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El documento describe las siete cavidades de la cara, incluyendo la cavidad bucal, las fosas nasales, las cavidades orbitarias, y la fosa pterigomaxilar. Detalla la ubicaci坦n y estructura de cada cavidad, as鱈 como los huesos que forman sus paredes. Tambi辿n describe las partes de la cavidad oral y sus funciones principales como la masticaci坦n, la salivaci坦n, el sentido del gusto, el habla y la degluci坦n.
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La ATM est叩 compuesta de huesos, m炭sculos y ligamentos que permiten el movimiento de la mand鱈bula. Incluye el c坦ndilo de la mand鱈bula, la cavidad glenoidea del hueso temporal y el disco articular entre ellos. La ATM permite los movimientos de apertura y cierre de la boca, as鱈 como protrusi坦n y retrusi坦n de la mand鱈bula. Es la articulaci坦n m叩s compleja del cuerpo humano.
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El documento describe la anatom鱈a dental. Los dientes est叩n formados por tejidos como el esmalte, dentina, cemento y pulpa. Cada diente tiene una corona visible y una ra鱈z oculta unida al hueso alveolar por el ligamento periodontal. Los dientes se clasifican en incisivos, caninos, premolares y molares seg炭n su forma y funci坦n en la masticaci坦n. El sistema dentario forma parte del aparato masticatorio junto con las mand鱈bulas, m炭sculos y articulaciones temporomaxilares.
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There are few stresses to which the body is exposed that even nearly approach the extreme stresses of heavy exercise. In fact, if some of the extremes of exercise were continued for even moderately prolonged periods, they might be lethal. Therefore, in the main, sports physiology is a discussion of the ultimate limits to which several of the bodily mechanisms can be stressed. To give one simple example: In a person who has extremely high fever approaching the level of lethality, the body metabolism increases to about 100 per cent above normal. By comparison, the metabolism of the body during a marathon race may increase to 2000 per cent above normal.
Female and Male Athletes Most of the quantitative data that are given for the young male athlete, not because it is desirable to know only these values but because it is only in male athletes that relatively complete measurements have been made. However, for those measurements that have been made in the female athlete, almost identical basic physiologic principles apply, except for quantitative differences caused by differences in body size, body composition, and the presence or absence of the male sex hormone testosterone.
In general, most quantitative values for womensuch as muscle strength, pulmonary ventilation, and cardiac output, all of which are related mainly to the muscle massvary between two thirds and three quarters of the values recorded in men. When measured in terms of strength per square centimetre of cross-sectional area, the female muscle can achieve almost exactly the same maximal force of contraction as that of the male-between 3 and 4 kg/cm2. Therefore, most of the difference in total muscle performance lies in the extra percentage of the male body that is muscle, caused by endocrine differences that we discuss later.
The performance capabilities of the female versus the male athlete are illustrated by the relative running speeds for a marathon race. In a recent comparison, the top female performer had a running speed that was 11 per cent less than that of the top male performer. For other events, however, women have at times held records faster than menfor instance, for the two-way swim across the English Channel, where the availability of extra fat seems to be an advantage for heat insulation, buoyancy, and extra long-term energy.
Testosterone secreted by the male testes has a powerful anabolic effect in causing greatly increased deposition of protein everywhere in the body, but especially in the muscles. In fact, even a male who participates in very little sports activity but who nevertheless is well endowed with testosterone will have muscles that grow about 40 per cent larger than those of a comparable female without the testosterone.
The female sex hormone estrogen probably also accounts for some of the difference between female and male performance, although not nearly so much as testosterone. Estrogen is known to increase the deposition of fat in theSYNOVITIS.DEFINITION ,CAUSES ,SYMPTOMS AND TREATMENT
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Description
Synovial fluid is a transparent, viscid fluid secreted by the synovial membrane and found in joint cavities, bursae, and tendon sheaths.
Analysis of synovial fluid aspirated from a joint can confirm or rule out various joint diseases such as traumatic arthritis, osteoarthritis, gout, and rheumatoid arthritis.
Causes
Synovitis Causes
Synovitis is a major problem in rheumatoid arthritis, in juvenile arthritis, in lupus, and in psoriatic arthritis. It may also be associated with rheumatic fever, tuberculosis, trauma, or gout.
Rheumatoid arthritis involves synovitis. In rheumatoid arthritis, the synovial membrane lining the joint becomes inflamed. The cells in the membrane divide and grow and inflammatory cells come into the joint from other parts of the body.
Symptoms
Symptoms of Synovitis
Because of the mass of inflammatory cells in rheumatoid arthritis, the joint appears swollen and feels puffy or boggy to the touch. The increased blood flow that is a feature of the inflammation makes the joint warm. The cells release enzymes into the joint space which causes further pain and irritation. If the process continues for years, the enzymes may gradually digest the cartilage and bone of the joint leading to chronic pain and degenerative changes.
Diagnosis
Diagnosing Synovitis
In addition to the clinical presentation (warm, red, and swollen joints), the diagnosis may be helped by Synovial Fluid Analysis. This is a test that examines the lubricating fluid secreted by synovial membranes. The test is useful in the diagnosis of some types of arthritis (primarily those caused by infection, gout, or pseudogout).
The test takes about half an hour and usually is done in a physician's office or hospital. No special preparations are necessary. The skin over the joint is cleaned with an antiseptic. Usually, a local anesthetic is injected. Using a thin needle, the physician will withdraw a sample of fluid for analysis, including culture of the fluid if infection is a possible diagnosis, and examination for crystals to diagnose gout or pseudogout.
If needed, medication (usually a corticosteroid preparation) can be injected into the joint space through this needle after the specimen is taken.
Treatment
Synovitis Treatment
Synovitis is treated with anti-inflammatory drugs ranging from aspirin to ibuprofen to corticosteroids. Specific treatment is based upon both the presumed cause of the synovitis and the particular patient's response and level of tolerance of any particular medication.
Prevention of Synovitis
Here are ways to prevent synovitis:
Regular Exercise: Engage in low-impact exercises (e.g., swimming, cycling) to keep joints flexible and strong.
Proper Joint Protection: Use protective gear during sports or high-risk activities to prevent injury to the joints.
Maintain Healthy Weight: Achieving and maintaining an optimal weight helps reduce stress on weight-bearing joints, especially the knees and hips.
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