervates
cardiac plexus leads to decrease of SA node rate
Both regulates normal heart rate and blood pressure
11. CARDIAC ASSESSMENT:
HISTORY AND CHIEF COMPLAINTS
Prior history collection the nurse should know the data of
the patient
Read old charts or summary : previous admission, current
and past drugs, reason of admission, social support,
allergies, discharge information, chronic medical
problems
Ask symptoms of present illness
Maintain a soothing conversation
Dont rush the patient to answer
Ask the patient to describe illness finally
12. Record response and can ask more details if necessary
Ask for specific cardiac conditions
Find out any cardiac drugs patient is taking
Assess for any use of alcohol or nicotin
Assess the social factors
Record the findings
13. ASSESSMENT
INSPECTION
Explain the procedure to the patient
Provide privacy and undress the patient
Sit upright to inspect thorax
Inspect posture and symmetry, color of skin, deformities
of bone, the neck, face and eyes
Breathing pattern to be noted
Aware of cyanosis
14. Central cyanosis -lips mouth and conjunctiva indicates
heart disease and poor oxygenation
Peripheral cyanosis- lips, ear lobes, nail buds
PMI (point of maximum intensity): ask to lay on left side
and assess skin color of thorax
EYES: Arcus senilis- light gray ring surrounding iris
common in older patient. If in younger patient indicate
lipid metabolism disorder.
Xanthelasma- yellowish plaques on skin surrounding the
eyes ,also in elbow indicate hypercholesterolemia
15. PALPATION
SKIN: frank edema ,puffiness, pitting edema
+1,+2,+3,+4.(feet, ankle, face , sacrum, trunk, abdomen)
BREATHING: lay hands on chest at different location ,
feel -respiration pattern , rib elevation, precordial pulses
ARTERIES: apical HR, radial, carotid, brachial, femoral,
popliteal, posterior tibialis, dorsalis pedis pulses. Check
pulse alternans for radial pulse
17. CENTRAL VENOUS PRESSURE: ask the patient to sit in bed then
lean backward at 45 degree angle, relax for few seconds. Look for
internal jugular vein , visible pulsation at the level of sternal notch,
- Pulsation > 3 cm shows elevation of CVP- indicate right heart
failure
- Normal CVP= 5 to 12 cm H2O , measured by placing a catheter
into large vein and attach to manometer
- Hepato jugular reflex- placing the hand in right upper quadrant of
abdomen with firm pressure for one full minute and observe jugular
vein. If its pulsation is high then CVP is high
18. CLUBBING OF FINGERS AND TOES: diffuse, bulbous
enlargement of the finger and toe tips. Nails appear shiny and
curves downward , loss of normal angulations due prolonged
hypoxemia
JUGULAR VEINS:
- Venous pulsation feels by gentle pressure
- Carotid pulse requires firm pressure
- Venous pulse descends upon inspiration and rises in
expiration, usually collapse in sitting position
- Carotid pulse unaffected
20. HAIR: feel the consistency and texture of hair
- Very fine hair shafts hyperthyroidism
- Very coarse hair shafts- hypothyroidism
- Both have adverse effect on cardiovascular system
21. PERCUSSION
On precordial area of chest percussion-listening for
resonant sound indicates normal tissue beneath the
fingers
Lung tissue- resonant, semi hollow , medium pitched
sound will be flat or dull in pitch
22. ASCULTATION
lub and dub two major sounds called as S1 and S2
respectively
The time between S1 and S2 is shorter than the time
between S2 and beginning of next cycle (S1 of the next
beat)
The time between S1 and S2 corresponds systolic
pressure of cardiac cycle
Additional heart sounds S3 and S4. S3- early rapid
diastolic filling of ventricles. S4 last heart sound
23. S1- closing of mitral and tricuspid valve
S2- closing of aortic and pulmonic valves
Physiological or pathological spitting is the valves closed
in different timings, one valve closes slightly slower than
another valve.
To auscultate make the patient relax comfortably, and
ascultate the areas
25. MURMURS: abnormal heart sound due turbulent or rapid
blood flow via heart, greater blood vessels and heart
valves.
