This document describes various dental anomalies involving abnormalities of the dental pulp, alterations in tooth number, size and shape. It discusses conditions such as tooth resorption, pulp calcifications, anodontia, supernumerary teeth, macrodontia, microdontia, fusion and others. Diagrams and radiographs are provided to illustrate different pathological conditions, including physiologic root resorption, idiopathic resorption, impacted teeth, odontomes and mesiodens. The document serves as a reference for dental students to learn about anomalies affecting the teeth, pulp and jaws.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
middle layer of tooth the dentin which has yellowish in colorRenu710209
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dentin is the resilient structure of tooth which gives yellowish color and protect the underlying dentalpulp and innervated structures from external stimuli
This document discusses dental trauma classifications and management of avulsed teeth. It outlines 9 classes of dental injuries from fractures to tooth displacement. Avulsion, the complete displacement of a tooth, is most common in maxillary teeth of children ages 7-9 years. Prompt reimplantation within 15-20 minutes maximizes success. Complications of reimplantation include ankylosis and inflammatory root resorption. Splinting and antibiotics can reduce complications and promote healing of pulp and periodontal ligament. Regular follow up is needed to monitor healing and detect any issues.
This document discusses root resorption, including its history, types, causes, pathogenesis, classification, and treatment. It describes internal resorption in detail, noting that it begins with a breach in the dentin layer that allows resorption to spread towards the cementum. Internal resorption can be inflammatory or replacement, and treatment involves root canal therapy to remove pulpal tissue and arrest resorption, as well as disinfecting and sealing the root canal system. For large defects, biocompatible materials like MTA or Biodentine may be used to fill the area.
This document provides an overview of pulp therapy procedures for permanent and young primary teeth. It discusses the histology and structural elements of the dental pulp, as well as the reactions of pulp to dental caries and operative procedures. Indirect and direct pulp capping techniques are described, along with the medicaments and materials used. The document also notes the limitations of direct pulp capping in primary teeth and points to consider during these procedures.
This document discusses the structure and properties of dentin. It is composed of 70% hydroxyapatite crystals, 20% collagen, and 10% water. Dentin is lighter yellow in young individuals and darker with age. It is approximately 3-3.5mm thick on coronal surfaces and increases with secondary and tertiary dentin formation. Dentin is one-fifth the hardness of enamel and its hardness increases with age. It contains dentinal tubules that originate from odontoblasts and provide pathways for stimulation of the dental pulp. There are three types of dentin - primary, secondary, and tertiary - which differ in their location, rate of formation, orientation of tubules, and permeability.
alveolar ridge expansion and socket preservationMaherFouda1
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Alveolar bone resorption occurs rapidly after tooth extraction or avulsion, especially in the first 3 years when 40-60% of bone can be lost. Extraction of anterior maxillary teeth is associated with progressive labial bone loss. The causes of alveolar bone resorption are thought to include disuse atrophy, decreased blood supply, localized inflammation, and prosthesis pressure. Immediate implant placement can help preserve the alveolar ridge after tooth extraction.
The document summarizes the process of primary tooth shedding and replacement by permanent teeth. It describes how odontoclasts, cells similar to osteoclasts, initiate root resorption through secretion of acids and enzymes. This causes dissolution of the dental hard tissues and degradation of the organic matrix. Shedding occurs through intermittent periods of root resorption by odontoclasts and recovery periods where tissues are repaired, until the tooth is loosened and lost.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
Genuine work. I hope it helps during the your study.
It is about the alveolar bone as a part of the periodontium and its components together with the binding cells.
Mechanisms of Tooth Eruption & Mammalian Teeth 3 Categories on the Basis of E...Jansen Calibo
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This document discusses tooth eruption, including its definition, phases, and mechanisms. It begins by defining tooth eruption as the movement of developing teeth through bone and gum tissue to emerge in the mouth. It then describes the three phases of eruption: pre-eruptive (tooth development below gum), eruptive (root formation and emergence), and post-eruptive (teeth in function). The mechanisms of eruption involve bodily movement and growth changes. Teeth can be categorized based on their eruptive characteristics as continuously growing, continuously extruding, or continuously erupting.
Dentin is a hard yellowish substance that forms the bulk of teeth. It is composed of 70% hydroxyapatite crystals and 30% organic materials like collagen. Dentin is formed by odontoblasts cells differentiated from dental papilla cells. It determines the shape of teeth and contains microscopic tubules that house the processes of odontoblast cells. Dentin is harder than bone but softer than enamel. It has different layers with varying properties located at different regions of the tooth.
This document summarizes the treatment of traumatic dental injuries in primary and permanent teeth. It discusses that primary teeth are more likely to be displaced than fractured due to thinner bone and root resorption. The effects of injury can be immediate on primary teeth through displacement like intrusion or extrusion, or indirect on permanent teeth through conditions like hypoplasia. Treatment depends on the specific injury and may include repositioning displaced teeth, extraction, or pulp therapy and fillings for fractures. Complications from injuries can include pulp necrosis, discoloration, resorption, or ankylosis. Prompt treatment is important to prevent further issues.
This document discusses delayed tooth eruption (DTE). It defines various types of DTE and outlines the normal process of tooth eruption. It identifies several potential local and systemic causes of DTE, including supernumerary teeth, ankylosed deciduous teeth, premature loss of deciduous teeth, arch length deficiencies, nutritional deficiencies, endocrine disorders, and genetic syndromes. The document also provides details on the typical chronology and sequence of primary and permanent tooth eruption.
Dental hard tissues are resorbed by multinucleate cells called odontoclasts or dentinoclasts. They are classified as physiological or pathological, with pathological further divided into external root resorption due to trauma, pulp/apical pathology, or pressure and internal root resorption. A new clinical classification is based on injury to protective tissues by chemical or mechanical means and stimulation by infection or pressure. Odontoclasts/dentinoclasts resorb dental tissues through a process that begins with injury exposing mineralized tissue, followed by colonization and resorption stimulated continuously by pressure or infection.
The document discusses the formation and types of dentin. It begins by explaining that dentinogenesis is initiated by odontoblasts and forms the bulk of each tooth crown and root. There are several types of dentin that form at different stages: mantle dentin forms first along the enamel layer, primary dentin makes up most of the tooth, and secondary and tertiary dentins are deposited throughout life. Tertiary dentin specifically forms in response to stimuli like decay. Dentin contains tubules that house odontoblast processes and provide sensitivity; it is made up of both organic and inorganic components including collagen and hydroxyapatite.
Dentin /certified fixed orthodontic courses by Indian dental academy Indian dental academy
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Welcome to Indian Dental Academy
The Indian Dental Academy is the Leader in continuing dental education , training dentists in all aspects of dentistry and offering a wide range of dental certified courses in different formats.
Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
State of the art comprehensive training-Faculty of world wide repute &Very affordable.
Tooth resorption is a process by which all parts of a Tooth Structure is lost due to activation of bodys innate capacity to remove mineralized tissue as mediated via cells such as Osteoclast.
1) Orthodontic treatment can be used as an adjunct to periodontal therapy to resolve problems caused by loss of periodontal support like pathologic tooth migration, diastemas, and collapsed posterior occlusion.
2) Orthodontics can improve periodontal health by reducing plaque retention in situations with crowding, tipped teeth, and open contacts. It can also improve gingival and osseous form by uprighting tilted teeth.
3) Orthodontic treatment may facilitate prosthetic replacements and improve aesthetics by correcting diastemas and tooth positions.
This document discusses prospects for tooth regeneration. It begins by reviewing tooth development as a model for regeneration, noting the stages of bud, cap, bell, crown and root. It then discusses the regenerative capabilities of naturally formed dental tissues, noting that enamel has no regenerative ability while dentin, cementum, pulp, and alveolar bone do to varying degrees. The document concludes by discussing postnatal dental stem cells that have been identified and their role in dental tissue regeneration experiments that have generated tooth crowns containing enamel, dentin, pulp and bone.