-It is a forward blood flow of constricted valve or dilated
chamber
-and also backflow of blood in incompetent valve- rushing
or swooshing sound
-record timing , characteristics (loudness, intensity, pitch,
quality), location and radiation of murmur
26. GALLOPS: S3 and S4 are generally low pitched sounds
and are heard best by using bell in the stethoscope. S3
ventricular gallop- sound of blood prematurely rushing into
ventricle
CLICKS: extra sounds heard in mitral valve prolapse,
aortic stenosis, prosthetic heart valve. Opening snaps
caused by mitral / tricuspid stenosis
RUBS: visceral and parietal layer of pericardium rub
together due to inflammation in uremic pericarditis, MI,
inflammatory condition
27. HEART SOUNDS
Systolic murmur- between S1 and S2
Pansystolic or holosystolic murmur- throughout the
systolic S1 to S2 phase
Diastolic murmur- between S2 and S1
holodiastolic murmur- throughout the systolic S2 to S1
phase
Murmurs early systolic, mid systolic, early diastolic, mid
diastolic, late systolic, late diastolic
Mitral stenosis loud S1
29. CARDIAC ENZYME STUDIES
Used since mid 20th
century with MI suspects
Troponin is widely used elevation within 2-3 hours of MI
Creatinine kinase elevation in 6-12 hours
Elevation of enzymes interpreted with ECG findings
31. TROPONINS T AND I
Troponins are protein released from myocytes in
irreversible myocardial damage
Troponin dependent on infarct size
Lower level troponin assay used to know non ST elevated
MI
Troponin level increases within 3-12 hours from the onset
of damage, peak at 24- 48 hours, return to baseline 5-14
days.
Normal troponin= 0 and 0.4 ng/ml
32. Troponin should be measured at presentation and again
10-12 hrs from the onset of symptoms
Elevated troponin without ACS CHF, Pulmonary
embolism, sepsis, CKD, myocarditis, aortic dissection ,
often higher after thrombolytic therapy
33. CREATINE KINASE
CK MB increases with 3-12 hours, reach peak within 24
hours, return to baseline 48-72 hrs
Normal value 22- 198 U/L
34. MYOGLOBIN
Found in cardiac and skeletal muscle
Released more rapidly than other enzymes i.e. within 2
hours
Normal value-= 25-72 ng/ml
35. NATRIURETRIC PEPTIDES
ACS shows elevated level B type natriuretic peptide BNP
NORMAL < 100 micograms/ ml
36. OTHERS
Leucocytosis peaks 2-4 days of MI and returns normal
within one week
Increased C Reactive Protein CRP . Normal <10 mg/ L
ESR values rises within 3 days and elevated for week.
Normal 0-22mm/hr
37. FUTURE DEVELOPMENT
Heart type fatty acid binding protein
Mid regional pro atrial natriuretic peptide
ST2
Growth differentiation factor 15
C terminal pro- endothelia 1
39. It is a trans thoracic interpretation of electrical activity of
the heart overtime captured and externally recorded by
skin electrodes
The device used is ELECTROCARDIOGRAPH
41. ECG TIME LINE
1872- Alexander Muirhead- attach wires to patient wrist
1887- Augustus publishes first human ECG, heartbeat
projected on photographic plate
1893-Einthoven introduce electrocardiogram
1895- distinguish 5 deflection P,Q,R,S,T
1902- first ECG record on string galvanometer
1912- addresses equilateral triangle formed by standard
leads I, II, III called Einthovens triangle
1924- Einthoven won Noble prize
42. DEPOLARIZATION
During the rise of potential the membrane becomes
permeable to sodium ions and potential rise to positive
direction.
Depolarization is followed by muscle contraction
43. REPOLARIZATION
The sodium channel close and there is a rapid diffusion of
K ions into exterior, re establishing resting membrane
potential.