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This presentation provides an overview of syncope, a common medical emergency in dental practice. Created during my internship, this presentation aims to educate dental students on the causes, symptoms, diagnosis and management of syncope with a focus on dental specific considerations.
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Alveolar bone resorption occurs rapidly after tooth extraction or avulsion, especially in the first 3 years when 40-60% of bone can be lost. Extraction of anterior maxillary teeth is associated with progressive labial bone loss. The causes of alveolar bone resorption are thought to include disuse atrophy, decreased blood supply, localized inflammation, and prosthesis pressure. Immediate implant placement can help preserve the alveolar ridge after tooth extraction.
The document summarizes the process of primary tooth shedding and replacement by permanent teeth. It describes how odontoclasts, cells similar to osteoclasts, initiate root resorption through secretion of acids and enzymes. This causes dissolution of the dental hard tissues and degradation of the organic matrix. Shedding occurs through intermittent periods of root resorption by odontoclasts and recovery periods where tissues are repaired, until the tooth is loosened and lost.
Indian Dental Academy: will be one of the most relevant and exciting training center with best faculty and flexible training programs for dental professionals who wish to advance in their dental practice,Offers certified courses in Dental implants,Orthodontics,Endodontics,Cosmetic Dentistry, Prosthetic Dentistry, Periodontics and General Dentistry.
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Genuine work. I hope it helps during the your study.
It is about the alveolar bone as a part of the periodontium and its components together with the binding cells.
Mechanisms of Tooth Eruption & Mammalian Teeth 3 Categories on the Basis of E...Jansen Calibo
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This document discusses tooth eruption, including its definition, phases, and mechanisms. It begins by defining tooth eruption as the movement of developing teeth through bone and gum tissue to emerge in the mouth. It then describes the three phases of eruption: pre-eruptive (tooth development below gum), eruptive (root formation and emergence), and post-eruptive (teeth in function). The mechanisms of eruption involve bodily movement and growth changes. Teeth can be categorized based on their eruptive characteristics as continuously growing, continuously extruding, or continuously erupting.
Dentin is a hard yellowish substance that forms the bulk of teeth. It is composed of 70% hydroxyapatite crystals and 30% organic materials like collagen. Dentin is formed by odontoblasts cells differentiated from dental papilla cells. It determines the shape of teeth and contains microscopic tubules that house the processes of odontoblast cells. Dentin is harder than bone but softer than enamel. It has different layers with varying properties located at different regions of the tooth.
This document summarizes the treatment of traumatic dental injuries in primary and permanent teeth. It discusses that primary teeth are more likely to be displaced than fractured due to thinner bone and root resorption. The effects of injury can be immediate on primary teeth through displacement like intrusion or extrusion, or indirect on permanent teeth through conditions like hypoplasia. Treatment depends on the specific injury and may include repositioning displaced teeth, extraction, or pulp therapy and fillings for fractures. Complications from injuries can include pulp necrosis, discoloration, resorption, or ankylosis. Prompt treatment is important to prevent further issues.
This document discusses delayed tooth eruption (DTE). It defines various types of DTE and outlines the normal process of tooth eruption. It identifies several potential local and systemic causes of DTE, including supernumerary teeth, ankylosed deciduous teeth, premature loss of deciduous teeth, arch length deficiencies, nutritional deficiencies, endocrine disorders, and genetic syndromes. The document also provides details on the typical chronology and sequence of primary and permanent tooth eruption.
Dental hard tissues are resorbed by multinucleate cells called odontoclasts or dentinoclasts. They are classified as physiological or pathological, with pathological further divided into external root resorption due to trauma, pulp/apical pathology, or pressure and internal root resorption. A new clinical classification is based on injury to protective tissues by chemical or mechanical means and stimulation by infection or pressure. Odontoclasts/dentinoclasts resorb dental tissues through a process that begins with injury exposing mineralized tissue, followed by colonization and resorption stimulated continuously by pressure or infection.