Repolarization is followed by muscle relaxation
50. MAKING ECG REPORTING
ECG strip should be correctly labelled with patient data
and lead markings, should contain
- Heart rate
- Rhythm
- Various conduction intervals
- QRS complex, ST segment , T wave
- Cardiac axis
- Any abnormal wave
60. Secondary ST segment changes with conduction abnormalities
o RBBB,
o LBBB,
o WPW
o hypokalemia
61. ABNORMAL ECG FINDINGS
Myocardial Ischaemia
T inversion, ST depression, U wave inversion
Acute Myocardial Infarction
Hyperacute T waves, ST elevation, new LBBB, TWI, Q waves
Pericarditis
ST elevation, PR depression
Pericardial Effusion
Sinus tachycardia, low QRS voltage, QRS alternans
62. Hyperkalaemia
Tented T waves, long PR, wide QRS, short QT
Hypokalaemia
PR prolongation, ST depression, flat T waves, U waves
Hypercalcaemia
Shortened QT interval, J waves
Hypocalcaemia
Prolonged QT interval
63. Brugada Syndrome
Wolff-Parkinson-White Syndrome
Short PR interval, delta wave, wide QRS
Congenital Long QT Syndrome
Arrhythmogenic Right Ventricular Dysplasia
Epsilon waves in V1-V3; localised QRS widening and TWI
Catecholaminergic Polymorphic VT
Polymorphic VT with alternating QRS morphology
Early Repolarisation Syndrome
J-point elevation, J-point notching
64. Sodium Channel Blockade
QRS widening, ventricular arrhythmias
Digoxin Toxicity
Scooped ST depression, T wave flattening / inversion
Pulmonary Embolism
Sinus tachycardia, RBBB, S1 Q3 T3
Acute Stroke
ST depression, prolonged QT, T wave inversion
Hypothermia
Movement artefact, Osborn waves, bradyarrhythmias
65. HOLTER MONITORING
The Holter monitor study is a type of
ambulatory electrocardiographic (ECG) monitoring. It is named
for Norman J. Holter, the physicist chiefly responsible for its
invention in the 1950s.
67. Holter monitor study is to analyze the
electrical activity of the heart outside of the clinical setting that
is, as a person goes about his or her normal daily activities.
68. When a person has a Holter monitor study, they wear the
monitoring device for either 24 or 48 hours, and the ECG
recorded during this time is subsequently analyzed for any
cardiac arrhythmias that might have occurred during the
monitoring period, as well as for any signs of cardiac ischemia.
69. PURPOSES
if a person is suspected to have an arrhythmia, it is important to
diagnose the precise nature of the arrhythmia in order to decide
The odds of capturing one of these rare or fleeting arrhythmias
while a doctor happens to be recording a standard ECG (which
records the heart rhythm for only 12 seconds), is quite small.
70. The Holter monitor was developed to address the problem of
diagnosing infrequent or fleeting cardiac arrhythmias. By
recording each and every heartbeat that occurs over a
prolonged period of time, while a person goes about their
activities of daily life
71. WHEN SHOULD DONE
Who have transient episodes
Syncope
Unexplained light headedness
palpitations
72. RISK AND CONTRA INDICATION
Free of risk, except skin irritation due to electrodes
It can used for longer weeks to be implement
73. BEFORE THE TEST
24-48hrs period
Patient is not travelling, not doing water activities, not
making body sweat
Done in outside clinic by technician
A comfortable loose clothing should be wear
Can eat and drink normally prior to test
Bath before the study
74. DURING THE TEST
Men should be shaved to remove hairs to attach patch
Instruct the patient dos and donts
Keep diary with him
Normal routine can be followed
Note the time he experience symptoms
He will return to lab to remove holter
75. AFTER THE PROCEDURE
Can meet the doctor within one or two weeks
Doctor plan about further treatment
76. INTERPRETATION
Many people (most people, in fact) have occasional, benign
types of arrhythmias that do not cause any symptoms at all.
Seeing such an arrhythmia on the Holter report, without
simultaneous symptoms, indicates that this arrhythmia
is not causing a problem, and (usually) does not require any
treatment of further evaluation.
77. On the other hand, when symptoms are well-correlated with a
cardiac arrhythmia, thats an arrhythmia that is causing a
problem (at the very least, it is producing symptoms), and that
deserves to be addressed.