The document discusses the formation and types of dentin. It begins by explaining that dentinogenesis is initiated by odontoblasts and forms the bulk of each tooth crown and root. There are several types of dentin that form at different stages: mantle dentin forms first along the enamel layer, primary dentin makes up most of the tooth, and secondary and tertiary dentins are deposited throughout life. Tertiary dentin specifically forms in response to stimuli like decay. Dentin contains tubules that house odontoblast processes and provide sensitivity; it is made up of both organic and inorganic components including collagen and hydroxyapatite.
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Indian dental academy has a unique training program & curriculum that provides students with exceptional clinical skills and enabling them to return to their office with high level confidence and start treating patients
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Tooth resorption is a process by which all parts of a Tooth Structure is lost due to activation of bodys innate capacity to remove mineralized tissue as mediated via cells such as Osteoclast.
1) Orthodontic treatment can be used as an adjunct to periodontal therapy to resolve problems caused by loss of periodontal support like pathologic tooth migration, diastemas, and collapsed posterior occlusion.
2) Orthodontics can improve periodontal health by reducing plaque retention in situations with crowding, tipped teeth, and open contacts. It can also improve gingival and osseous form by uprighting tilted teeth.
3) Orthodontic treatment may facilitate prosthetic replacements and improve aesthetics by correcting diastemas and tooth positions.
This document discusses prospects for tooth regeneration. It begins by reviewing tooth development as a model for regeneration, noting the stages of bud, cap, bell, crown and root. It then discusses the regenerative capabilities of naturally formed dental tissues, noting that enamel has no regenerative ability while dentin, cementum, pulp, and alveolar bone do to varying degrees. The document concludes by discussing postnatal dental stem cells that have been identified and their role in dental tissue regeneration experiments that have generated tooth crowns containing enamel, dentin, pulp and bone.
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Tertiary Dentin: Reactionary/Response and Reparativ
1. 悋愆惆悋惆 惶悋忰 惺惡惆悋 悴惆 惺惡惆悋 : 悋悋愕
Tertiary Dentin: Reactionary/Response And Reparativ
Reparative or tertiary dentin results from pulpal stimulation And forms
only at the site of odontoblastic activation. Whether The formation is
the result of attrition, abrasion, caries, or re-Storative procedures, this
dentin is deposited underlying only Those stimulated areas (Figs. 8-6
and 8-7). It may be deposited Rapidly, in which case the resulting dentin
appears irregular With sparse and twisted tubules and possible cell
inclusions (Fig. 8-6B to E). Odontoblasts, fibroblasts, and blood cell Have
been found in this type of dentin. In contrast, if it is formed slowly
because of fewer stimuli, the dentin appears More regular, much like
primary or secondary dentin (Figs. 8-6 And 8-7A). Reparative dentin at
times resembles bone more Than dentin and is then termed
osteodentin (Fig. 8-7C); It is the type of dentin that is formed under
an exposure That has a hard-set calcium hydroxide as the pulp capping
Medicament. It can also appear as a combination of several Types (Fig.
8-7E). Recent terminology suggests that the term Reactionary/response
dentin be used when the original odonto-Blasts function in deposition
and that reparative dentin be Used when newly recruited (replacement)
odontoblasts begin Depositing dentin. The latter case occurs with a
more severe Injury to the tooth, such as a pulp exposure, which then
neces-Sitates recruitment of progenitor cells that then differentiate
Into new odontoblasts. It is interesting to speculate why the Newly
recruited odontoblasts do not recapitulate development And produce
mantle dentin but instead produce various other Types of dentin,
2. including osteodentin initially and at a much Later time during the
pulpal healing sequence, tubular dentin. Perhaps this is because of the
urgency of protecting the pulp From further damage and thereby
forming a scar,which then Seals the pulp during dentin bridge
formation.