78. The doctor may also discuss with the other results shown on
the Holter monitor report, including the maximum, minimum,
and average heart rate, the total number of
premature atrial complexes (PACs) and
premature ventricular complexes (PVCs) that the person may
have had, and any episodes of possible ischemia.
79. If a person is having a Holter study to look for silent ischemia,
finding strong signs of ischemia on the test will likely lead
either to further testing (perhaps with a nuclear stress test or a
cardiac catheterization), or to a change in your anti-ischemia
therapy.
80. STRESS TEST
An exercise stress test is used to determine how well the
individual heart responds during times when its working its
hardest.
During the test, the person will be asked to exercise typically
on a treadmill while he hooked up to an
electrocardiogram (EKG) machine. This allows the doctor to
monitor the individual heart rate.
The exercise stress test is also referred to as an exercise test or
treadmill test.
82. PURPOSE
Determine person heart receives enough oxygen
Order for people experiencing chest pain
Determine the level of health
To know the risk of heart disease
84. PREPERATION
Perform history collection and physical examination
Assess for stiff joints
Check blood sugar
Wear loose dress
NPO three hours before procedure
Stop certain drugs
Report any chest pains
85. DURING PROCEDURE
Attached to EKG machine
Start off walking slowly
Watch for difficulties
POST PROCEDURE
Give water and make patient rest
If results reveal arrhythmias plan for cardiologist
consultation
86. ECHO
Echocardiography is a test that uses sound waves to produce
live images of the heart. The image is an echocardiogram. This
test allows the doctor to monitor how the heart and its valves
are functioning. The images can help them spot:
blood clots in the heart
fluid in the sac around the heart
problems with the aorta, which is the main artery connected
to the heart
88. USES
To inspect heart valves , chambers
Check hearts ability to pump
92. AFTER ECHO
If any abnormalities found out , patient refers to
cardiologist for further treatment
93. CORONARY ANGIOGRAPHY
Coronary Angiography is a procedure that uses a special dye
[contrast material ] and x- rays to see how contrast material
filled blood flows through the coronary arteries of the heart.
96. IMPORTANCE
Coronary angiography is the gold standard for the evaluation of
coronary artery disease.
It can be used to identify the exact location and severity of
coronary artery disease.
97. INDICATIONS
Acute MI
Unstable Angina
Chronic stable angina
Abnormal stress test
Ventricular arrythmias
98. Left ventricular dysfunction
Valvular heart disease
Pre operative coronary assessment
Periodic follow up after cardiac
transplantation
100. Fever
Active systemic infection
Transient ischemic attack
101. BEFORE PROCEDURE
Explain procedure and get consent
Record vital signs
Blood tests- BUN, Cr, PT,PTT,INR, Blood glucose level
ECG must take
Skin preparation
IV access
Remove ornaments
Check puncture site for allergy and also pulsation
102. EQUIPMENTS
Sterile gowns and gloves
Sterile towels or drapes
Gauze
Syringes for heparin, lidocaine, saline flush
105. PROCEDURE
Done with cardiac catheterization
Will give mild sedation
Patient lies on X ray table, may be tilted
Xray cameras move around
Electrodes on chest monitor HR
A small amount hair is shaved at access site
A small incision is made for easy insertion of catheter
106. Dye is injected through the catheter- patient feels flushing
and warmth
As dye moves through can visualize blockages
Based on report plan for further treatment
The total procedure take an hour to complete
107. RISKS
Heart attack
Stroke
Injury to the catheterized artery
Irregular heart rhythms (arrhythmias)
Allergic reactions to the dye or medications used during
the procedure
Kidney damage
Excessive bleeding
Infection
108. POST PROCEDURE
Remove the sheath.
Tight dressing should be done.
Continous monitoring of vital signs, saturation.
Check for any bleeding, hematoma.
Check the pedal pulse.
Instruct to avoid flexing or hyperextending the affected
extremityfor 12- 24 hours.
Unless contraindicated encourage oral fluids.